Malou Friederich‐Persson

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Malou Friederich Friederich-persson, Malou Friederich-persson
Publication record
37 papers · 932 citations · H-index 16

PAPERS

Browning of perivascular adipose tissue prevents vascular dysfunction and reduces hypertension in Angiotensin-II infused mice.
202310 citationsauthor #3
Dose-dependent regulation of kidney mitochondrial function by angiotensin II
20234 citationsauthor #5
Thyroid hormone increases oxygen metabolism causing intrarenal tissue hypoxia; a pathway to kidney disease
202214 citationsauthor #2
Adipocyte-Mineralocorticoid Receptor Alters Mitochondrial Quality Control Leading to Mitochondrial Dysfunction and Senescence of Visceral Adipose Tissue
202112 citationsauthor #2
Inhibition of angiotensin-induced aortic aneurysm by metformin in apolipoprotein E–deficient mice
202119 citationsauthor #3
Determinants of renal oxygen metabolism during low Na+ diet: effect of angiotensin II AT1 and aldosterone receptor blockade
20204 citationsauthor #6
Mitochondrial angiotensin II receptors regulate oxygen consumption in kidney mitochondria from healthy and type 1 diabetic rats.
202027 citationsauthor #1
Hypoxia Inducible Factor Activation Prevents Renal Mitochondria Dysfunction and Improves Cortical Oxygenation in Type 1 Diabetic Mice
20190 citationsauthor #11
MR (Mineralocorticoid Receptor) Induces Adipose Tissue Senescence and Mitochondrial Dysfunction Leading to Vascular Dysfunction in Obesity
201951 citationsauthor #2
Inhibition of mammalian target of rapamycin decreases intrarenal oxygen availability and alters glomerular permeability.
20188 citationsauthor #2
Vascular dysfunction in obese diabetic db/db mice involves the interplay between aldosterone/mineralocorticoid receptor and Rho kinase signaling
201839 citationsauthor #3
Mitochondrial oxidative stress in obesity: role of the mineralocorticoid receptor.
201860 citationsauthor #2
Low Sodium Intake Causes Kidney Cortical Hypoxia and Proteinuria in Rats
20180 citationsauthor #4
Deletion of Uncoupling Protein‐2 reduces renal mitochondrial leak respiration, intrarenal hypoxia and proteinuria in a mouse model of type 1 diabetes
201823 citationsauthor #1
Mitochondrial Reactive Oxygen Species and Kidney Hypoxia in the Development of Diabetic Nephropathy
201755 citationsauthor #2
Increased ROS generation involves mitochondria in MR-overexpressing adipocytes – impact on vascular function
20170 citationsauthor #2
Hyaluronan Production by Renomedullary Interstitial Cells: Influence of Endothelin, Angiotensin II and Vasopressin
20177 citationsauthor #5
Brown Adipose Tissue Regulates Small Artery Function Through NADPH Oxidase 4–Derived Hydrogen Peroxide and Redox-Sensitive Protein Kinase G-1&agr;
201741 citationsauthor #1
diabetesnephropathy in four mouse strains with type 1 Differences in susceptibility to develop parameters of
20160 citationsauthor #4
[PP.03.16] INCREASED ROS GENERATION INVOLVES MITOCHONDRIA IN MR-OVEREXPRESSING ADIPOCYTES – IMPACT ON VASCULAR FUNCTION.
20160 citationsauthor #2
Adipocytes, aldosterone and obesity-related hypertension.
201664 citationsauthor #2
Adenosine A2a receptor stimulation prevents proteinuria in diabetic rats by promoting an anti‐inflammatory phenotype without affecting oxidative stress
201530 citationsauthor #2
Renal transplantation induces mitochondrial uncoupling, increased kidney oxygen consumption, and decreased kidney oxygen tension.
201529 citationsauthor #2
Activation of hypoxia-inducible factors prevents diabetic nephropathy.
2015185 citationsauthor #2
Renal Hypoxia Renal transplantation induces mitochondrial uncoupling , increased kidney oxygen consumption , and decreased kidney oxygen tension X
20140 citationsauthor #2
Renal hypoxia due to increased oxygen metabolism is an independent pathway to nephropathy (890.6)
20140 citationsauthor #1
Donor Pretreatment with Mitotempo Decreases Mitochondrial Uncoupling after Experimental Renal Transplantation
20140 citationsauthor #2
Decreased kidney oxygenation due to mitochondrial uncoupling: a mechanism to injury after renal transplantation (890.7)
20140 citationsauthor #1
Differences in susceptibility to develop parameters of diabetic nephropathy in four mouse strains with type 1 diabetes.
201420 citationsauthor #2
Angiotensin II reduces transport-dependent oxygen consumption but increases transport-independent oxygen consumption in immortalized mouse proximal tubular cells.
20144 citationsauthor #1
TRANSPLANTATION BASIC SCIENCE, ALLOGENIC AND XENOGENIC TOLERANCE
20140 citationsauthor #134
Increased kidney metabolism as a pathway to kidney tissue hypoxia and damage: effects of triiodothyronine and dinitrophenol in normoglycemic rats.
201323 citationsauthor #1
Inhibition of mammalian target of rapamycin induces renal mitochondrial uncoupling in rats.
20132 citationsauthor #2
KIDNEY HYPOXIA , DUE TO INCREASED OXYGEN CONSUMPTION , INDUCES NEPHROPATHY INDEPENDENTLY OF HYPERGLYCEMIA AND OXIDATIVE STRESS
20131 citationsauthor #1
Hyaluronan turnover by renomedullary interstitial cells. Influence of fluid regulating hormones
20130 citationsauthor #5
Kidney Hypoxia, Attributable to Increased Oxygen Consumption, Induces Nephropathy Independently of Hyperglycemia and Oxidative Stress
2013164 citationsauthor #1
Acute Knockdown of Uncoupling Protein-2 Increases Uncoupling via the Adenine Nucleotide Transporter and Decreases Oxidative Stress in Diabetic Kidneys
201239 citationsauthor #1

TIMELINE

2012 39 citations

2013 190 citations

2014 24 citations

2015 244 citations

2016 64 citations

2017 103 citations

2018 130 citations

2019 51 citations

2020 31 citations

2021 31 citations

2022 14 citations

2023 14 citations

Malou Friederich‐Persson

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