Stephanie A. Zlatic

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Stephanie A Zlatic, Stephanie Zlatic
Publication record
39 papers · 887 citations · H-index 19

PAPERS

Distinct signaling mechanisms and proteome phenotypes are elicited by compartment-specific genetic defects of copper homeostasis
20260 citationsauthor #4
Suppressive Genetic Interactions Between Haploinsufficient Mitochondrial Genes Encoded in the 22q11.2 Microdeletion Locus Define Brain and Cardiac Phenotypes
20261 citationsauthor #2
Mitochondrially Transcribed dsRNA Mediates Manganese-induced Neuroinflammation
20250 citationsauthor #7
Brain Mecp2 Gene Dosage and Gene Therapy Shape Multi-Omic Signatures and Biomarkers in Rett Syndrome
20250 citationsauthor #1
Adaptive protein synthesis in genetic models of copper deficiency and childhood neurodegeneration
20251 citationsauthor #4
Adaptive protein synthesis in genetic models of copper deficiency and childhood neurodegeneration
20244 citationsauthor #4
Systemic Proteome Phenotypes Reveal Defective Metabolic Flexibility in Mecp2 Mutants
20238 citationsauthor #1
APOE expression and secretion are modulated by mitochondrial dysfunction
202222 citationsauthor #5
FMRP attenuates activity dependent modifications in the mitochondrial proteome
202111 citationsauthor #2
Mitochondrial Proteostasis Requires Genes Encoded in a Neurodevelopmental Syndrome Locus
202124 citationsauthor #3
Heterogeneous Expression of Nuclear Encoded Mitochondrial Genes Distinguishes Inhibitory and Excitatory Neurons
202118 citationsauthor #4
Convergent cerebrospinal fluid proteomes and metabolic ontologies in humans and animal models of Rett syndrome
202111 citationsauthor #1
FMRP attenuates activity dependent modifications in the mitochondrial proteome
20210 citationsauthor #2
Activity-Dependent Mitochondrial Plasticity and Mitoproteome are Dysregulated in Fragile X Syndrome
20200 citationsauthor #2
Golgi-Dependent Copper Homeostasis Sustains Synaptic Development and Mitochondrial Content
202026 citationsauthor #5
Mitochondrial Proteostasis Requires Genes Encoded in a Neurodevelopmental Syndrome Locus that are Necessary for Synapse Function
20200 citationsauthor #3
Ratiometric two-photon microscopy reveals attomolar copper buffering in normal and Menkes mutant cells
201972 citationsauthor #5
Adaptor protein-3 complex is required for Vangl2 trafficking and planar cell polarity of the inner ear
201911 citationsauthor #2
Trafficking mechanisms of P-type ATPase copper transporters.
201954 citationsauthor #2
Rare Disease Mechanisms Identified by Genealogical Proteomics of Copper Homeostasis Mutant Pedigrees.
201819 citationsauthor #1
Systems Analysis of the 22q11.2 Microdeletion Syndrome Converges on a Mitochondrial Interactome Necessary for Synapse Function and Behavior
201850 citationsauthor #5
Dysbindin Deficiency Modifies the Expression of GABA Neuron and Ion Permeation Transcripts in the Developing Hippocampus
201723 citationsauthor #2
Author response: The interactome of the copper transporter ATP7A belongs to a network of neurodevelopmental and neurodegeneration factors
20171 citationsauthor #6
The interactome of the copper transporter ATP7A belongs to a network of neurodevelopmental and neurodegeneration factors
201760 citationsauthor #6
Identification of the Interactome of a Palmitoylated Membrane Protein, Phosphatidylinositol 4-Kinase Type II Alpha.
20167 citationsauthor #3
The Proteome of BLOC-1 Genetic Defects Identifies the Arp2/3 Actin Polymerization Complex to Function Downstream of the Schizophrenia Susceptibility Factor Dysbindin at the Synapse
201624 citationsauthor #5
The Endosome Localized Arf-GAP AGAP1 Modulates Dendritic Spine Morphology Downstream of the Neurodevelopmental Disorder Factor Dysbindin
201619 citationsauthor #4
The N-Ethylmaleimide-Sensitive Factor and Dysbindin Interact To Modulate Synaptic Plasticity
201529 citationsauthor #3
Neuronal copper homeostasis susceptibility by genetic defects in dysbindin, a schizophrenia susceptibility factor.
201527 citationsauthor #5
Molecular basis of neurodegeneration and neurodevelopmental defects in Menkes disease.
201553 citationsauthor #1
Mutations in the BLOC-1 Subunits Dysbindin and Muted Generate Divergent and Dosage-dependent Phenotypes*
201433 citationsauthor #2
Chemical-genetic disruption of clathrin function spares adaptor complex 3–dependent endosome vesicle biogenesis
201323 citationsauthor #1
Vps33b pathogenic mutations preferentially affect VIPAS39/SPE-39-positive endosomes.
201325 citationsauthor #2
The schizophrenia susceptibility factor dysbindin and its associated complex sort cargoes from cell bodies to the synapse
201176 citationsauthor #5
Metazoan cell biology of the HOPS tethering complex
201117 citationsauthor #1
Clathrin-dependent mechanisms modulate the subcellular distribution of class C Vps/HOPS tether subunits in polarized and nonpolarized cells
201126 citationsauthor #1
Hermansky-Pudlak Syndrome Protein Complexes Associate with Phosphatidylinositol 4-Kinase Type II α in Neuronal and Non-neuronal Cells*
200954 citationsauthor #2
SPE-39 family proteins interact with the HOPS complex and function in lysosomal delivery.
200967 citationsauthor #3
Hermansky-Pudlak Syndrome Protein Complexes Associate withPhosphatidylinositol 4-Kinase Type II α in Neuronal and Non-neuronal Cells
year unknown0 citationsauthor #2

TIMELINE

2009 121 citations

2011 119 citations

2013 48 citations

2014 33 citations

2015 109 citations

2016 50 citations

2017 84 citations

2018 69 citations

2019 137 citations

2020 26 citations

2021 64 citations

2022 22 citations

2023 8 citations

2024 4 citations

2025 1 citations

2026 1 citations

Unknown 0 citations

Stephanie A. Zlatic

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