Resistance of Optogenetically Evoked Motor Function to Global Ischemia and Reperfusion in Mouse in Vivo

No abstract is available for this paper.

• Publication year

  2013

• Venue

  Journal of Cerebral Blood Flow and Metabolism

• Publication date

  2013-08-01

• Fields of study

  Biology, Medicine

• Identifiers
  DOI 10.1038/jcbfm.2013.89PMID 23736644PMCID PMC3734785
• External record

  Open on Semantic Scholar

• Source metadata

  Semantic Scholar, PubMed

• No claims are published for this paper.

• No concepts are published for this paper.

• Brain excitability in stroke: the yin and yang of stroke progression.

  2012cited by this paper
• Brain Excitability in StrokeThe Yin and Yang of Stroke Progression

  2012cited by this paper
• In vivo Large-Scale Cortical Mapping Using Channelrhodopsin-2 Stimulation in Transgenic Mice Reveals Asymmetric and Reciprocal Relationships between Cortical Areas

  2012cited by this paper
• Optogenetic Analysis of Neuronal Excitability during Global Ischemia Reveals Selective Deficits in Sensory Processing following Reperfusion in Mouse Cortex

  2012cited by this paper
• The organization of the forelimb representation of the C57BL/6 mouse motor cortex as defined by intracortical microstimulation and cytoarchitecture.

  2011cited by this paper
• The sigma-1 receptor enhances brain plasticity and functional recovery after experimental stroke.

  2011cited by this paper
• Mild Sensory Stimulation Reestablishes Cortical Function during the Acute Phase of Ischemia

  2011cited by this paper
• Plasticity during stroke recovery: from synapse to behaviour

  2009cited by this paper
• Automated light-based mapping of motor cortex by photoactivation of channelrhodopsin-2 transgenic mice

  2009cited by this paper
• Two-Photon Imaging of Stroke Onset In Vivo Reveals That NMDA-Receptor Independent Ischemic Depolarization Is the Major Cause of Rapid Reversible Damage to Dendrites and Spines

  2008cited by this paper
• In vivo light-induced activation of neural circuitry in transgenic mice expressing channelrhodopsin-2.

  2007cited by this paper
• Mechanisms for recovery of motor function following cortical damage.

  2006cited by this paper
• Persistent Defect in Transmitter Release and Synapsin Phosphorylation in Cerebral Cortex After Transient Moderate Ischemic Injury

  2002cited by this paper
• Mechanisms of Motor Dysfunction After Transient MCA Occlusion: Persistent Transmission Failure in Cortical Synapses Is a Major Determinant

  1998cited by this paper
• Mechanisms of motor dysfunction after transient MCA occlusion: persistent transmission failure in cortical synapses is a major determinant.

  1998cited by this paper
• Recovery of integrative central nervous function after one hour global cerebro-circulatory arrest in normothermic cat.

  1987cited by this paper
• Thalamic and corticocortical connections of the second somatic sensory area of the mouse

  1987cited by this paper

• Cerebral blood flow patterns induced by photoactivation based on laser speckle contrast imaging

  2024influential citation
• Application of optogenetic glial cells to neuron–glial communication

  2023cites this paper
• Optogenetic Interrogation of Circuits Following Neurotrauma

  2021cites this paper
• Neurovascular Reactivity in the Aging Mouse Brain Assessed by Laser Speckle Contrast Imaging and 2-Photon Microscopy: Quantification by an Investigator-Independent Analysis Tool

  2021cites this paper
• Optogenetics in Brain Research: From a Strategy to Investigate Physiological Function to a Therapeutic Tool

  2019cites this paper
• Selective photoactivation of neural activity combined with laser speckle imaging of cerebral blood flow.

  2018cites this paper
• Regeneration and plasticity of descending motor pathways following spinal cord injury

  2016cites this paper
• Hypothermia-induced ischemic tolerance is associated with Drp1 inhibition in cerebral ischemia-reperfusion injury of mice.

  2016cites this paper
• COX-2-Derived Prostaglandin E2 Produced by Pyramidal Neurons Contributes to Neurovascular Coupling in the Rodent Cerebral Cortex

  2015cites this paper
• Spatially resolving acute functional changes in descending cortical motor output after targeted stroke

  2015cites this paper
• Optogenetic Stimulation of GABA Neurons can Decrease Local Neuronal Activity While Increasing Cortical Blood Flow

  2015cites this paper
• Patterned Optogenetic Modulation of Neurovascular and Metabolic Signals

  2015cites this paper
• Expanding applications, accuracy, and interpretation of laser speckle contrast imaging of cerebral blood flow

  2015cites this paper
• Optogenetic investigation of neuronal excitability and sensory-motor function following a transient global ischemia in mice

  2015influential citation
• Prolonged Deficits in Parvalbumin Neuron Stimulation-Evoked Network Activity Despite Recovery of Dendritic Structure and Excitability in the Somatosensory Cortex following Global Ischemia in Mice

  2014cites this paper
• Les neurones pyramidaux corticaux dans le couplage neurovasculaire et neurométabolique : mécanismes cellulaires et moléculaires

  2014cites this paper
• Ministrokes in Channelrhodopsin-2 Transgenic Mice Reveal Widespread Deficits in Motor Output Despite Maintenance of Cortical Neuronal Excitability

  2014cites this paper
• Stroke and the connectome: how connectivity guides therapeutic intervention.

  2014cites this paper