The blood-brain barrier and selective vulnerability in experimental thiamine-deficiency encephalopathy in the mouse

No abstract is available for this paper.

• Publication year

  2005

• Venue

  Metabolic brain disease

• Publication date

  Unknown publication date

• Fields of study

  Biology, Medicine

• Identifiers
  DOI 10.1007/BF02237965PMID 8815390
• External record

  Open on Semantic Scholar

• Source metadata

  Semantic Scholar, PubMed

• No claims are published for this paper.

• No concepts are published for this paper.

• Mechanisms of blood-brain barrier breakdown

  2004cited by this paper
• Regional alterations of thiamine phosphate esters and of thiamine diphosphate-dependent enzymes in relation to function in experimental Wernicke's encephalopathy

  2004cited by this paper
• Blood-brain barrier dysfunction in thiamine-deficient, alcohol-treated rats

  2004cited by this paper
• Evidence for early blood-brain barrier breakdown in experimental thiamine deficiency in the mouse

  1995cited by this paper
• GFAP‐positive hippocampal astrocytes in situ respond to glutamatergic neuroligands with increases in [Ca2+]i

  1995cited by this paper
• Excitotoxic Cytopathology, Progression, and Reversibility of Thiamine Deficiency‐induced Diencephalic Lesions

  1995cited by this paper
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• Glutamate-induced swelling of single astroglial cells in primary culture.

  1994cited by this paper
• Glutamate receptor agonists stimulate diverse calcium responses in different types of cultured rat cortical glial cells

  1994cited by this paper
• Kainate activates Ca2+-permeable glutamate receptors and blocks voltage-gated K+ currents in glial cells of mouse hippocampal slices

  1994cited by this paper
• Metabotropic glutamate receptor activation induces astroglial swelling.

  1994cited by this paper
• The early events of oxygen and glucose deprivation: setting the scene for neuronal death?

  1994cited by this paper
• A comparison of non‐NMDA receptor channels in type‐2 astrocytes and granule cells from rat cerebellum.

  1994cited by this paper
• Reappraisal of regional thiamine content in the central nervous system of the normal and thiamine-deficient mice

  1993cited by this paper
• Extracellular Glutamate Is Increased in Thalamus During Thiamine Deficiency‐Induced Lesions and Is Blocked by MK‐801

  1993cited by this paper
• Cerebral Vulnerability Is Associated with Selective Increase in Extracellular Glutamate Concentration in Experimental Thiamine Deficiency

  1993cited by this paper
• NMDA-activated currents in Bergmann glial cells.

  1993cited by this paper
• Calcium-permeable AMPA-kainate receptors in fusiform cerebellar glial cells.

  1992cited by this paper
• Breakdown of the Blood–Brain Barrier After Fluid Percussion Brain Injury in the Rat: Part 2: Effect of Hypoxia on Permeability to Plasma Proteins

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• A cell culture model of the blood-brain barrier

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• Blood-brain-barrier disruption in acute Wernicke encephalopathy: MR findings.

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  1990cited by this paper
• Wernicke encephalopathy: MR findings in five patients.

  1990cited by this paper
• Protective effects of the glutamate antagonist MK-801 on pyrithiamine- induced lesions and amino acid changes in rat brain

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• Age-associated accumulation of heat-labile aminoacyl-tRNA synthetases in mice and rats.

  1987cited by this paper
• Activities of thiamine-dependent enzymes in two experimental models of thiamine-deficiency encephalopathy. 2. alpha-Ketoglutarate dehydrogenase.

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  1982cited by this paper
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  1981cited by this paper
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  1981cited by this paper
• Edematous Necrosis in Thiamine‐Deficient Encephalopathy of the Mouse

  1981cited by this paper
• Developing nervous tissue induces formation of blood-brain barrier characteristics in invading endothelial cells: a study using quail--chick transplantation chimeras.

  1981cited by this paper
• Low Energy Levels in Thiamine‐Deficient Encephalopathy

  1981cited by this paper
• gamma-Glutamyl transpeptidase in isolated brain endothelial cells: induction by glial cells in vitro.

  1980cited by this paper
• Thiamine content and turnover rates of some rat nervous regions, using labeled thiamine as a tracer

  1980cited by this paper
• EARLY EDEMATOUS LESION OF PYRITHIAMINE INDUCED ACUTE THIAMINE DEFICIENT ENCEPHALOPATHY IN THE MOUSE

  1978cited by this paper
• Pyrithiamine-induced acute thiamine-deficient encephalopathy in the mouse.

  1978cited by this paper
• TRANSPORT OF METABOLIC SUBSTRATES THROUGH THE BLOOD‐BRAIN BARRIER 1

  1977cited by this paper
• Vascular permeability to horseradish peroxidase in brainstem lesions of thiamine-deficient rats.

  1972cited by this paper
• Effect of thiamine deficiency on the competence of the blood-brain barrier to albumin labeled with fluorescent dyes.

  1971cited by this paper
• JUNCTIONS BETWEEN INTIMATELY APPOSED CELL MEMBRANES IN THE VERTEBRATE BRAIN

  1969cited by this paper
• Ultrastructural features of early brain stem lesions of thiamine-deficient rats.

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  1968cited by this paper
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• [Experimental electron microscopic studies on the formal pathogenesis of Wernick's encephalopathy].

  1967cited by this paper
• FINE STRUCTURAL LOCALIZATION OF A BLOOD-BRAIN BARRIER TO EXOGENOUS PEROXIDASE

  1967cited by this paper
• Glial cell changes in the brain stem of thiamine-deficient rats.

  1967cited by this paper
• Lead encephalo-myelopathy of the suckling rat and its implications on the porphyrinopathic nervous diseases

  1966cited by this paper
• TRANSPORT AND METABOLISM OF THIAMINE IN RAT BRAIN CORTEX IN VITRO.

  1965cited by this paper
• THE RELATIONSHIPS BETWEEN SUBSTRATES AND ENZYMES OF GLYCOLYSIS IN BRAIN.

  1964cited by this paper
• THE QUANTITATIVE HISTOCHEMICAL DISTRIBUTION OF THIAMINE IN DEFICIENT RAT BRAIN *

  1961cited by this paper
• Effects of thiamine deficiency on the central nervous system.

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  1960cited by this paper
• SUBACUTE NECROTIZING ENCEPHALOMYELOPATHY IN AN INFANT

  1951cited by this paper
• Effect of experimental thiamine deficiency on the nervous system of the rhesus monkey.

  1949cited by this paper
• Lesions of chronic thiamine deficiency in mice.

  1947cited by this paper
• Wernicke's disease: Identity of lesions produced experimentally by B(1) avitaminosis in pigeons with hemorrhagic polioencephalitis occurring in chronic alcoholism in man.

  1940cited by this paper
• On the chemical nature of the substance which cures polyneuritis in birds induced by a diet of polished rice

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