Voltage-dependent open-channel block of G protein-gated inward-rectifying K+ (GIRK) current in rat atrial myocytes by tamoxifen

No abstract is available for this paper.

• Publication year

  2012

• Venue

  Naunyn-Schmiedeberg's Archives of Pharmacology

• Publication date

  2012-10-25

• Fields of study

  Biology, Medicine, Chemistry

• Identifiers
  DOI 10.1007/s00210-012-0801-8PMID 23096593
• External record

  Open on Semantic Scholar

• Source metadata

  Semantic Scholar, PubMed

• No claims are published for this paper.

• No concepts are published for this paper.

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• Inwardly rectifying potassium channels: their structure, function, and physiological roles.

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• Cardiac strong inward rectifier potassium channels.

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• 4-Hydroxytamoxifen inhibits K(+) currents in mouse ventricular myocytes.

  2010cited by this paper
• Emerging roles for G protein-gated inwardly rectifying potassium (GIRK) channels in health and disease

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  2010influential reference
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• Tamoxifen‐induced QT interval prolongation

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  2003influential reference
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  2002cited by this paper
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  2002cited by this paper
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  1998influential reference
• Non‐receptor‐mediated activation of IK(ATP) and inhibition of IK(ACh) by diadenosine polyphosphates in guinea‐pig atrial myocytes

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  1998cited by this paper
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• A phase I study of high-dose tamoxifen for the treatment of refractory malignant gliomas of childhood.

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  1995cited by this paper
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