Comparison of the Ca2+ entry channels responsible for mechanical responses of guinea-pig aorta to noradrenaline and thapsigargin using SK&F 96365 and LOE 908

No abstract is available for this paper.

• Publication year

  2000

• Venue

  Naunyn-Schmiedeberg's Archives of Pharmacology

• Publication date

  2000-07-18

• Fields of study

  Biology, Medicine, Chemistry

• Identifiers
  DOI 10.1007/s002100000272PMID 10961379
• External record

  Open on Semantic Scholar

• Source metadata

  Semantic Scholar, PubMed

• No claims are published for this paper.

• No concepts are published for this paper.

• Receptor-activated Ca2+ inflow in animal cells: a variety of pathways tailored to meet different intracellular Ca2+ signalling requirements.

  1999influential reference
• Pharmacological characterization of Ca2+ entry channels in endothelin‐1‐induced contraction of rat aorta using LOE 908 and SK&F 96365

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• Evidence for a contribution of store-operated Ca2+ channels to NO-mediated endothelium-dependent relaxation of guinea-pig aorta in response to a Ca2+ ionophore, A23187

  1999cited by this paper
• Possible Involvement of Ca2+ Entry and its Pharmacological Characteristics Responsible for Endothelium‐dependent, NO‐mediated Relaxation Induced by Thapsigargin in Guinea‐pig Aorta

  1999cited by this paper
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  1998cited by this paper
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  1998cited by this paper
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  1997cited by this paper
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  1996cited by this paper
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