Increased AS 160 phosphorylation , but not TBC 1 D 1 phosphorylation , with increased postexercise insulin sensitivity in rat skeletal muscle

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• Inhibition of Contraction-Stimulated AMP-Activated Protein Kinase Inhibits Contraction-Stimulated Increases in PAS-TBC1D1 and Glucose Transport Without Altering PAS-AS160 in Rat Skeletal Muscle

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• Potential role of TBC1D4 in enhanced post-exercise insulin action in human skeletal muscle

  2009cited by this paper
• A myosin II ATPase inhibitor reduces force production, glucose transport, and phosphorylation of AMPK and TBC1D1 in electrically stimulated rat skeletal muscle.

  2009cited by this paper
• Contractile C2C12 myotube model for studying exercise-inducible responses in skeletal muscle.

  2008cited by this paper
• Resistance exercise increases human skeletal muscle AS160/TBC1D4 phosphorylation in association with enhanced leg glucose uptake during postexercise recovery.

  2008cited by this paper
• GLUT4 Vesicle Recruitment and Fusion Are Differentially Regulated by Rac, AS160, and Rab8A in Muscle Cells*

  2008cited by this paper
• Contraction-stimulated glucose transport in rat skeletal muscle is sustained despite reversal of increased PAS-phosphorylation of AS160 and TBC1D1.

  2008cited by this paper
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• Edinburgh Research Explorer The glycogen-binding domain on the AMPK beta subunit allows the kinase to act as a glycogen sensor

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• Inhibition of GLUT4 Translocation by Tbc1d1, a Rab GTPase-activating Protein Abundant in Skeletal Muscle, Is Partially Relieved by AMP-activated Protein Kinase Activation*

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• Discovery of TBC1D1 as an Insulin-, AICAR-, and Contraction-stimulated Signaling Nexus in Mouse Skeletal Muscle*

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• Calmodulin-Binding Domain of AS160 Regulates Contraction- but Not Insulin-Stimulated Glucose Uptake in Skeletal Muscle

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• AS160 phosphorylation is associated with activation of α2β2γ1- but not α2β2γ3-AMPK trimeric complex in skeletal muscle during exercise in humans

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• Prior exercise increases phosphorylation of Akt substrate of 160 kDa (AS160) in rat skeletal muscle.

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• Regulation of multisite phosphorylation and 14-3-3 binding of AS160 in response to IGF-1, EGF, PMA and AICAR.

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• A Method to Identify Serine Kinase Substrates

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• Modelling glucose dynamics during moderate exercise in individuals with type 1 diabetes

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• The Influence of Physical Activity on the Bioactive Lipids Metabolism in Obesity-Induced Muscle Insulin Resistance

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• Whole body glucoregulation and tissue-specific glucose uptake in a novel Akt substrate of 160 kDa knockout rat model

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• Acute exercise alters skeletal muscle mitochondrial respiration and H2O2 emission in response to hyperinsulinemic-euglycemic clamp in middle-aged obese men

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• Update on the effects of physical activity on insulin sensitivity in humans

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• Exercise training prevents skeletal muscle plasma membrane cholesterol accumulation, cortical actin filament loss, and insulin resistance in C57BL/6J mice fed a western‐style high‐fat diet

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• Acute exercise increases insulin sensitivity in adult sheep: a new preclinical model.

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• Mechanisms for independent and combined effects of calorie restriction and acute exercise on insulin-stimulated glucose uptake by skeletal muscle of old rats.

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• reducing intramuscular lipids whilemuscle insulin resistance and fatty acid oxidation Restoring AS 160 phosphorylation rescues skeletal

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• Restoring AS160 phosphorylation rescues skeletal muscle insulin resistance and fatty acid oxidation while not reducing intramuscular lipids.

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