Membrane ion transport in erythrocytes of salt hypertensive Dahl rats and their F2 hybrids: the importance of cholesterol.

No abstract is available for this paper.

• Publication year

  2003

• Venue

  Hypertension Research

• Publication date

  2003-05-01

• Fields of study

  Biology, Medicine, Chemistry

• Identifiers
  DOI 10.1291/HYPRES.26.397PMID 12887131
• External record

  Open on Semantic Scholar

• Source metadata

  Semantic Scholar, PubMed

• No claims are published for this paper.

• No concepts are published for this paper.

• Ca2+ buffering function of the sarcoplasmic reticulum is increased in the carotid artery from spontaneously hypertensive rats.

  2002cited by this paper
• Altered Na+-K+ pump activity and plasma lipids in salt-hypertensive Dahl rats: relationship to Atp1a1 gene.

  2001cited by this paper
• Voltage-dependent Ca2+ channels in resistance arteries from Dahl salt-sensitive rats.

  2000influential reference
• Abnormalities of membrane function and lipid metabolism in hypertension: a review.

  1999cited by this paper
• The erythrocyte Na,K,Cl cotransporter and its circulating inhibitor in Dahl salt‐sensitive rats

  1998cited by this paper
• Single L-type calcium channels in smooth muscle cells from resistance arteries of spontaneously hypertensive rats.

  1998cited by this paper
• Role of eicosanoids in alteration of membrane electrical properties in isolated mesenteric arteries of salt‐loaded, Dahl salt‐sensitive rats

  1997influential reference
• Plasma triglycerides and red cell ion transport alterations in genetically hypertensive rats.

  1997cited by this paper
• Pathophysiological consequences of changes in the coupling ratio of Na,K-ATPase for renal sodium reabsorption and its implications for hypertension.

  1996cited by this paper
• Alterations of cytosolic calcium in platelets and erythrocytes of Lyon hypertensive rats.

  1995cited by this paper
• D1 - Increased erythrocyte rubidium leak as a marker of salt sensitivity in the rat?

  1995cited by this paper
• Erythrocyte ion transport alterations in hypertriglyceridaemic rats.

  1994cited by this paper
• Mechanism of hypertriglyceridemia in Dahl salt-sensitive rats, an animal model of spontaneous nephrotic syndrome.

  1994cited by this paper
• Calcium current in smooth muscle cells from normotensive and genetically hypertensive rats.

  1994cited by this paper
• The α1 Na+−K+ pump of the Dahl salt-sensitive rat exhibits altered Na+ modulation of K+ transport in red blood cells

  1993cited by this paper
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  1993cited by this paper
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  1993cited by this paper
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  1993cited by this paper
• Cholesterol increases the L-type voltage-sensitive calcium channel current in arterial smooth muscle cells.

  1992influential reference
• Interactions of hypercholesterolemia and hypertension in initiation of glomerular injury.

  1992cited by this paper
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  1992cited by this paper
• Association of red blood cell sodium leak with blood pressure in recombinant inbred strains.

  1992cited by this paper
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  1991cited by this paper
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  1991cited by this paper
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  1991influential reference
• Membrane Sodium Transport and Salt Sensitivity of Blood Pressure

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  1990cited by this paper
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  1990cited by this paper
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  1990cited by this paper
• Kinetics of Na+ and K+ transport in red blood cells of Dahl rats. Effects of age and salt.

  1990cited by this paper
• Risk factors for glomerular injury in rats with genetic hypertension.

  1989cited by this paper
• Effect of low-density lipoprotein on intracellular calcium, intracellular pH and DNA synthesis in cultured vascular smooth muscle cells.

  1989cited by this paper
• Sodium transport kinetics in erythrocytes from spontaneously hypertensive rats.

  1988influential reference
• Calcium Currents Are Altered in the Vascular Muscle Cell Membrane of Spontaneously Hypertensive Rats

  1988cited by this paper
• Erythrocyte cation fluxes in the normotensive and hypertensive clients of a health screening clinic.

  1987cited by this paper
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  1987cited by this paper
• [Genetic analysis of inheritance of Na+, K+-cotransport rate, calcium level in erythrocytes and blood pressure in F2 hybrids of spontaneously hypertensive and normotensive rats].

  1987cited by this paper
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  1986cited by this paper
• Role of exogenous factors in alterations of red cell Na+-Li+ exchange and Na+-K+ cotransport in essential hypertension, primary hyperaldosteronism, and hypokalemia.

  1986cited by this paper
• Ion transport across plasma membrane in primary hypertension.

  1985cited by this paper
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  1985cited by this paper
• Erythrocytic sodium ion transport systems in primary and secondary hypertension of the rat.

  1982influential reference
• Intracellular cation activities and concentrations in spontaneously hypertensive and normotensive rats.

  1981cited by this paper
• Abnormal net Na+ and K+ fluxes in erythrocytes of three varieties of genetically hypertensive rats.

  1980cited by this paper
• The effect of membrane cholesterol on the sodium pump in red blood cells.

  1978cited by this paper
• Altered permeability of the erythrocyte membrane for sodium and potassium ions in spontaneously hypertensive rats

  1976influential reference
• Increased erythrocyte permeability to Li and Na in the spontaneously hypertensive rat

  1976influential reference
• Increased erythrocytes sodium efflux in genetic hypertensive rats of the Hebrew University strain

  1975influential reference
• Effects of chronic excess saltfeeding. Elevation of plasma cholesterol in rats and dogs.

  1960cited by this paper

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  2013cites this paper
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  2006cites this paper
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  2005cites this paper
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  2005cites this paper