Influence of XPB helicase on recruitment and redistribution of nucleotide excision repair proteins at sites of UV-induced DNA damage.

No abstract is available for this paper.

• Publication year

  2007

• Venue

  DNA Repair

• Publication date

  2007-09-01

• Fields of study

  Biology, Medicine

• Identifiers
  DOI 10.1016/J.DNAREP.2007.03.025PMID 17509950PMCID PMC3471374
• External record

  Open on Semantic Scholar

• Source metadata

  Semantic Scholar, PubMed

• No claims are published for this paper.

• No concepts are published for this paper.

• Phenotypic heterogeneity in the XPB DNA helicase gene (ERCC3): xeroderma pigmentosum without and with Cockayne syndrome

  2006influential reference
• DNA Repair and Mutagenesis

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• Conserved XPB core structure and motifs for DNA unwinding: implications for pathway selection of transcription or excision repair.

  2006cited by this paper
• Recruitment of the Nucleotide Excision Repair Endonuclease XPG to Sites of UV-Induced DNA Damage Depends on Functional TFIIH

  2006cited by this paper
• The Spacer Region of XPG Mediates Recruitment to Nucleotide Excision Repair Complexes and Determines Substrate Specificity*

  2005cited by this paper
• Centrin 2 Stimulates Nucleotide Excision Repair by Interacting with Xeroderma Pigmentosum Group C Protein

  2005cited by this paper
• Nonsense-mediated mRNA decay in mammals

  2005cited by this paper
• Recognition of RNA polymerase II and transcription bubbles by XPG, CSB, and TFIIH: insights for transcription-coupled repair and Cockayne Syndrome.

  2005cited by this paper
• Differential Repair of the Two Major UV-Induced Photolesions in Trichothiodystrophy Fibroblasts

  2004cited by this paper
• Phosphorylation of XPB helicase regulates TFIIH nucleotide excision repair activity

  2004cited by this paper
• In vivo dynamics of chromatin-associated complex formation in mammalian nucleotide excision repair.

  2004cited by this paper
• Definition of a Short Region of XPG Necessary for TFIIH Interaction and Stable Recruitment to Sites of UV Damage

  2004cited by this paper
• Ordered Conformational Changes in Damaged DNA Induced by Nucleotide Excision Repair Factors*

  2004cited by this paper
• Xeroderma Pigmentosum Group A Protein Loads as a Separate Factor onto DNA Lesions

  2003cited by this paper
• Tumor suppressor p53 dependent recruitment of nucleotide excision repair factors XPC and TFIIH to DNA damage.

  2003cited by this paper
• The comings and goings of nucleotide excision repair factors on damaged DNA

  2003cited by this paper
• Relationship of neurologic degeneration to genotype in three xeroderma pigmentosum group G patients.

  2002influential reference
• Reduced level of the repair/transcription factor TFIIH in trichothiodystrophy.

  2002cited by this paper
• The total amount of DNA damage determines ultraviolet-radiation-induced cytotoxicity after uniformor localized irradiation of human cells.

  2002cited by this paper
• Xeroderma pigmentosum – bridging a gap between clinic and laboratory

  2001cited by this paper
• Local UV‐induced DNA damage in cell nuclei results in local transcription inhibition

  2001cited by this paper
• In situ visualization of ultraviolet-light-induced DNA damage repair in locally irradiated human fibroblasts.

  2001cited by this paper
• Quantitation and visualization of ultraviolet-induced DNA damage using specific antibodies: application to pigment cell biology.

  2001influential reference
• Sequential assembly of the nucleotide excision repair factors in vivo.

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• Stable binding of human XPC complex to irradiated DNA confers strong discrimination for damaged sites.

  2000cited by this paper
• Rare case of Cockayne syndrome with xeroderma pigmentosum.

  2000cited by this paper
• The relative expression of mutated XPB genes results in xeroderma pigmentosum/Cockayne's syndrome or trichothiodystrophy cellular phenotypes.

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• Nucleotide excision repair: from E. coli to man.

  1999cited by this paper
• Quality control by DNA repair.

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• Order of Assembly of Human DNA Repair Excision Nuclease*

  1999cited by this paper
• Molecular mechanism of nucleotide excision repair.

  1999cited by this paper
• Three-dimensional visualization of ultraviolet-induced DNA damage and its repair in human cell nuclei.

  1998cited by this paper
• Xeroderma pigmentosum group C protein complex is the initiator of global genome nucleotide excision repair.

  1998cited by this paper
• Mechanism of open complex and dual incision formation by human nucleotide excision repair factors

  1997cited by this paper
• The Genetic Basis of Human Cancer

  1997cited by this paper
• Interactions involving the human RNA polymerase II transcription/nucleotide excision repair complex TFIIH, the nucleotide excision repair protein XPG, and Cockayne syndrome group B (CSB) protein.

  1996cited by this paper
• A 3′ → 5′ XPB Helicase Defect in Repair/Transcription Factor TFIIH of Xeroderma Pigmentosum Group B Affects Both DNA Repair and Transcription*

  1996cited by this paper
• Clinical heterogeneity within xeroderma pigmentosum associated with mutations in the DNA repair and transcription gene ERCC3.

  1994cited by this paper
• Structure and expression of the human XPBC/ERCC-3 gene involved in DNA repair disorders xeroderma pigmentosum and Cockayne's syndrome.

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• SIMULTANEOUS ESTABLISHMENT OF MONOCLONAL ANTIBODIES SPECIFIC FOR EITHER CYCLOBUTANE PYRIMIDINE DIMER OR (6‐4)PHOTOPRODUCT FROM THE SAME MOUSE IMMUNIZED WITH ULTRAVIOLET‐IRRADIATED DNA

  1991cited by this paper
• A presumed DNA helicase encoded by ERCC-3 is involved in the human repair disorders xeroderma pigmentosum and Cockayne's syndrome.

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• Molecular cloning and biological characterization of the human excision repair gene ERCC-3

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• Genetic heterogeneity in ataxia-telangiectasia studied by cell fusion.

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• Regulation of XPC Binding Dynamics and Global Nucleotide Excision Repair by p63 and Vitamin D Receptor.

  2023cites this paper
• Heterogeneity and overlaps in nucleotide excision repair disorders

  2020influential citation
• Triptolide: reflections on two decades of research and prospects for the future.

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• ERCC3 Gene

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• In Situ Analysis of DNA-Protein Complex Formation upon Radiation-Induced DNA Damage

  2019cites this paper
• The Role of Dynamic m6A RNA Methylation in Photobiology

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• Protein phosphatase 4 regulatory subunit 2 (PPP4R2) is recurrently deleted in acute myeloid leukemia and required for efficient DNA double strand break repair

  2017cites this paper
• A unified model for the molecular basis of Xeroderma pigmentosum-Cockayne Syndrome

  2015cites this paper
• MC1R and NR4A receptors in cellular stress and DNA repair: implications for UVR protection

  2014cites this paper
• Novel Mechanisms through which translesion synthesis protects genome integrity

  2013influential citation
• Characterization of Three XPG-Defective Patients Identifies Three Missense Mutations that Impair Repair

  2013cites this paper
• Repair of UV photolesions in xeroderma pigmentosum group C cells induced by translational readthrough of premature termination codons

  2013cites this paper
• Characterization of three XPG-defective patients identifies three missense mutations that impair repair and transcription.

  2013cites this paper
• The NR4A2 Nuclear Receptor Is Recruited to Novel Nuclear Foci in Response to UV Irradiation and Participates in Nucleotide Excision Repair

  2013cites this paper
• Lack of CAK complex accumulation at DNA damage sites in XP-B and XP-B/CS fibroblasts reveals differential regulation of CAK anchoring to core TFIIH by XPB and XPD helicases during nucleotide excision repair.

  2012cites this paper
• Rare hereditary diseases with defects in DNA-repair.

  2012cites this paper
• UV-induced histone H2AX phosphorylation and DNA damage related proteins accumulate and persist in nucleotide excision repair-deficient XP-B cells.

  2011cites this paper
• Nucleotide Excision Repair Proteins Rapidly Accumulate but Fail to Persist in Human XP‐E (DDB2 Mutant) Cells

  2011cites this paper
• XPB Induces C1D Expression to Counteract UV-Induced Apoptosis

  2010cites this paper
• Diagnosing Xeroderma Pigmentosum Group C by Immunohistochemistry

  2010cites this paper
• XPC branch‐point sequence mutations disrupt U2 snRNP binding, resulting in abnormal pre‐mRNA splicing in xeroderma pigmentosum patients

  2010influential citation
• Single-stranded DNA binding activity of XPBI, but not XPBII, from Sulfolobus tokodaii causes double-stranded DNA melting

  2010cites this paper
• The chromatin remodeling factor BRG1 stimulates nucleotide excision repair by facilitating recruitment of XPC to sites of DNA damage

  2009cites this paper
• Disorders of nucleotide excision repair: the genetic and molecular basis of heterogeneity

  2009cites this paper
• Telomere attrition and genomic instability in xeroderma pigmentosum type-b deficient fibroblasts under oxidative stress

  2009cites this paper
• XPC initiation codon mutation in xeroderma pigmentosum patients with and without neurological symptoms.

  2009cites this paper
• Chapter 6. Application of new methods for detection of DNA damage and repair.

  2009cites this paper
• Cell and Molecular Biology

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• Dynamics of nucleotide excision repair complex assembly and disassembly in vivo

  2008cites this paper
• Excision repair is required for genotoxin-induced mutagenesis in mammalian cells.

  2008cites this paper
• Dissection of the Molecular Defects Caused by Pathogenic Mutations in the DNA Repair Factor XPC

  2008cites this paper
• Persistence of repair proteins at unrepaired DNA damage distinguishes diseases with ERCC2 (XPD) mutations: cancer‐prone xeroderma pigmentosum vs. non‐cancer‐prone trichothiodystrophy

  2008cites this paper
• Sequential recruitment of the repair factors during NER: the role of XPG in initiating the resynthesis step

  2008cites this paper
• Diagnosis of Xeroderma Pigmentosum and Related DNA Repair-Deficient Cutaneous Diseases.

  2008cites this paper
• Coin and Jean Marc Egly Factor Xpc by Pathogenic Mutations in the Dna Repair Dissection of the Molecular Defects Caused Supplemental Material

  2008cites this paper