Glycemic state regulates melanocortin, but not nesfatin-1, responsiveness of glucose-sensing neurons in the nucleus of the solitary tract.

No abstract is available for this paper.

• Publication year

  2015

• Venue

  American Journal of Physiology. Regulatory Integrative and Comparative Physiology

• Publication date

  2015-04-15

• Fields of study

  Biology, Medicine

• Identifiers
  DOI 10.1152/ajpregu.00477.2014PMID 25695291PMCID PMC4398857
• External record

  Open on Semantic Scholar

• Source metadata

  Semantic Scholar, PubMed

• No claims are published for this paper.

• No concepts are published for this paper.

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  2012cited by this paper
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  2012cited by this paper
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  2010cited by this paper
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• Adaptive sugar sensors in hypothalamic feeding circuits

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• Oxytocin Enhances Cranial Visceral Afferent Synaptic Transmission to the Solitary Tract Nucleus

  2008cited by this paper
• D-glucose modulates synaptic transmission from the central terminals of vagal afferent fibers.

  2008influential reference
• The vagus nerve, food intake and obesity.

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• Behavioral / Systems / Cognitive Presynaptic Melanocortin-4 Receptors on Vagal Afferent Fibers Modulate the Excitability of Rat Nucleus Tractus Solitarius Neurons

  2008cited by this paper
• Glucose increases synaptic transmission from vagal afferent central nerve terminals via modulation of 5-HT3 receptors.

  2008cited by this paper
• Nesfatin-1: distribution and interaction with a G protein-coupled receptor in the rat brain.

  2007cited by this paper
• Differential effects of fasting and leptin on proopiomelanocortin peptides in the arcuate nucleus and in the nucleus of the solitary tract.

  2007cited by this paper
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  2006cited by this paper
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  2004cited by this paper
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  2003influential reference
• Glucose-sensing neurons: are they physiologically relevant?

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• Convergence of pre- and postsynaptic influences on glucosensing neurons in the ventromedial hypothalamic nucleus.

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  2001cited by this paper
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• Involvement of adenosine triphosphate-sensitive K+ channels in glucose-sensing in the rat solitary tract nucleus.

  2000cited by this paper
• Localization of hindbrain glucoreceptive sites controlling food intake and blood glucose.

  2000cited by this paper
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  2000cited by this paper
• Altered energy balance causes selective changes in melanocortin-4(MC4-R), but not melanocortin-3 (MC3-R), receptors in specific hypothalamic regions: further evidence that activation of MC4-R is a physiological inhibitor of feeding.

  1999cited by this paper
• Solitary tract nucleus sensitivity to moderate changes in glucose level.

  1999cited by this paper
• Brainstem Application of Melanocortin Receptor Ligands Produces Long-Lasting Effects on Feeding and Body Weight

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• Characteristics of glycemia-sensitive neurons in the nucleus tractus solitarii: possible involvement in nutritional regulation.

  1997cited by this paper
• Rapid Changes in Local Extracellular Rat Brain Glucose Observed with an In Vivo Glucose Sensor

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• Extracellular glucose concentration in mammalian brain: continuous monitoring of changes during increased neuronal activity and upon limitation in oxygen supply in normo-, hypo-, and hyperglycemic animals

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• Cholecystokinin-gated currents in neurons of the rat solitary complex in vitro.

  1993cited by this paper
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  1992cited by this paper
• Glucose responding neurons in the nucleus tractus solitarius of the rat : in vitro study

  1985cited by this paper
• Glucose responding neurons in the nucleus tractus solitarius of the rat: in vitro study.

  1984cited by this paper
• Influence of peripheral and intracerebroventricular glucose and insulin infusions on peripheral and cerebrospinal fluid glucose and insulin levels.

  1983cited by this paper
• Printed in U.S.A. Copyright © 2000 by The Endocrine Society The Role of the Dorsal Vagal Complex and the Vagus Nerve in Feeding Effects of Melanocortin-3/4 Receptor Stimulation*

  year unknowncited by this paper

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  2019cites this paper
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  2017cites this paper
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  2017cites this paper
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