Tumor Suppressor α via the TAp 73 Netrin-1 Induces Apoptosis in Human Cervical Tumor Cells

No abstract is available for this paper.

• Publication year

  2008

• Venue

  Unknown venue

• Publication date

  Unknown publication date

• Fields of study

  Not labeled

• Identifiers

  No identifiers available.

• External record

  Open on Semantic Scholar

• Source metadata

  Semantic Scholar

• No claims are published for this paper.

• No concepts are published for this paper.

• microRNAs and death receptors.

  2008cited by this paper
• p73 poses a barrier to malignant transformation by limiting anchorage‐independent growth

  2008cited by this paper
• Yes-Associated Protein (YAP) is a critical mediator of c-Jun-dependent apoptosis

  2008cited by this paper
• Activation of the UNC5B receptor by Netrin-1 inhibits sprouting angiogenesis.

  2007cited by this paper
• Inactivation of the UNC5C Netrin-1 receptor is associated with tumor progression in colorectal malignancies.

  2007cited by this paper
• Dependence receptors: when apoptosis controls tumor progression.

  2007cited by this paper
• Making the case for DCC and UNC5C as tumor-suppressor genes in the colon.

  2007cited by this paper
• Opposing roles of netrin-1 and the dependence receptor DCC in cancer cell invasion, tumor growth and metastasis

  2007cited by this paper
• Large-scale CpG methylation analysis identifies novel candidate genes and reveals methylation hotspots in acute lymphoblastic leukemia.

  2007cited by this paper
• The adaptor Grb7 links netrin‐1 signaling to regulation of mRNA translation

  2007cited by this paper
• Regulation of p73 by Hck through kinase-dependent and independent mechanisms

  2007cited by this paper
• The Yes-associated protein 1 stabilizes p73 by preventing Itch-mediated ubiquitination of p73

  2007influential reference
• p63 mediates survival in squamous cell carcinoma by suppression of p73-dependent apoptosis.

  2006cited by this paper
• Deleted in colorectal cancer is a putative conditional tumor-suppressor gene inactivated by promoter hypermethylation in head and neck squamous cell carcinoma.

  2006cited by this paper
• Ubiquitin and Ubiquitin-Like Modifications of the p 53 Family 1

  2006cited by this paper
• p53/p63/p73 isoforms: an orchestra of isoforms to harmonise cell differentiation and response to stress

  2006cited by this paper
• Protein Interacting with C-Kinase 1/Protein Kinase Cα-Mediated Endocytosis Converts Netrin-1-Mediated Repulsion to Attraction

  2006cited by this paper
• p63 regulates an adhesion programme and cell survival in epithelial cells

  2006cited by this paper
• Netrins Promote Developmental and Therapeutic Angiogenesis

  2006cited by this paper
• UNC5A promotes neuronal apoptosis during spinal cord development independent of netrin-1

  2006cited by this paper
• DeltaTAp73 upregulation correlates with poor prognosis in human tumors: putative in vivo network involving p73 isoforms, p53, and E2F-1.

  2006cited by this paper
• FAK and Src kinases are required for netrin-induced tyrosine phosphorylation of UNC5

  2006cited by this paper
• Netrin induces down‐regulation of its receptor, Deleted in Colorectal Cancer, through the ubiquitin–proteasome pathway in the embryonic cortical neuron

  2005cited by this paper
• New antibodies recognizing p73: comparison with commercial antibodies.

  2005cited by this paper
• A mechanism of ubiquitin-independent proteasomal degradation of the tumor suppressors p53 and p73.

  2005cited by this paper
• p73 induction after DNA damage is regulated by checkpoint kinases Chk1 and Chk2.

  2004influential reference
• Focal adhesion kinase in netrin-1 signaling

  2004cited by this paper
• Phosphorylation of DCC by Fyn mediates Netrin-1 signaling in growth cone guidance

  2004cited by this paper
• Netrin-1 and its receptors in tumorigenesis

  2004cited by this paper
• Promoter methylation status of tumor suppressor and tumor-related genes in neoplastic and non-neoplastic gastric epithelia.

  2004cited by this paper
• ΔNp73 stabilises TAp73 proteins but compromises their function due to inhibitory hetero-oligomer formation

  2004cited by this paper
• The axonal attractant Netrin-1 is an angiogenic factor.

  2004cited by this paper
• Functional regulation of p73 and p63: development and cancer.

  2003cited by this paper
• Quantification of expression of netrins, slits and their receptors in human prostate tumors

  2003cited by this paper
• Recognition of the Neural Chemoattractant Netrin-1 by Integrins α6β4 and α3β1 Regulates Epithelial Cell Adhesion and Migration

  2003cited by this paper
• The netrin-1 receptor DCC promotes filopodia formation and cell spreading by activating Cdc42 and Rac1.

  2002cited by this paper
• Aberrant methylation of multiple genes and clinicopathological features in oral squamous cell carcinoma.

  2002cited by this paper
• Expression of ΔNp73 is a molecular marker for adverse outcome in neuroblastoma patients

  2002cited by this paper
• p73: Friend or foe in tumorigenesis

  2002cited by this paper
• A Subset of Tumor-Derived Mutant Forms of p53 Down-Regulate p63 and p73 through a Direct Interaction with the p53 Core Domain

  2001cited by this paper
• Human delta Np73 regulates a dominant negative feedback loop for TAp73 and p53.

  2001cited by this paper
• p63 is essential for regenerative proliferation in limb, craniofacial and epithelial development

  1999cited by this paper
• A ligand-gated association between cytoplasmic domains of UNC5 and DCC family receptors converts netrin-induced growth cone attraction to repulsion.

  1999cited by this paper
• Netrin-1: interaction with deleted in colorectal cancer (DCC) and alterations in brain tumors and neuroblastomas.

  1999cited by this paper
• Viral Oncoproteins Discriminate between p53 and the p53 Homolog p73

  1998cited by this paper
• Two New p73 Splice Variants, γ and δ, with Different Transcriptional Activity

  1998cited by this paper
• Degradation of CFTR by the ubiquitin-proteasome pathway.

  1995cited by this paper
• Expression and alternative splicing of the deleted in colorectal cancer (DCC) gene in normal and malignant tissues.

  1994cited by this paper
• Identification of a chromosome 18q gene that is altered in colorectal cancers.

  1990cited by this paper

• No citing papers are available for this paper.