Ultraviolet radiation induces release of MIA: a new mechanism for UVR-induced progression of melanoma.

No abstract is available for this paper.

• Publication year

  2004

• Venue

  International Journal of Oncology

• Publication date

  2004-07-01

• Fields of study

  Biology, Medicine, Environmental Science

• Identifiers
  DOI 10.3892/IJO.25.1.105PMID 15201995
• External record

  Open on Semantic Scholar

• Source metadata

  Semantic Scholar, PubMed

• No claims are published for this paper.

• No concepts are published for this paper.

• Proliferative activity of keratinocytes correlates with that of melanocytes in naevi and melanomas

  2004cited by this paper
• TP53 mutations in human skin cancers

  2003cited by this paper
• Active Detachment Involves Inhibition of Cell-Matrix Contacts of Malignant Melanoma Cells by Secretion of Melanoma Inhibitory Activity

  2003cited by this paper
• Expression of tyrosinase, MIA and MART‐1 in sentinel lymph nodes of patients with malignant melanoma

  2002cited by this paper
• Ink4a/arf deficiency promotes ultraviolet radiation-induced melanomagenesis.

  2002cited by this paper
• Expression, function and clinical relevance of MIA (melanoma inhibitory activity).

  2002cited by this paper
• Tyrosinase gene expression is regulated by p53.

  2002cited by this paper
• Neonatal sunburn and melanoma in mice

  2001cited by this paper
• The extracellular human melanoma inhibitory activity (MIA) protein adopts an SH3 domain‐like fold

  2001cited by this paper
• Anoikis and metastatic potential of cloudman S91 melanoma cells.

  2001cited by this paper
• Structure of melanoma inhibitory activity protein, a member of a recently identified family of secreted proteins

  2001cited by this paper
• Basic fibroblast growth factor and ultraviolet B transform melanocytes in human skin.

  2001cited by this paper
• Functional role of melanoma inhibitory activity in regulating invasion and metastasis of malignant melanoma cells in vivo

  2001cited by this paper
• Expression of activated N-ras in a primary melanoma cell line counteracts growth inhibition by transforming growth factor-beta.

  2000cited by this paper
• Characterization of a transcription factor binding site, specifically activating MIA transcription in melanoma.

  2000cited by this paper
• In situ expression patterns of melanoma‐inhibiting activity (MIA) in melanomas and breast cancers

  1999cited by this paper
• Detection of melanoma cells in the blood of melanoma patients by melanoma-inhibitory activity (MIA) reverse transcription-PCR.

  1999cited by this paper
• Melanoma-inhibitory activity protein concentrations in blood of melanoma patients treated with immunotherapy.

  1999cited by this paper
• Overexpression of mutant ras in human melanoma increases invasiveness, proliferation and anchorage-independent growth in vitro and induces tumour formation and cachexia in vivo.

  1999cited by this paper
• MIA, a novel serum marker for progression of malignant melanoma.

  1999cited by this paper
• [MIA ("melanoma inhibitory activity"). Biological functions and clinical relevance in malignant melanoma].

  1998cited by this paper
• RNA fingerprinting displays UVB-specific disruption of transcriptional control in human melanocytes.

  1997cited by this paper
• Melanoma-inhibiting activity, a novel serum marker for progression of malignant melanoma.

  1997cited by this paper
• SH3 domains and drug design: ligands, structure, and biological function.

  1997cited by this paper
• Structure and Promoter Analysis of the Gene Encoding the Human Melanoma-inhibiting Protein MIA (*)

  1996cited by this paper
• Assignment of the human melanoma inhibitory activity gene (MIA) to 19q13.32-q13.33 by fluorescence in situ hybridization (FISH).

  1996cited by this paper
• Expression of E and P-cadherin by melanoma cells decreases in progressive melanomas and following ultraviolet radiation.

  1996cited by this paper
• Cloning of a Retinoic Acid-sensitive mRNA Expressed in Cartilage and during Chondrogenesis (*)

  1996cited by this paper
• TGF-beta mediated G1 arrest in a human melanoma cell line lacking p15INK4B: evidence for cooperation between p21Cip1/WAF1 and p27Kip1.

  1996cited by this paper
• Ultraviolet B irradiation promotes tumorigenic and metastatic properties in primary cutaneous melanoma via induction of interleukin 8.

  1995cited by this paper
• UV‐Irradiated Melanocytic Nevi Simulating Melanoma In Situ

  1995cited by this paper
• Expression of the integrin alpha 4 beta 1 on melanoma cells can inhibit the invasive stage of metastasis formation.

  1994cited by this paper
• Cloning of a novel malignant melanoma-derived growth-regulatory protein, MIA.

  1994cited by this paper
• Purification and analysis of growth regulating proteins secreted by a human melanoma cell line

  1992cited by this paper
• Melanoma-inhibiting activity inhibits cell proliferation by prolongation of the S-phase and arrest of cells in the G2 compartment.

  1990cited by this paper
• Suppression of human colorectal carcinoma cell growth by wild-type p53.

  1990cited by this paper
• Autocrine tumor cell growth-inhibiting activities from human malignant melanoma.

  1989cited by this paper

• The Role of Classical and Novel Forms of Vitamin D in the Pathogenesis and Progression of Nonmelanoma Skin Cancers.

  2020cites this paper
• Phototherapy‐induced elevation of serum level of melanoma inhibitory activity

  2019cites this paper
• Relevance of Vitamin D in Melanoma Development, Progression and Therapy

  2019cites this paper
• Percepciones del docente universitario sobre la comunicación científica de libre acceso

  2019cites this paper
• Ensuring the Safety of Sunscreens, and Their Efficacy in Preventing Skin Cancers: Challenges and Controversies for Clinicians, Formulators, and Regulators

  2019cites this paper
• Evaluation of UV radiation-induced toxicity and biophysical changes in various skin cells with photo-shielding molecules.

  2015cites this paper
• MIA--a new target protein for malignant melanoma therapy.

  2013cites this paper
• HMGB1-directed drug discovery targeting cutaneous inflammatory dysregulation.

  2010cites this paper
• Regulation of human skin pigmentation in situ by repetitive UV exposure: molecular characterization of responses to UVA and/or UVB.

  2010cites this paper
• Mechanism of UV-related carcinogenesis and its contribution to nevi/melanoma.

  2007cites this paper
• Serummarker des Melanoms

  2006cites this paper
• Serummarker des malignen Melanoms

  2005cites this paper
• [Serum markers for melanoma].

  2005cites this paper