Low susceptibility of NC/Nga mice to the lipopolysaccharide-mediated lethality with d-galactosamine sensitization and the involvement of fewer natural killer T cells

No abstract is available for this paper.

• Publication year

  2012

• Venue

  Innate Immunity

• Publication date

  2012-02-01

• Fields of study

  Biology, Medicine

• Identifiers
  DOI 10.1177/1753425910390400PMID 21113080
• External record

  Open on Semantic Scholar

• Source metadata

  Semantic Scholar, PubMed

• No claims are published for this paper.

• No concepts are published for this paper.

• Sustained Exogenous Expression of Therapeutic Levels of IFN-γ Ameliorates Atopic Dermatitis in NC/Nga Mice via Th1 Polarization

  2010cited by this paper
• Oral administration of a galactooligosaccharide preparation inhibits development of atopic dermatitis-like skin lesions in NC/Nga mice.

  2010cited by this paper
• Low susceptibility of NC/Nga mice to tumor necrosis factor-alpha-mediated lethality and hepatocellular damage with D-galactosamine sensitization.

  2009cited by this paper
• The mechanism of development of acute lung injury in lethal endotoxic shock using α‐galactosylceramide sensitization

  2008cited by this paper
• Suppression of spontaneous dermatitis in NC/Nga murine model by PG102 isolated from Actinidia arguta.

  2007cited by this paper
• Mice deficient in Vβ8+NKT cells are resistant to experimental hepatitis but are partially susceptible to generalised Shwartzman reaction

  2007influential reference
• Lipopolysaccharide enhances interferon-γ-induced nitric oxide (NO) production in murine vascular endothelial cells via augmentation of interferon regulatory factor-1 activation

  2007cited by this paper
• A murine model of NKT cell-mediated liver injury induced by alpha-galactosylceramide/d-galactosamine

  2006cited by this paper
• Contribution of IL-18 to atopic-dermatitis-like skin inflammation induced by Staphylococcus aureus product in mice.

  2006cited by this paper
• Lethal endotoxic shock using alpha-galactosylceramide sensitization as a new experimental model of septic shock.

  2006cited by this paper
• A murine model of NKT cell-mediated liver injury induced by alpha-galactosylceramide/d-galactosamine

  2005cited by this paper
• Augmentation of lipopolysaccharide-induced nitric oxide production by α-galactosylceramide in mouse peritoneal cells

  2005cited by this paper
• Contribution of IL-18 to atopic-dermatitis-like skin inflammation induced by Staphylococcus aureus product in mice

  2005cited by this paper
• Vital role of the itch–scratch response in development of spontaneous dermatitis in NC/Nga mice

  2004cited by this paper
• Establishment of allergic dermatitis in NC/Nga mice as a model for severe atopic dermatitis.

  2004cited by this paper
• D-galactosamine lethality model: scope and limitations.

  2004cited by this paper
• Animal models for atopic dermatitis: are they relevant to human disease?

  2004cited by this paper
• Therapeutic effects of streptococcal preparation OK-432 on atopic dermatitis-like lesions in NC/Nga mice: possible shift from a Th2- to Th1-predominance.

  2004cited by this paper
• Review: D-Galactosamine lethality model: scope and limitations

  2004cited by this paper
• STAT1 plays an essential role in LPS/D-galactosamine-induced liver apoptosis and injury.

  2003cited by this paper
• Inability of IL-12 to Down-Regulate IgE Synthesis Due to Defective Production of IFN-γ in Atopic NC/Nga Mice1

  2001cited by this paper
• The Mechanism of a Defective IFN-γ Response to Bacterial Toxins in an Atopic Dermatitis Model, NC/Nga Mice, and the Therapeutic Effect of IFN-γ, IL-12, or IL-18 on Dermatitis1

  2001influential reference
• Cytometric and functional analyses of NK and NKT cell deficiencies in NOD mice.

  2001cited by this paper
• Inducible nitric oxide synthase is critical for immune-mediated liver injury in mice.

  2001cited by this paper
• Reduced Susceptibility of Nonobese Diabetic Mice to TNF-α and d-Galactosamine-Mediated Hepatocellular Apoptosis and Lethality1

  2000cited by this paper
• Augmentation of Nitric Oxide Production by Gamma Interferon in a Mouse Vascular Endothelial Cell Line and Its Modulation by Tumor Necrosis Factor Alpha and Lipopolysaccharide

  2000cited by this paper
• IgE hyperproduction through enhanced tyrosine phosphorylation of Janus kinase 3 in NC/Nga mice, a model for human atopic dermatitis.

  1999cited by this paper
• Expression of Fas and Fas Ligand on Mouse Renal Tubular Epithelial Cells in the Generalized Shwartzman Reaction and Its Relationship to Apoptosis

  1999cited by this paper
• Role of Nitric Oxide in Lipopolysaccharide-Induced Hepatic Injury in d-Galactosamine-Sensitized Mice as an Experimental Endotoxic Shock Model

  1999cited by this paper
• Apoptotic cell death in the response of D-galactosamine-sensitized mice to lipopolysaccharide as an experimental endotoxic shock model

  1996cited by this paper
• Interleukin 12, interferon gamma, and tumor necrosis factor alpha are the key cytokines of the generalized Shwartzman reaction

  1994cited by this paper
• Murine hepatocyte apoptosis induced in vitro and in vivo by TNF-alpha requires transcriptional arrest.

  1994cited by this paper
• Interferon gamma receptor deficient mice are resistant to endotoxic shock

  1994cited by this paper
• Interferon gamma, a mediator of lethal lipopolysaccharide-induced Shwartzman-like shock reactions in mice

  1990cited by this paper
• Analysis of nitrate, nitrite, and [15N]nitrate in biological fluids.

  1982cited by this paper

• Absence of complement terminal pathway activity in C6-deficient mice prolongs survival in a mouse model of severe malarial infection.

  2025cites this paper
• Mouse NC/Jic strain provides novel insights into host genetic factors for malaria research

  2019cites this paper
• CD1d- and MR1-Restricted T Cells in Sepsis

  2015influential citation
• Augmentation of LPS-induced vascular endothelial cell growth factor production in macrophages by transforming growth factor-β1

  2014cites this paper