Homeostasis of mitochondrial calcium is disturbed in the cerebellum but not in other brain areas during chronic hyperammonemia

No abstract is available for this paper.

• Publication year

  2014

• Venue

  Neurochemical Journal

• Publication date

  2014-06-05

• Fields of study

  Biology, Medicine

• Identifiers
  DOI 10.1134/S1819712414020056
• External record

  Open on Semantic Scholar

• Source metadata

  Semantic Scholar

• No claims are published for this paper.

• No concepts are published for this paper.

• Encapsulation of glutamine synthetase in mouse erythrocytes: a new procedure for ammonia detoxification.

  2008cited by this paper
• Molecular mechanism of acute ammonia toxicity: role of NMDA receptors.

  2002cited by this paper
• Preparation and handling of brain mitochondria useful to study uptake and release of calcium.

  2001cited by this paper
• Recent development in NMDA receptors.

  2000cited by this paper
• Glutamate-induced neuron death requires mitochondrial calcium uptake

  1998cited by this paper
• Superoxide production and antioxidant enzymes in ammonia intoxication in rats.

  1997cited by this paper
• Functional Correlation of NMDA Receptor ε Subunits Expression with the Properties of Single-Channel and Synaptic Currents in the Developing Cerebellum

  1996cited by this paper
• Mitochondria and Na+/Ca2+ exchange buffer glutamate-induced calcium loads in cultured cortical neurons

  1995cited by this paper
• Distinct distributions of five N‐methyl‐D‐aspartate receptor channel subunit mRNAs in the forebrain

  1993cited by this paper
• Molecular diversity of the NMDA receptor channel

  1992cited by this paper
• Heteromeric NMDA Receptors: Molecular and Functional Distinction of Subtypes

  1992cited by this paper
• Ammonium ingestion prevents depletion of hepatic energy metabolites induced by acute ammonium intoxication.

  1991cited by this paper
• Regulation of Ca2+ transport in brain mitochondria. I. The mechanism of spermine enhancement of Ca2+ uptake and retention.

  1990cited by this paper
• Current status of calcium in hepatocellular injury

  1989cited by this paper
• Ca2+ release from mitochondria induced by prooxidants.

  1988cited by this paper
• Pathogenesis of hepatic encephalopathy

  1987cited by this paper
• Cerebral Ammonia Metabolism in Hyperammonemic Rats

  1985cited by this paper
• Hepatic encephalopathy: current status.

  1974cited by this paper

• No citing papers are available for this paper.