Undetectable phospho‐STAT1 in peripheral blood mononuclear cells from patients with chronic hepatitis C who do not respond to interferon‐α therapy

No abstract is available for this paper.

• Publication year

  2005

• Venue

  Liver international (Print)

• Publication date

  2005-10-01

• Fields of study

  Medicine

• Identifiers
  DOI 10.1111/j.1478-3231.2005.01124.xPMID 16162158
• External record

  Open on Semantic Scholar

• Source metadata

  Semantic Scholar, PubMed

• No claims are published for this paper.

• No concepts are published for this paper.

• Host factors and failure of interferon‐α treatment in hepatitis C virus

  2004cited by this paper
• Host factors and failure of interferon-alpha treatment in hepatitis C virus.

  2004cited by this paper
• Peginterferon Alfa-2a Plus Ribavirin for Chronic Hepatitis C Virus Infection

  2003cited by this paper
• Early virologic response to treatment with peginterferon alfa‐2b plus ribavirin in patients with chronic hepatitis C

  2003cited by this paper
• Expression of interferon alfa signaling components in human alcoholic liver disease

  2002cited by this paper
• Peginterferon alfa-2a plus ribavirin for chronic hepatitis C virus infection.

  2002cited by this paper
• SOCS-1/SSI-1-deficient NKT cells participate in severe hepatitis through dysregulated cross-talk inhibition of IFN-gamma and IL-4 signaling in vivo.

  2001cited by this paper
• Hepatitis C Virus Nonstructural 5A Protein Induces Interleukin-8, Leading to Partial Inhibition of the Interferon-Induced Antiviral Response

  2001cited by this paper
• SOCS1 Is a Critical Inhibitor of Interferon γ Signaling and Prevents the Potentially Fatal Neonatal Actions of this Cytokine

  1999cited by this paper
• Expression of Hepatitis C Virus Proteins Inhibits Signal Transduction through the Jak-STAT Pathway

  1999cited by this paper
• The STAT family of proteins in cytokine signaling.

  1999cited by this paper
• Loss of STAT1 expression confers resistance to IFN-gamma-induced apoptosis in ME180 cells.

  1999cited by this paper
• Current and evolving therapies for hepatitis C.

  1999cited by this paper
• SOCS1 is a critical inhibitor of interferon gamma signaling and prevents the potentially fatal neonatal actions of this cytokine.

  1999cited by this paper
• Molecular biology and gene therapy in gastroenterology and hepatology.

  1999cited by this paper
• Loss of STAT1 expression confers resistance to IFN‐γ‐induced apoptosis in ME180 cells

  1999cited by this paper
• SOCS1 deficiency causes a lymphocyte-dependent perinatal lethality.

  1999cited by this paper
• Correlation of Interferon-Induced Expression of MxA mRNA in Peripheral Blood Mononuclear Cells with the Response of Patients with Chronic Active Hepatitis C to IFN-alpha Therapy

  1999cited by this paper
• A Janus kinase inhibitor, JAB, is an interferon-gamma-inducible gene and confers resistance to interferons.

  1998cited by this paper
• The Suppressor of Cytokine Signaling (SOCS) 1 and SOCS3 but Not SOCS2 Proteins Inhibit Interferon-mediated Antiviral and Antiproliferative Activities*

  1998cited by this paper
• STATs and gene regulation.

  1997cited by this paper
• A new protein containing an SH2 domain that inhibits JAK kinases

  1997cited by this paper
• The treatment of chronic viral hepatitis.

  1997cited by this paper
• A family of cytokine-inducible inhibitors of signalling

  1997cited by this paper
• THE TREATMENT OF CHRONIC VIRAL HEPATITIS

  1997cited by this paper
• Structure and function of a new STAT-induced STAT inhibitor

  1997cited by this paper
• Hepatitis C virus RNA in peripheral blood mononuclear cells: Comparing acute and chronic hepatitis C virus infection

  1996cited by this paper
• Targeted disruption of the Stat1 gene in mice reveals unexpected physiologic specificity in the JAK-STAT signaling pathway.

  1996cited by this paper
• Targeted disruption of the mouse Stat1 gene results in compromised innate immunity to viral disease.

  1996cited by this paper
• Meta‐analysis of interferon randomized trials in the treatment of viral hepatitis C: Effects of dose and duration

  1996cited by this paper
• Transcriptional responses to polypeptide ligands: the JAK-STAT pathway.

  1995cited by this paper
• Cytokine signaling through nonreceptor protein tyrosine kinases.

  1995cited by this paper
• Activity of the Interferon-Induced 2′5′-Oligoadenylate Synthetase in Patients with Chronic Hepatitis C

  1995cited by this paper
• Signaling through the hematopoietic cytokine receptors.

  1995cited by this paper
• Treatment of chronic viral hepatitis.

  1994cited by this paper
• Jak-STAT pathways and transcriptional activation in response to IFNs and other extracellular signaling proteins.

  1994cited by this paper
• Hepatitis C virus‐RNAs in plasma and in peripheral blood mononuclear cells of hemophiliacs with chronic hepatitis C: Evidence for viral replication in peripheral blood mononuclear cells

  1994cited by this paper
• Treatment of chronic non-A,non-B hepatitis with recombinant human alpha interferon. A preliminary report.

  1986cited by this paper
• Electrophoretically pure mouse interferon inhibits growth, induces liver and kidney lesions, and kills suckling mice.

  1981cited by this paper

• Hepatitis C virus infection

  2017cites this paper
• Association of interleukin-10 with hepatitis B virus (HBV) mediated disease progression in Indian population

  2014cites this paper
• γ-H2AX + CD8+ T lymphocytes cannot respond to IFN-α, IL-2 or IL-6 in chronic hepatitis C virus infection

  2013cites this paper
• Intracellular expression of IRF9 Stat fusion protein overcomes the defective Jak-Stat signaling and inhibits HCV RNA replication

  2010cites this paper
• MxA induction may predict sustained virologic responses of chronic hepatitis B patients with IFN-alpha treatment.

  2007cites this paper
• MxA Induction May Predict Sustained Virologic Responses of Chronic Hepatitis B Patients with IFN-α Treatment

  2007cites this paper
• Adjunctive treatment of disseminated Mycobacterium avium complex infection with interferon alpha-2b in a patient with complete interferon-gamma receptor R1 deficiency

  2007cites this paper
• A Novel Modification of a Flow Cytometric Assay of Phosphorylated STAT1 in Whole Blood Monocytes for Immunomonitoring of Patients on IFNα Regimen

  2007cites this paper