A transgenic Drosophila model for arsenic methylation suggests a metabolic rationale for differential dose-dependent toxicity endpoints.

No abstract is available for this paper.

• Publication year

  2011

• Venue

  Toxicological Sciences

• Publication date

  2011-06-01

• Fields of study

  Biology, Medicine, Chemistry, Environmental Science

• Identifiers
  DOI 10.1093/toxsci/kfr074PMID 21447609PMCID PMC3098963
• External record

  Open on Semantic Scholar

• Source metadata

  Semantic Scholar, PubMed

• No claims are published for this paper.

• No concepts are published for this paper.

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