Gene expression of TWEAK/Fn14 and IP‐10/CXCR3 in glomerulus and tubulointerstitium of patients with lupus nephritis

No abstract is available for this paper.

• Publication year

  2011

• Venue

  Nephrology

• Publication date

  2011-05-01

• Fields of study

  Medicine

• Identifiers
  DOI 10.1111/j.1440-1797.2011.01449.xPMID 21303425
• External record

  Open on Semantic Scholar

• Source metadata

  Semantic Scholar, PubMed

• No claims are published for this paper.

• No concepts are published for this paper.

• TWEAK as a target for therapy in systemic lupus erythematosus

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• TNF-like weak inducer of apoptosis (TWEAK) induces inflammatory and proliferative effects in human kidney cells.

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• Serum protein profiles in lupus nephritis associated with initial-onset systemic lupus erythematosus: Characterization through PEA immunoassay and preliminary development of predictive model.

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• The Influence of Anti-ETAR and Anti-CXCR3 Antibody Levels on the Course of Specific Glomerulonephritis Types

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• Diagnostic value of TWEAK for predicting active lupus nephritis in patients with systemic lupus erythematosus: a systematic review and meta-analysis

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• Role of Chemokine (C–X–C Motif) Ligand 10 (CXCL10) in Renal Diseases

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• Interferon-Inducible Protein 10 and Disease Activity in Systemic Lupus Erythematosus and Lupus Nephritis: A Systematic Review and Meta-Analysis

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• Deficiency of fibroblast growth factor-inducible 14 (Fn14) preserves the filtration barrier and ameliorates lupus nephritis.

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• Studies of Binding of Tumor Necrosis Factor (TNF)-like Weak Inducer of Apoptosis (TWEAK) to Fibroblast Growth Factor Inducible 14 (Fn14)*

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