COOH-terminal truncated cardiac myosin-binding protein C mutants resulting from familial hypertrophic cardiomyopathy mutations exhibit altered expression and/or incorporation in fetal rat cardiomyocytes.

No abstract is available for this paper.

• Publication year

  1999

• Venue

  Journal of Molecular Biology

• Publication date

  1999-11-26

• Fields of study

  Biology, Medicine

• Identifiers
  DOI 10.1006/JMBI.1999.3276PMID 10610770
• External record

  Open on Semantic Scholar

• Source metadata

  Semantic Scholar, PubMed

• No claims are published for this paper.

• No concepts are published for this paper.

• Mutations in beta-myosin S2 that cause familial hypertrophic cardiomyopathy (FHC) abolish the interaction with the regulatory domain of myosin-binding protein-C.

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