T cell receptor expression and differential proliferative responses by T cells specific to a myasthenogenic peptide.

No abstract is available for this paper.

• Publication year

  1997

• Venue

  Cellular Immunology

• Publication date

  1997-08-25

• Fields of study

  Biology, Medicine

• Identifiers
  DOI 10.1006/CIMM.1997.1171PMID 9316635
• External record

  Open on Semantic Scholar

• Source metadata

  Semantic Scholar, PubMed

• No claims are published for this paper.

• No concepts are published for this paper.

• Mouse T-cell receptor variable gene segment families

  2004cited by this paper
• Molecular Cloning: A Laboratory Manual

  2001cited by this paper
• Homotypic interactions mediated through LFA-1/ICAM-3 decrease the proliferative response of activated T cells.

  1996cited by this paper
• Peptide analogs to pathogenic epitopes of the human acetylcholine receptor alpha subunit as potential modulators of myasthenia gravis.

  1996cited by this paper
• Both CD4+ and CD8+ T cells are essential to induce experimental autoimmune myasthenia gravis

  1996cited by this paper
• Treatment of myasthenia gravis with anti‐CD4 antibody

  1994cited by this paper
• Processing requirements of two acetylcholine receptor derived peptides for binding to antigen presenting cells and stimulation of murine T cell lines.

  1994cited by this paper
• Dichotomy between the T and the B cell epitopes of the synthetic polypeptide (T,G)‐A–L

  1994cited by this paper
• The nicotinic acetylcholine receptor: structure and autoimmune pathology.

  1994cited by this paper
• Fine specificity of T cell lines and clones that are capable of inducing autoimmune manifestations in mice.

  1994cited by this paper
• Major histocompatibility complex class II gene disruption prevents experimental autoimmune myasthenia gravis.

  1994cited by this paper
• Regulation of antibody production by helper T cell clones in experimental autoimmune myasthenia gravis is mediated by IL-4 and antigen-specific T cell factors.

  1993cited by this paper
• Inhibition of T-cell reactivity to myasthenogenic epitopes of the human acetylcholine receptor by synthetic analogs.

  1993cited by this paper
• T cell receptor V alpha-V beta repertoire and cytokine gene expression in active multiple sclerosis lesions

  1992cited by this paper
• Spreading of T-cell autoimmunity to cryptic determinants of an autoantigen

  1992cited by this paper
• Regulation of TCR α and β gene allelic exclusion during T-cell development

  1992cited by this paper
• Regulation of TCR alpha and beta gene allelic exclusion during T-cell development.

  1992cited by this paper
• Preferential use of a T cell receptor V beta gene by acetylcholine receptor reactive T cells from myasthenia gravis-susceptible mice.

  1992cited by this paper
• Sequences of proteins of immunological interest

  1991cited by this paper
• The V beta 17+ T cell repertoire: skewed J beta usage after thymic selection; dissimilar CDR3s in CD4+ versus CD8+ cells

  1991cited by this paper
• Restricted T-cell receptor V beta gene usage by myelin basic protein-specific T-cell clones in multiple sclerosis: predominant genes vary in individuals.

  1991cited by this paper
• Conformational difference of T cell antigen receptors revealed by monoclonal antibodies to mouse V beta 5 T cell receptor for antigen determinants.

  1991cited by this paper
• Idiotype specific T-cell lines inducing experimental systemic lupus erythematosus in mice.

  1991cited by this paper
• Lack of T cell oligoclonality in enzyme‐digested synovial tissue and in synovial fluid in most patients with rheumatoid arthritis

  1991cited by this paper
• The autoimmune response of different mouse strains to T-cell epitopes of the human acetylcholine receptor alpha subunit.

  1990cited by this paper
• Inhibition of T cell proliferation specific for acetylcholine receptor epitopes related to myasthenia gravis with antibody to T cell receptor or with competitive synthetic polymers.

  1990cited by this paper
• The MHC molecule I-E is necessary but not sufficient for the clonal deletion of V beta 11-bearing T cells

  1989cited by this paper
• T cell lines selected with synthetic peptides are highly encephalitogenic in SJL/J mice.

  1989cited by this paper
• T-cell reactivity in myasthenia gravis.

  1989cited by this paper
• In vitro proliferative responses and antibody titers specific to human acetylcholine receptor synthetic peptides in patients with myasthenia gravis and relation to HLA class II genes.

  1988cited by this paper
• Regulation of antibody production by helper T cell clones in experimental autoimmune myasthenia gravis.

  1988cited by this paper
• Limited heterogeneity of T cell receptors from lymphocytes mediating autoimmune encephalomyelitis allows specific immune intervention.

  1988cited by this paper
• Myasthenia gravis.

  1988cited by this paper
• A T cell receptor Vβ segment that imparts reactivity to a class II major histocompatibility complex product

  1987cited by this paper
• Analysis of the biological functions and fine specificity of (T,G)-A--L specific T cell clones.

  1986cited by this paper
• Characterization of a murine monoclonal antibody specific for an allotypic determinant on T cell antigen receptor.

  1985cited by this paper
• Detection of autoimmune cells proliferating to myelin basic protein and selection of T cell lines that mediate experimental autoimmune encephalomyelitis (EAE) in mice.

  1983cited by this paper
• DNA sequencing with chain-terminating inhibitors.

  1977cited by this paper

• A Sensitive Method for Detecting Peptide-specific CD4+ T Cell Responses in Peripheral Blood from Patients with Myasthenia Gravis

  2017cites this paper
• Responses in vitro of peripheral blood lymphocytes from patients with myasthenia gravis to stimulation with human acetylcholine receptor alpha-chain peptides: analysis in relation to age, thymic abnormality, and ethnicity.

  2005cites this paper
• The Involvement of Myasthenogenic T-cell Epitopes and Their Analogs in the Clinical Manifestations and Treatment of Myasthenia Gravis

  1999cites this paper