The Rowett rat strain is resistant to renal fibrosis.

No abstract is available for this paper.

• Publication year

  2010

• Venue

  Nephrology, Dialysis and Transplantation

• Publication date

  2010-05-01

• Fields of study

  Biology, Medicine

• Identifiers
  DOI 10.1093/ndt/gfp689PMID 20028828
• External record

  Open on Semantic Scholar

• Source metadata

  Semantic Scholar, PubMed

• No claims are published for this paper.

• No concepts are published for this paper.

• Inhibition of nuclear factor kappa B attenuates tumour progression in an animal model of renal cell carcinoma.

  2010cited by this paper
• Susceptibility loci for murine HIV-associated nephropathy encode trans-regulators of podocyte gene expression.

  2009cited by this paper
• Nephron number determines susceptibility to renal mass reduction-induced CKD in Lewis and Fisher 344 rats: implications for development of experimentally induced chronic allograft nephropathy.

  2008cited by this paper
• Suitability of 5/6 nephrectomy (5/6NX) for the induction of interstitial renal fibrosis in rats--influence of sex, strain, and surgical procedure.

  2006cited by this paper
• Upregulation of nestin, vimentin, and desmin in rat podocytes in response to injury

  2006cited by this paper
• A Mendelian locus on chromosome 16 determines susceptibility to doxorubicin nephropathy in the mouse.

  2005cited by this paper
• NAD(P)H oxidase and uncoupled nitric oxide synthase are major sources of glomerular superoxide in rats with experimental diabetic nephropathy.

  2005cited by this paper
• Effect of aldosterone on renal transforming growth factor-beta.

  2004cited by this paper
• No aggravation of renal injury in apolipoprotein E knockout mice (ApoE(-/-)) after subtotal nephrectomy.

  2004cited by this paper
• Renal neuronal nitric oxide synthase protein expression as a marker of renal injury.

  2003cited by this paper
• Oxidative stress and dysregulation of superoxide dismutase and NADPH oxidase in renal insufficiency.

  2003cited by this paper
• ACE-inhibitors but not endothelin receptor blockers prevent podocyte loss in early diabetic nephropathy

  2003cited by this paper
• Genes expressed by the kidney, but not by bone marrow-derived cells, underlie the genetic predisposition to progressive glomerulosclerosis after mesangial injury.

  2003cited by this paper
• Podocyte Injury Promotes Progressive Nephropathy in Zucker Diabetic Fatty Rats

  2002influential reference
• Effects of low dose sympathetic inhibition on glomerulosclerosis and albuminuria in subtotally nephrectomized rats.

  2000cited by this paper
• Functional and structural correlates of glomerulosclerosis after renal mass reduction in the rat.

  2000cited by this paper
• Mutations in ACTN4, encoding alpha-actinin-4, cause familial focal segmental glomerulosclerosis.

  2000cited by this paper
• Mutations in ACTN4, encoding α-actinin-4, cause familial focal segmental glomerulosclerosis

  2000cited by this paper
• Resistance to remnant nephropathy in the Wistar-Furth rat.

  1999cited by this paper
• Pathophysiology of progressive nephropathies.

  1998cited by this paper
• Progression of glomerular diseases: is the podocyte the culprit?

  1998cited by this paper
• Adaptation and distortion of podocytes in rat remnant kidney

  1998cited by this paper
• Age-related glomerulosclerosis and interstitial fibrosis in Milan normotensive rats: a podocyte disease.

  1997influential reference
• Angiotensin II stimulates extracellular matrix protein synthesis through induction of transforming growth factor-beta expression in rat glomerular mesangial cells.

  1994cited by this paper
• Method of renal mass reduction is a critical modulator of subsequent hypertension and glomerular injury.

  1994cited by this paper
• Extracellular matrix component mRNA expression in glomeruli in experimental focal glomerulosclerosis.

  1993cited by this paper
• Glomerular damage after uninephrectomy in young rats. II. Mechanical stress on podocytes as a pathway to sclerosis.

  1992cited by this paper
• Glomerular cells, extracellular matrix accumulation, and the development of glomerulosclerosis in the remnant kidney model.

  1992cited by this paper
• Variable expression of desmin in rat glomerular epithelial cells.

  1990cited by this paper
• Is glomerulosclerosis a consequence of altered glomerular permeability to macromolecules?

  1990cited by this paper
• Glomerular hypertrophy and epithelial cell injury modulate progressive glomerulosclerosis in the rat.

  1989cited by this paper
• Microplate measurement of urinary albumin and creatinine.

  1989cited by this paper
• Interstitial nephritis induced by protein-overload proteinuria.

  1989cited by this paper
• Differences in puromycin aminonucleoside nephrosis in two rat strains.

  1988cited by this paper
• Renal disease and the development of hypertension in salt-sensitive Dahl rats.

  1988cited by this paper
• Chronic renal failure after nephrotoxic nephritis in rats: contributions to progression.

  1987cited by this paper
• Analysis of renal structural and functional features in two rat strains with a different susceptibility to glomerular sclerosis.

  1986cited by this paper
• Age-dependent glomerulosclerosis and proteinuria occurring in rats of the Milan normotensive strain and not in rats of the Milan hypertensive strain.

  1986cited by this paper
• Control of glomerular hypertension limits glomerular injury in rats with reduced renal mass.

  1985cited by this paper
• Altered glomerular permselectivity and progressive sclerosis following extreme ablation of renal mass.

  1982cited by this paper
• Dietary protein intake and the progressive nature of kidney disease: the role of hemodynamically mediated glomerular injury in the pathogenesis of progressive glomerular sclerosis in aging, renal ablation, and intrinsic renal disease.

  1982cited by this paper
• Racial differences in the incidence of treatment for end-stage renal disease.

  1982cited by this paper

• Renal Epithelial Complement C3 Expression Affects Kidney Fibrosis Progression

  2024cites this paper
• Susceptibility to kidney fibrosis in mice is associated with early growth response-2 protein and tissue inhibitor of metaloproteinase-1 expression.

  2022cites this paper
• How Acute Kidney Injury Contributes to Renal Fibrosis.

  2019cites this paper
• Role of Nitric Oxide Pathway in Development and Progression of Chronic Kidney Disease in Rats Sensitive and Resistant to its Occurrence in an Experimental Model of 5/6 Nephrectomy

  2017cites this paper
• Progression of Tubulointerstitial Fibrosis and the Chronic Kidney Disease Phenotype – Role of Risk Factors and Epigenetics

  2017cites this paper
• Oxidative/Nitrative Stress and Inflammation Drive Progression of Doxorubicin-Induced Renal Fibrosis in Rats as Revealed by Comparing a Normal and a Fibrosis-Resistant Rat Strain

  2015cites this paper
• Moderately Decreased Dietary Salt Intake Suppresses the Progression of Renal Insufficiency in Rats with 5/6 Nephrectomy

  2014cites this paper