Copper depletion increases the mitochondrial-associated SOD1 in neuronal cells

No abstract is available for this paper.

• Publication year

  2011

• Venue

  Biometals

• Publication date

  2011-04-01

• Fields of study

  Biology, Medicine, Chemistry

• Identifiers
  DOI 10.1007/s10534-010-9392-3PMID 21140194
• External record

  Open on Semantic Scholar

• Source metadata

  Semantic Scholar, PubMed

• No claims are published for this paper.

• No concepts are published for this paper.

• Altered microglial copper homeostasis in a mouse model of Alzheimer’s disease

  2010cited by this paper
• Inactivation of cytochrome c oxidase by mutant SOD1s in mouse motoneuronal NSC‐34 cells is independent from copper availability but is because of nitric oxide

  2010influential reference
• Posttranslational regulation of copper transporters

  2009cited by this paper
• Oligomerization of Mutant SOD1 in Mitochondria of Motoneuronal Cells Drives Mitochondrial Damage and Cell Toxicity

  2009cited by this paper
• Importing mitochondrial proteins: machineries and mechanisms.

  2009cited by this paper
• Dysregulation of intracellular copper trafficking pathway in a mouse model of mutant copper/zinc superoxide dismutase‐linked familial amyotrophic lateral sclerosis

  2009cited by this paper
• Metals in Alzheimer's and Parkinson's diseases.

  2008cited by this paper
• Ammonium tetrathiomolybdate delays onset, prolongs survival, and slows progression of disease in a mouse model for amyotrophic lateral sclerosis.

  2008cited by this paper
• Amyotrophic lateral sclerosis: from current developments in the laboratory to clinical implications.

  2008cited by this paper
• Selective association of misfolded ALS-linked mutant SOD1 with the cytoplasmic face of mitochondria

  2008cited by this paper
• Overexpression of CCS in G93A-SOD1 mice leads to accelerated neurological deficits with severe mitochondrial pathology

  2007cited by this paper
• Motor neuron disease associated with copper deficiency

  2006cited by this paper
• Conversion to the amyotrophic lateral sclerosis phenotype is associated with intermolecular linked insoluble aggregates of SOD1 in mitochondria.

  2006cited by this paper
• Familial ALS-superoxide dismutases associate with mitochondria and shift their redox potentials

  2006cited by this paper
• Mia40, a novel factor for protein import into the intermembrane space of mitochondria is able to bind metal ions

  2005cited by this paper
• Copper-zinc superoxide dismutase and amyotrophic lateral sclerosis.

  2005cited by this paper
• Oxygen‐induced maturation of SOD1: a key role for disulfide formation by the copper chaperone CCS

  2004cited by this paper
• Genetically Decreased Spinal Cord Copper Concentration Prolongs Life in a Transgenic Mouse Model of Amyotrophic Lateral Sclerosis

  2004cited by this paper
• Prolonged copper depletion induces expression of antioxidants and triggers apoptosis in SH-SY5Y neuroblastoma cells

  2003influential reference
• Copper Modulates the Degradation of Copper Chaperone for Cu,Zn Superoxide Dismutase by the 26 S Proteosome*

  2003cited by this paper
• Factors Controlling the Uptake of Yeast Copper/Zinc Superoxide Dismutase into Mitochondria*

  2003cited by this paper
• Metallochaperone for Cu,Zn-superoxide dismutase (CCS) protein but not mRNA is higher in organs from copper-deficient mice and rats.

  2003cited by this paper
• Mutant SOD1 causes motor neuron disease independent of copper chaperone–mediated copper loading

  2002cited by this paper
• Mutated Human SOD1 Causes Dysfunction of Oxidative Phosphorylation in Mitochondria of Transgenic Mice*

  2002influential reference
• A fraction of yeast Cu,Zn-superoxide dismutase and its metallochaperone, CCS, localize to the intermembrane space of mitochondria. A physiological role for SOD1 in guarding against mitochondrial oxidative damage.

  2001influential reference
• Function, structure, and mechanism of intracellular copper trafficking proteins.

  2001cited by this paper
• Subcellular Distribution of Superoxide Dismutases (SOD) in Rat Liver

  2001cited by this paper
• The Copper Chelator d‐Penicillamine Delays Onset of Disease and Extends Survival in a Transgenic Mouse Model of Familial Amyotrophic Lateral Sclerosis

  1997cited by this paper
• Purification of a fully metal-depleted Cu, Zn superoxide dismutase from copper-deficient rat liver

  1997cited by this paper
• Differential decrease of copper content and of copper binding to superoxide dismutase in liver, heart and brain of copper-deficient rats.

  1994cited by this paper
• Neuroblastoma × spinal cord (NSC) hybrid cell lines resemble developing motor neurons

  1992cited by this paper
• Increase of Cu,Zn-superoxide dismutase activity during differentiation of human K562 cells involves activation by copper of a constantly expressed copper-deficient protein.

  1991cited by this paper
• Copper activation of superoxide dismutase in rat erythrocytes.

  1989cited by this paper
• Effect of dietary or genetic copper deficiency on brain catecholamines, trace metals and enzymes in mice and rats.

  1982cited by this paper

• Copper homeostasis and neurodegenerative diseases

  2024cites this paper
• Copper toxicity and deficiency: the vicious cycle at the core of protein aggregation in ALS

  2024cites this paper
• Coordination chemistry of mitochondrial copper metalloenzymes: exploring implications for copper dyshomeostasis in cell death

  2023cites this paper
• Multifaceted Roles of Copper Ions in Anticancer Nanomedicine

  2023cites this paper
• Integration of various protein similarities using random forest technique to infer augmented drug-protein matrix for enhancing drug-disease association prediction

  2022cites this paper
• Copper-Dependent Kinases and Their Role in Cancer Inception, Progression and Metastasis

  2022cites this paper
• Copper Increases Brain Oxidative Stress and Enhances the Ability of 6-Hydroxydopamine to Cause Dopaminergic Degeneration in a Rat Model of Parkinson’s Disease

  2018cites this paper
• Bone morphogenetic protein signaling in vertebrate motor neurons and neuromuscular communication

  2015cites this paper
• SOD1 misplacing and mitochondrial dysfunction in amyotrophic lateral sclerosis pathogenesis

  2015cites this paper
• Adaptive Responses of Mitochondria to Mild Copper Deprivation Involve Changes in Morphology, OXPHOS Remodeling and Bioenergetics

  2014cites this paper
• Copper as a Target for Treatment of Neuroblastoma: Molecular and Cellular Mechanisms

  2013influential citation
• Characterization of Wnt/β-catenin and BMP/Smad signaling pathways in an in vitro model of amyotrophic lateral sclerosis

  2013cites this paper
• Honey Supplementation in Spontaneously Hypertensive Rats Elicits Antihypertensive Effect via Amelioration of Renal Oxidative Stress

  2012cites this paper