Bucillamine Prevents Myocardial Reperfusion Injury

No abstract is available for this paper.

• Publication year

  2001

• Venue

  Journal of Cardiovascular Pharmacology

• Publication date

  2001-12-01

• Fields of study

  Medicine

• Identifiers
  DOI 10.1097/00005344-200112000-00007PMID 11707689
• External record

  Open on Semantic Scholar

• Source metadata

  Semantic Scholar, PubMed

• No claims are published for this paper.

• No concepts are published for this paper.

• The Role of a Mitochondrial Pathway in the Induction of Apoptosis by Chemicals Extracted from Diesel Exhaust Particles1

  2000cited by this paper
• Bucillamine suppresses human Th1 cell development by a hydrogen peroxide-independent mechanism.

  2000cited by this paper
• Creatine kinase-MB enzyme elevation following successful saphenous vein graft intervention is associated with late mortality.

  1999cited by this paper
• Myocardial Infarction Adenosine as an Adjunct to Thrombolytic Therapy for Acute Myocardial Infarction

  1999cited by this paper
• Failure of N-2-mercaptopropionyl glycine to reduce myocardial infarction after 3 days of reperfusion in rabbits

  1999cited by this paper
• Pharmacology and biological efficacy of a recombinant, humanized, single-chain antibody C5 complement inhibitor in patients undergoing coronary artery bypass graft surgery with cardiopulmonary bypass.

  1999cited by this paper
• Timing of treatment for myocardial reperfusion injury.

  1999cited by this paper
• Pharmacological effects of SA96 (bucillamine) and its metabolites as immunomodulating drugs--the disulfide structure of SA-96 metabolites plays a critical role in the pharmacological action of the drug.

  1998cited by this paper
• Effect of N-acetylcysteine on myocardial infarct size following ischemia and reperfusion in dogs.

  1998cited by this paper
• Lipophilic siderophores of Mycobacterium tuberculosis prevent cardiac reperfusion injury.

  1998cited by this paper
• The Antioxidant, N-(2-mercaptopropionyl)-glycine (MPG), Does Not Reduce Myocardial Infarct Size in an Acute Canine Model of Myocardial Ischemia and Reperfusion

  1998cited by this paper
• Marked reduction in myocardial infarct size due to prolonged infusion of an antioxidant during reperfusion.

  1994cited by this paper
• Hydrogen peroxide cytotoxicity in cultured cardiac myocytes is iron dependent.

  1994cited by this paper
• Permeability properties of rat renal lysosomes.

  1994cited by this paper
• Protective effect of N-acetylcysteine on ischaemia-induced myocardial damage in canine heart

  1991cited by this paper
• The effects of bucillamine on glutathione and glutathione-related enzymes in the mouse.

  1991cited by this paper
• Inflammation in the course of early myocardial ischemia

  1991cited by this paper
• Cardioprotective effects of N-acetylcysteine: the reduction in the extent of infarction and occurrence of reperfusion arrhythmias in the dog.

  1990cited by this paper
• Deferoxamine pretreatment reduces canine infarct size and oxidative injury.

  1990cited by this paper
• Neutrophil adherence to isolated adult canine myocytes. Evidence for a CD18-dependent mechanism.

  1990cited by this paper
• Marked Reduction of Free Radical Generation and Contractile Dysfunction by Antioxidant Therapy Begun at the Time of Reperfusion Evidence That Myocardial "Stunning" Is a Manifestation of Reperfusion Injury

  1989cited by this paper
• Early treatment with deferoxamine limits myocardial ischemic/reperfusion injury.

  1989cited by this paper
• Superoxide dismutase reduces reperfusion arrhythmias but fails to salvage regional function or myocardium at risk in conscious dogs.

  1989cited by this paper
• Glutathione redox pathway and reperfusion injury. Effect of N-acetylcysteine on infarct size and ventricular function.

  1988cited by this paper
• Therapy to reduce free radicals during early reperfusion does not limit the size of myocardial infarcts caused by 90 minutes of ischemia in dogs.

  1988cited by this paper
• The role of glutathione status in the protection against ischaemic and reperfusion damage: effects of N-acetyl cysteine.

  1988cited by this paper
• Failure of superoxide dismutase to limit size of myocardial infarction after 40 minutes of ischemia and 4 days of reperfusion in dogs.

  1987cited by this paper
• Canine Myocardial Reperfusion Injury: Protection by a Free Radical Scavenger, N‐2‐Mcrcaptopropionyl Glycine

  1986cited by this paper
• Protective effects of N-2-mercaptopropionyl glycine against myocardial reperfusion injury after neutrophil depletion in the dog: evidence for the role of intracellular-derived free radicals.

  1986cited by this paper
• Animal Models for Protecting Ischemic Myocardium: Results of the NHLBI Cooperative Study Comparison of Unconscious and Conscious Dog Models

  1985influential reference
• Canine Myocardial Reperfusion Injury: Its Reduction by the Combined Administration of Superoxide Dismutase and Catalase

  1984cited by this paper
• Respiratory burst enzyme in human neutrophils. Evidence for multiple mechanisms of activation.

  1981cited by this paper
• Role of oxygen in the cellular damage induced by re-oxygenation of hypoxic heart.

  1980cited by this paper
• Ultrastructural damage associated with reoxygenation of the anoxic myocardium.

  1975cited by this paper
• The mitochondrial generation of hydrogen peroxide. General properties and effect of hyperbaric oxygen.

  1973cited by this paper

• Exploring antiviral and anti-inflammatory effects of thiol drugs in COVID-19

  2022cites this paper
• Bucillamine Inhibits UVB‐Induced MAPK Activation and Apoptosis in Human HaCaT Keratinocytes and SKH‐1 Hairless Mouse Skin

  2020cites this paper
• Thiol drugs decrease SARS-CoV-2 lung injury in vivo and disrupt SARS-CoV-2 spike complex binding to ACE2 in vitro

  2020cites this paper
• TREATMENT OF GOUT

  2017cites this paper
• Bucillamine prevents cisplatin-induced ototoxicity through induction of glutathione and antioxidant genes

  2015cites this paper
• Curcumin protects against ischemia/reperfusion injury in rat skeletal muscle.

  2013cites this paper
• Particles in a Murine Model for Ovalbumin Effects of Aerosolized Diesel Exhaust Thiol Antioxidants Inhibit the Adjuvant

  2013cites this paper
• Protective effects of N-(2-mercaptopropionyl)-glycine against ischemia-reperfusion injury in hypertrophied hearts.

  2013cites this paper
• Bucillamine improves hepatic microcirculation and reduces hepatocellular injury after liver warm ischaemia-reperfusion injury.

  2009cites this paper
• Photoprotective Effects of Bucillamine Against UV‐induced Damage in an SKH‐1 Hairless Mouse Model †

  2008cites this paper
• Prolonged administration of a dithiol antioxidant protects against ventricular remodeling due to ischemia-reperfusion in mice.

  2008influential citation
• Bucillamine: a potent thiol donor with multiple clinical applications.

  2006cites this paper
• Anti-oxidant and pro-oxidant behaviour of bucillamine.

  2006cites this paper
• The role of thiols in liver ischemia-reperfusion injury.

  2006cites this paper
• Bucillamine induces glutathione biosynthesis via activation of the transcription factor Nrf2.

  2006cites this paper
• Oxidative stress in organ preservation: a multifaceted approach to cardioplegia.

  2005cites this paper
• Pharmacological therapy for myocardial reperfusion injury.

  2004cites this paper
• Clinical course of bucillamine-induced nephropathy in patients with rheumatoid arthritis

  2003cites this paper
• Thiol Antioxidants Inhibit the Adjuvant Effects of Aerosolized Diesel Exhaust Particles in a Murine Model for Ovalbumin Sensitization1

  2002cites this paper
• Bucillamine, a thiol antioxidant, prevents transplantation-associated reperfusion injury

  2002cites this paper