[Protective effects of a delta-opioid-receptor agonist and an oxygen radical scavenger on postischemic hearts].

No abstract is available for this paper.

• Publication year

  2004

• Venue

  Herz

• Publication date

  2004-05-01

• Fields of study

  Medicine

• Identifiers
  DOI 10.1007/S00059-004-2576-2PMID 15167961
• External record

  Open on Semantic Scholar

• Source metadata

  Semantic Scholar, PubMed

• No claims are published for this paper.

• No concepts are published for this paper.

• Quantification of [Ca2']i in Perfused Hearts Critical Evaluation of the 5F-BAPTA and Nuclear Magnetic Resonance Method as Applied to the Study of Ischemia and Reperfusion

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  2003cited by this paper
• K(ATP) opener-induced delayed cardioprotection: involvement of sarcolemmal and mitochondrial K(ATP) channels, free radicals and MEK1/2.

  2003cited by this paper
• Effect of aspirin on late preconditioning against myocardial stunning in conscious rabbits.

  2003cited by this paper
• [The isolated rabbit heart: comparison between five different modifications].

  2002cited by this paper
• Role of nitric-oxide synthase, free radicals, and protein kinase C delta in opioid-induced cardioprotection.

  2002cited by this paper
• Ischemic preconditioning. Experimental facts and clinical perspective.

  2002cited by this paper
• Mannitol in cardioplegia as an oxygen free radical scavenger measured by malondialdehyde

  2002cited by this paper
• Oxygen wastage of stunned myocardium in vivo is due to an increased oxygen cost of contractility and a decreased myofibrillar efficiency.

  2001cited by this paper
• Diazoxide-Induced Cardioprotection Requires Signaling Through a Redox-Sensitive Mechanism

  2001cited by this paper
• Do mitochondrial KATP channels serve as triggers rather than end-effectors of ischemic preconditioning's protection?

  2000cited by this paper
• Hibernating myocardium, stunning, ischemic preconditioning: clinical relevance.

  2000cited by this paper
• Opening of Mitochondrial KATP Channels Triggers the Preconditioned State by Generating Free Radicals

  2000cited by this paper
• The effects of mannitol , albumin , and cardioplegia enhancers on 24h rat heart preservation

  1999influential reference
• Direct preconditioning of cardiac myocytes via opioid receptors and KATP channels.

  1999cited by this paper
• Role of reactive oxygen species in acetylcholine-induced preconditioning in cardiomyocytes.

  1999influential reference
• Opiate Drugs and δ-Receptor–Mediated Myocardial Protection

  1999cited by this paper
• Opening of potassium channels: the common cardioprotective link between preconditioning and natural hibernation?

  1999cited by this paper
• Molecular and cellular mechanisms of myocardial stunning.

  1999influential reference
• TAN-67, a δ1-opioid receptor agonist, reduces infarct size via activation of Gi/o proteins and KATP channels.

  1998cited by this paper
• Ischemic Preconditioning in the Intact Rat Heart Is Mediated by δ1- But Not μ- or κ-Opioid Receptors

  1998cited by this paper
• Functional characterization of cardiac myosin isoforms.

  1998influential reference
• Ischemic preconditioning in the intact rat heart is mediated by delta1- but not mu- or kappa-opioid receptors.

  1998cited by this paper
• Energy-dependent transport of calcium to the extracellular space during acute ischemia of the rat heart.

  1998cited by this paper
• Opioid receptor contributes to ischemic preconditioning through protein kinase C activation in rabbits

  1998cited by this paper
• Why myocardial stunning is clinically important

  1998cited by this paper
• The protective effect of late preconditioning against myocardial stunning in conscious rabbits is mediated by nitric oxide synthase. Evidence that nitric oxide acts both as a trigger and as a mediator of the late phase of ischemic preconditioning.

  1997cited by this paper
• Cardioprotective effect of diazoxide and its interaction with mitochondrial ATP-sensitive K+ channels. Possible mechanism of cardioprotection.

  1997cited by this paper
• The use of hibernation induction triggers for cardiac transplant preservation.

  1997influential reference
• Haemodynamic and energetic properties of stunned myocardium in rabbit hearts.

  1996cited by this paper
• Preconditioning with potassium channel openers. A new concept for enhancing cardioplegic protection?

  1995cited by this paper
• Myocardial 'stunning' in man.

  1992cited by this paper
• Identification of major differences in postischemic myocardial “stunning” and free radical generation between open-chest and conscious dogs

  1992influential reference
• Cellular mechanisms of myocardial stunning.

  1992cited by this paper
• [Hibernation, stunning, ischemic preconditioning--new paradigms in coronary disease?].

  1992cited by this paper
• Clinical relevance of myocardial “stunning”

  1991cited by this paper
• Relationship between total mechanical energy and oxygen consumption in the stunned myocardium.

  1990cited by this paper
• Iron-mediated radical reactions upon reperfusion contribute to myocardial "stunning".

  1990cited by this paper
• Effects of drugs interacting with opioid receptors during normal perfusion or ischemia and reperfusion in the isolated rat heart--an attempt to identify cardiac opioid receptor subtype(s) involved in arrhythmogenesis.

  1990cited by this paper
• Temporary contractile blockade prevents hypercontracture in anoxic-reoxygenated cardiomyocytes.

  1990influential reference
• Prolonged impairment of coronary vasodilation after reversible ischemia. Evidence for microvascular "stunning".

  1990influential reference
• Marked Reduction of Free Radical Generation and Contractile Dysfunction by Antioxidant Therapy Begun at the Time of Reperfusion Evidence That Myocardial "Stunning" Is a Manifestation of Reperfusion Injury

  1989cited by this paper
• Reduced aerobic metabolic efficiency in globally "stunned" myocardium.

  1989cited by this paper
• Oxygen Consumption and Coronary Reactivity in Postischemic Myocardium

  1989cited by this paper
• Relationships between pre-ischemic ATP and glycogen content and post-ischemic recovery of rat heart.

  1988cited by this paper
• Demonstration of free radical generation in the “stunned” myocardium in the intact dog

  1988cited by this paper
• Demonstration of free radical generation in "stunned" myocardium of intact dogs with the use of the spin trap alpha-phenyl N-tert-butyl nitrone.

  1988cited by this paper
• Pathophysiology and pathogenesis of stunned myocardium. Depressed Ca2+ activation of contraction as a consequence of reperfusion-induced cellular calcium overload in ferret hearts.

  1987cited by this paper
• Pathophysiology and pathogenesis of stunned myocardium : depressed Ca[2+] activation of contraction as a consequence of reperfusion-induced cellular calcium overload in ferret hearts

  1987cited by this paper
• Reduction in experimental infarct size by recombinant human superoxide dismutase: insights into the pathophysiology of reperfusion injury.

  1986cited by this paper
• Protective effects of N-2-mercaptopropionyl glycine against myocardial reperfusion injury after neutrophil depletion in the dog: evidence for the role of intracellular-derived free radicals.

  1986cited by this paper
• Preservation of myocardial function with mannitol reperfusate.

  1985cited by this paper
• The mechanism of mannitol in reducing ischemic injury: hyperosmolarity or hydroxyl scavenger?

  1984cited by this paper
• Reduction of myocardial ischemic injury with oxygen-derived free radical scavengers.

  1983cited by this paper
• Work as a Correlate of Canine Left Ventricular Oxygen Consumption, and the Problem of Catecholamine Oxygen Wasting

  1982cited by this paper
• Hyperosmolar reperfusion following ischemic cardiac arrest: Critical importance of the timing of mannitol administration on preservation of myocardial structure and function.

  1981cited by this paper
• Scintigraphic and hemodynamic demonstration of transient left ventricular dysfunction immediately after uncomplicated coronary artery bypass grafting.

  1979cited by this paper
• Ischemic contracture of the myocardium: mechanisms and prevention.

  1977influential reference
• Ischemic contracture of the left ventricle. Production and prevention.

  1975cited by this paper
• Reactive hyperaemia in the dog heart: studies on mechanisms.

  1974cited by this paper
• The "stone heart": a challenge to the biochemist.

  1972cited by this paper

• Metformin ameliorates hypoxia/reoxygenation-induced cardiomyocyte apoptosis based on the SIRT3 signaling pathway.

  2017cites this paper
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  2016cites this paper
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  2016cites this paper
• Mitochondrial permeability transition triggers the release of mtDNA fragments

  2004cites this paper