Ankylosing spondylitis, IgA, and transforming growth factors.

No abstract is available for this paper.

• Publication year

  1995

• Venue

  Annals of the Rheumatic Diseases

• Publication date

  1995-07-01

• Fields of study

  Medicine

• Identifiers
  DOI 10.1136/ard.54.7.544PMID 7668896PMCID PMC1009930
• External record

  Open on Semantic Scholar

• Source metadata

  Semantic Scholar, PubMed

• No claims are published for this paper.

• No concepts are published for this paper.

• Chemical reactivity of an HLA‐B27 thiol group

  1993cited by this paper
• HLA-B27-restricted CD8 T cells derived from synovial fluids of patients with reactive arthritis and ankylosing spondylitis.

  1993cited by this paper
• A hypothesis for the HLA-B27 immune dysregulation in spondyloarthropathy: contributions from enteric organisms, B27 structure, peptides bound by B27, and convergent evolution.

  1993cited by this paper
• Serum IgA in ankylosing spondylitis: biological vs statistical significances of experimental results.

  1993cited by this paper
• Differential activation of CD8+ T cells by transforming growth factor-beta 1.

  1993cited by this paper
• An anergic, islet-infiltrating T-cell clone that suppresses murine diabetes secretes a factor that blocks interleukin 2/interleukin 4-dependent proliferation.

  1992cited by this paper
• Transforming growth factor-beta directs IgA switching in human B cells.

  1992cited by this paper
• Cloning and expression of class I major histocompatibility complex genes of the rat

  1992cited by this paper
• Molecular heterogeneity of human IgA antibodies during an immune response

  1992cited by this paper
• Bone marrow IgA and IgA subclass synthesis in ankylosing spondylitis.

  1992cited by this paper
• Interleukin 10 and transforming growth factor beta cooperate to induce anti-CD40-activated naive human B cells to secrete immunoglobulin A

  1992cited by this paper
• Increased levels of serum IgA as IgA1 monomers in ankylosing spondylitis.

  1992cited by this paper
• Suppressor T cells generated by oral tolerization to myelin basic protein suppress both in vitro and in vivo immune responses by the release of transforming growth factor beta after antigen-specific triggering.

  1992cited by this paper
• A protective NOD islet‐infiltrating CD8+ T cell clone, I.S. 2.15, has in vitro immunosuppressive properties

  1992cited by this paper
• Identification and activation of latent transforming growth factor beta.

  1991cited by this paper
• Differing lymphokine profiles of functional subsets of human CD4 and CD8 T cell clones.

  1991cited by this paper
• Ankylosing spondylitis and selective IgA deficiency.

  1991cited by this paper
• Nature of the ossicular joints and their involvement in rheumatoid arthritis.

  1991cited by this paper
• Effects of transforming growth factor beta-1 and interleukin-5 on IgA isotype switching at the clonal level

  1991cited by this paper
• Transforming growth factor-beta and remodeling of bone.

  1991cited by this paper
• Selective IgA deficiency and spondyloarthropathy: a distinct disease?

  1990cited by this paper
• Spontaneous inflammatory disease in transgenic rats expressing HLA-B27 and human beta 2m: an animal model of HLA-B27-associated human disorders.

  1990cited by this paper
• Follow-up study of ankylosing spondylitis over a period of 12 years (1977-1989).

  1990cited by this paper
• Alloreactive murine CD8+ T cell clones secrete the Th1 pattern of cytokines.

  1990cited by this paper
• Transforming growth factor-beta in the regulation of fracture repair.

  1990cited by this paper
• IgA and rheumatoid factor in ankylosing spondylitis.

  1988cited by this paper
• Immunoglobulin synthesis by peripheral blood lymphocytes in patients with ankylosing spondylitis.

  1988cited by this paper
• Interleukin 5 enhances interleukin 4‐induced IgE production by normal human B cells. The role of soluble CD23 antigen

  1988cited by this paper
• Isolation and characterization of lymphokine cDNA clones encoding mouse and human IgA-enhancing factor and eosinophil colony-stimulating factor activities: relationship to interleukin 5.

  1987cited by this paper
• [Erythrocyte sedimentation rate and serum immunoglobulins in rheumatic pelvispondylitis].

  1987cited by this paper
• IgA serum levels and disease activity in ankylosing spondylitis: a prospective study.

  1985cited by this paper
• Human transforming growth factor-β complementary DNA sequence and expression in normal and transformed cells

  1985cited by this paper
• Ankylosing spondylitis, selective IgA deficiency and systemic amyloidosis with intrasynovial amyloid deposition.

  1983cited by this paper
• Histopathology of the synovial membrane of peripheral joints in ankylosing spondylitis.

  1982cited by this paper
• Ankylosing spondylitis with selective IgA deficiency and a circulating anticoagulant.

  1977cited by this paper
• Differentiation of inflammation and ossification in spondylarthritis ankylopoietica.

  1971cited by this paper
• [Enteropathy and selective IgA deficiency. Apropos of 3 further cases].

  1971cited by this paper

• Circulating Retinol-Binding Protein 4 as a Possible Biomarker of Treatment Response for Ankylosing Spondylitis: An Array-Based Comparative Study

  2020cites this paper
• Upregulation of transforming growth factor-B1 gene in ankylosing spondylitis patients

  2017cites this paper
• Osteomalacia with Looser zones caused by celiac disease

  2017cites this paper
• The synovio-entheseal complex in enthesoarthritis

  2016cites this paper
• The synovio-entheseal complex in enthesoarthritis

  2015cites this paper
• Susceptibility to ankylosing spondylitis: evidence for the role of ERAP1, TGFb1 and TLR9 gene polymorphisms

  2012cites this paper
• Dendritic cell expression of A20 preserves immune homeostasis and prevents colitis and spondyloarthritis

  2011cites this paper
• Existe-il une dissociation entre inflammation et ossification dans la spondylarthrite ?

  2008cites this paper
• Are inflammation and ossification on separate tracks in ankylosing spondylitis?

  2008cites this paper
• ) gene polymorphisms TGFB1 factor ß1 ( growth evidence for the involvement of transforming Susceptibility to ankylosing spondylitis: no

  2007cites this paper
• Non-major-histocompatibility-complex genetics of ankylosing spondylitis.

  2006cites this paper
• The genetic basis of ankylosing spondylitis

  2006cites this paper
• Transforming growth factor beta-1 and gene polymorphisms in oriental ankylosing spondylitis.

  2005cites this paper
• Susceptibility to ankylosing spondylitis: no evidence for the involvement of transforming growth factor β1 (TGFB1) gene polymorphisms

  2005cites this paper
• The effect of transforming growth factor beta1 gene polymorphisms in ankylosing spondylitis.

  2004cites this paper
• The effect of transforming growth factor beta1 gene polymorphisms in ankylosing spondylitis.

  2004cites this paper
• Widespread Scleredema Accompanied with a Monoclonal Gammopathy in a Patient with Advanced Ankylosing Spondylitis

  2004cites this paper
• Genetic susceptibility to ankylosing spondylitis.

  2004cites this paper
• DISEASE STAGING: Clinical Criteria

  2003cites this paper
• Investigating genetic determinants of ankylosing spondylitis.

  2003cites this paper
• Pathways of cell activation in spondyloarthropathies

  2001cites this paper
• Ankylosing spondylitis: time to focus on ankylosis.

  1999cites this paper
• Inflammatory disease in HLA‐B27 transgenic rats

  1999cites this paper