Romk 1 Knockout Mice Do Not Produce Bartter Phenotype but Exhibit Impaired

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  2016

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• Regulation of glomerulotubular balance: II: impact of angiotensin II on flow-dependent transport.

  2012cited by this paper
• Renal outer medullary potassium channel knockout models reveal thick ascending limb function and dysfunction

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• Female ROMK null mice manifest more severe Bartter II phenotype on renal function and higher PGE2 production.

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• Protein tyrosine kinase is expressed and regulates ROMK1 location in the cortical collecting duct.

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• Dietary potassium restriction stimulates endocytosis of ROMK channel in rat cortical collecting duct.

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• Evidence for endocytosis of ROMK potassium channel via clathrin-coated vesicles.

  2002cited by this paper
• Impaired Renal NaCl Absorption in Mice Lacking the ROMK Potassium Channel, a Model for Type II Bartter's Syndrome*

  2002cited by this paper
• Absence of Small Conductance K+ Channel (SK) Activity in Apical Membranes of Thick Ascending Limb and Cortical Collecting Duct in ROMK (Bartter's) Knockout Mice*

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• Inhibition of Protein-tyrosine Phosphatase Stimulates the Dynamin-dependent Endocytosis of ROMK1*

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• Extracellular Atp Inhibits the Small-Conductance K Channel on the Apical Membrane of the Cortical Collecting Duct from Mouse Kidney

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• Splicing of a retained intron within ROMK K+ channel RNA generates a novel set of isoforms in rat kidney.

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• MICROPUNCTURE STUDY OF RENAL POTASSIUM EXCRETION IN THE RAT.

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• No citing papers are available for this paper.