Prolonged survival of class II transactivator-deficient cardiac allografts

No abstract is available for this paper.

• Publication year

  2002

• Venue

  Transplantation

• Publication date

  2002-11-15

• Fields of study

  Biology, Medicine

• Identifiers
  DOI 10.1097/00007890-200211150-00024PMID 12451276
• External record

  Open on Semantic Scholar

• Source metadata

  Semantic Scholar, PubMed

• No claims are published for this paper.

• No concepts are published for this paper.

• Transforming growth factor-beta in T-cell biology.

  2002cited by this paper
• Comparison of Abeta(b-/-), H2-DM(-), and CIITA(-/-) in second-set skin allograft rejection.

  2002cited by this paper
• Comparison of Ab−/−β, H2-DM−, and CIITA−/− in Second-Set Skin Allograft Rejection

  2002cited by this paper
• Genetic control of MHC class II expression.

  2002cited by this paper
• Maturation of Dendritic Cells Is Accompanied by Rapid Transcriptional Silencing of Class II Transactivator (Ciita) Expression

  2001cited by this paper
• Multiple Paths for Activation of Naive CD8+ T Cells: CD4-Independent Help1

  2001cited by this paper
• Cutting Edge: The Class II Transactivator Prevents Activation-Induced Cell Death by Inhibiting Fas Ligand Gene Expression1

  2001influential reference
• The bare lymphocyte syndrome and the regulation of MHC expression.

  2001cited by this paper
• Differential Susceptibility of Heart, Skin, and Islet Allografts to T Cell-Mediated Rejection1

  2001cited by this paper
• A 36-Amino-Acid Region of CIITA Is an Effective Inhibitor of CBP: Novel Mechanism of Gamma Interferon-Mediated Suppression of Collagen α2(I) and Other Promoters

  2001cited by this paper
• Class II Transactivator: Mastering the Art of Major Histocompatibility Complex Expression

  2000cited by this paper
• Transcriptional Scaffold: CIITA Interacts with NF-Y, RFX, and CREB To Cause Stereospecific Regulation of the Class II Major Histocompatibility Complex Promoter

  2000cited by this paper
• CD4 T cell-mediated cardiac allograft rejection requires donor but not host MHC class II.

  2000cited by this paper
• H2-DMα−/− Mice Show the Importance of Major Histocompatibility Complex–Bound Peptide in Cardiac Allograft Rejection

  2000cited by this paper
• The role of T cell apoptosis in transplantation tolerance.

  2000cited by this paper
• Transcriptional regulation of the MHC class Ib genes HLA-E, HLA-F, and HLA-G.

  2000cited by this paper
• TGF-beta1: immunosuppressant and viability factor for T lymphocytes.

  1999cited by this paper
• Inducible nitric oxide synthase promotes cytokine expression in cardiac allografts but is not required for efficient rejection.

  1999cited by this paper
• Reduced IL-4-, lipopolysaccharide-, and IFN-gamma-induced MHC class II expression in mice lacking class II transactivator due to targeted deletion of the GTP-binding domain.

  1999cited by this paper
• TGF-β1: immunosuppressant and viability factor for T lymphocytes

  1999cited by this paper
• Direct and indirect recognition: the role of MHC antigens in graft rejection.

  1999cited by this paper
• Pancreatic infiltration but not diabetes occurs in the relative absence of MHC class II-restricted CD4 T cells: studies using NOD/CIITA-deficient mice.

  1999cited by this paper
• The alloresponse.

  1999cited by this paper
• Pattern of graft- and host-specific MHC class II expression in long-term murine cardiac allografts: origin of inflammatory and vascular wall cells.

  1998cited by this paper
• Mice lacking the transcription factor CIITA--a second look.

  1998influential reference
• Class II transactivator-independent endothelial cell MHC class II gene activation induced by lymphocyte adhesion.

  1998cited by this paper
• Induction of MHC class I expression by the MHC class II transactivator CIITA.

  1997cited by this paper
• Impact of donor MHC class I or class II antigen deficiency on first‐ and second‐set rejection of mouse heart or liver allografts

  1996cited by this paper
• Mice lacking the MHC class II transactivator (CIITA) show tissue-specific impairment of MHC class II expression.

  1996cited by this paper
• CD4+ but not CD8+ cells are essential for allorejection

  1996cited by this paper
• Comparative study of the role of professional versus semiprofessional or nonprofessional antigen presenting cells in the rejection of vascularized organ allografts.

  1995cited by this paper
• The two novel MHC class II transactivators RFX5 and CIITA both control expression of HLA-DM genes.

  1995cited by this paper
• Survival of MHC deficient mouse heterotopic cardiac allografts and xenografts.

  1995cited by this paper
• Class II transactivator regulates the expression of multiple genes involved in antigen presentation

  1995cited by this paper
• Immunopathology of cardiac transplant rejection

  1995cited by this paper
• Molecular analysis of G1B and G3A IFN gamma mutants reveals that defects in CIITA or RFX result in defective class II MHC and Ii gene induction.

  1994cited by this paper
• PROLONGATION OF CARDIAC ALLOGRAFT SURVIVAL WITH TRANSFORMING GROWTH FACTOR-β1 IN RATS

  1994cited by this paper
• Prolongation of cardiac allograft survival with transforming growth factor-beta 1 in rats.

  1994cited by this paper
• Complementation cloning of an MHC class II transactivator mutated in hereditary MHC class II deficiency (or bare lymphocyte syndrome).

  1993cited by this paper
• The role of "indirect" recognition in initiating rejection of skin grafts from major histocompatibility complex class II-deficient mice.

  1993cited by this paper
• Mice lacking major histocompatibility complex class I and class II molecules.

  1993cited by this paper
• The JAM test. A simple assay for DNA fragmentation and cell death.

  1991cited by this paper
• Depletion of CD4+ T cells in major histocompatibility complex class II-deficient mice

  1991cited by this paper
• The influence of MHC and non-MHC genes on the nature of murine cardiac allograft rejection. I. Kinetic analysis of mononuclear cell infiltrate and MHC-class I/class II expression in donor tissue.

  1990cited by this paper
• THE ROLE OF CLASS I AND CLASS II MHC ANTIGENS IN THE REJECTION OF VASCULARIZED HEART ALLOGRAFTS IN MICE

  1987cited by this paper
• PRIMARILY VASCULARIZED ALLOGRAFTS OF HEARTS IN MICE: THE ROLE OF H‐2D, H‐2K, AND NON‐H-2 ANTIGENS IN REJECTION

  1973cited by this paper

• Class II transactivator knockdown limits major histocompatibility complex II expression, diminishes immune rejection, and improves survival of allogeneic bone marrow stem cells in the infarcted heart

  2016cites this paper
• Attenuation of allograft rejection by intragraft inhibition of class II transcativator in high responder rat liver transplantation

  2015cites this paper
• Downregulating immunogenicity of Schwann cells via inhibiting a potential target of class II transactivator (CIITA) gene.

  2013cites this paper
• The 'Mighty Mouse' Model in experimental cardiac transplantation

  2011cites this paper
• Inhibitory effects of anti-CII TA M1-RNA on IFN-gamma induced major histocompatibility complex class II antigens expression on cultured human chondrocytes.

  2007cites this paper
• CCL19-IgG prevents allograft rejection by impairment of immune cell trafficking.

  2006cites this paper
• Graft rejection mediated by CD4+ T cells via indirect recognition of alloantigen is associated with a dominant Th2 response

  2005cites this paper
• Class II histone deacetylases confer signal responsiveness to the ankyrin-repeat proteins ANKRA2 and RFXANK.

  2005cites this paper
• Inhibition of alloresponse by a human trophoblast non-coding RNA suppressing class II transactivator promoter III and major histocompatibility class II expression in murine B-lymphocytes.

  2004cites this paper
• Differential Usage of Class II Transactivator Promoters PI and PIV during Inflammation and Injury in Kidney

  2003cites this paper
• CIITA-regulated plexin-A1 affects T-cell–dendritic cell interactions

  2003cites this paper
• Differential usage of class II transactivator promoters PI and PIV during inflammation and injury in kidney.

  2003cites this paper
• Repression of allo-cell transplant rejection through CIITA ribonuclease P+ hepatocyte.

  2003cites this paper