Protein Kinase Cα Mediates the Effect of Antiarrhythmic Peptide on Gap Junction Conductance

No abstract is available for this paper.

• Publication year

  2001

• Venue

  Cell Communication & Adhesion

• Publication date

  2001-01-01

• Fields of study

  Medicine, Chemistry

• Identifiers
  DOI 10.3109/15419060109080734PMID 12064599
• External record

  Open on Semantic Scholar

• Source metadata

  Semantic Scholar, PubMed

• No claims are published for this paper.

• No concepts are published for this paper.

• regulation cardiac

  2013cited by this paper
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• The potential for isoenzyme‐selective modulation of protein kinase C

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• Selective monoclonal antibody recognition and cellular localization of an unphosphorylated form of connexin43.

  1997cited by this paper
• Increase in gap junction conductance by an antiarrhythmic peptide.

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• Regulation of connexin43 function by activated tyrosine protein kinases

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• Cell Surface Receptors: A Short Course on Theory and Methods

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• Regulation of cardiac gap junction channel permeability and conductance by several phosphorylating conditions

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• A new synthetic antiarrhythmic peptide reduces dispersion of epicardial activation recovery interval and diminishes alterations of epicardial activation patterns induced by regional ischemia

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• New hexahydroxybiphenyl derivatives as inhibitors of protein kinase C.

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• 心臓の研究(第22報)心臓ペプタイド(AAP)の薬物誘発不整脈に対する抑制作用

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• Effects of Yunaconitine on Calcium Homeostasis in Arrhythmia Induced by Aconitum vilmorinianum Kom in Cardiomyocytes (H9C2 Cells)

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• The Role of Gap Junctions in Impulse Propagation in the Heart: New Aspects of Arrhythmogenesis and New Antiarrhythmic Agents Targeting Gap Junctions

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• Reference List

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