Expression of the recombination activating genes (RAG1 and RAG2) is not detectable in Epstein‐Barr virus‐associated human lymphomas

No abstract is available for this paper.

• Publication year

  2001

• Venue

  International Journal of Cancer

• Publication date

  2001-04-01

• Fields of study

  Biology, Medicine

• Identifiers
  DOI 10.1002/1097-0215(200102)9999:9999<::AID-IJC1163>3.0.CO;2-MPMID 11279609
• External record

  Open on Semantic Scholar

• Source metadata

  Semantic Scholar, PubMed

• No claims are published for this paper.

• No concepts are published for this paper.

• Hodgkin's disease and infectious mononucleosis.

  2009cited by this paper
• Recombinase-activating gene (RAG) 2-mediated V(D)J recombination is not essential for tumorigenesis in Atm-deficient mice.

  2000cited by this paper
• Cellular origin of human B-cell lymphomas.

  1999cited by this paper
• Regulated genomic instability and neoplasia in the lymphoid lineage.

  1999cited by this paper
• Genetic pathway to recurrent chromosome translocations in murine lymphoma involves V(D)J recombinase.

  1999cited by this paper
• Frequent occurrence of deletions and duplications during somatic hypermutation: implications for oncogene translocations and heavy chain disease.

  1998cited by this paper
• Immunohistochemical Detection of the Epstein-Barr Virus–Encoded Latent Membrane Protein 2A in Hodgkin's Disease and Infectious Mononucleosis

  1997cited by this paper
• Intraclonal molecular heterogeneity suggests a hierarchy of pathogenetic events in Burkitt's lymphoma.

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  1997cited by this paper
• Reexpression of RAG-1 and RAG-2 Genes in Activated Mature Mouse B cells

  1996cited by this paper
• Hodgkin and Reed-Sternberg cells in Hodgkin's disease represent the outgrowth of a dominant tumor clone derived from (crippled) germinal center B cells

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• Neoteny in Lymphocytes: Rag1 and Rag2 Expression in Germinal Center B Cells

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• Heterogeneous expression of Epstein-Barr virus latent proteins in endemic Burkitt's lymphoma.

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• Expression of recombination activating genes (RAG-1 and RAG-2) in Epstein-Barr virus-bearing B cells.

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• Epstein-Barr virus induction of recombinase-activating genes RAG1 and RAG2

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• Recombination activating gene-1 (RAG-1) expression in all differentiation stages of B-lineage precursor acute lymphoblastic leukemia.

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• Expression of human recombination activating genes (RAG-1 and RAG-2) in Hodgkin's disease.

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• Epstein-Barr virus infection in oral hairy leukoplakia: virus replication in the absence of a detectable latent phase.

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• RAG-1 and RAG-2, adjacent genes that synergistically activate V(D)J recombination.

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• Differences in B cell growth phenotype reflect novel patterns of Epstein‐Barr virus latent gene expression in Burkitt's lymphoma cells.

  1987cited by this paper
• The t(8; 14) chromosomal translocation occurring in B-cell malignancies results from mistakes in V–D–J joining

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  2004cites this paper
• Epstein-Barr Virus (EBV) LMP2A induces alterations in gene transcription similar to those observed in Reed-Sternberg cells of Hodgkin lymphoma.

  2003cites this paper
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• bcl-xL and RAG genes are induced and the response to IL-2 enhanced in EμEBNA-1 transgenic mouse lymphocytes

  2002cites this paper
• Expression of the recombination-activating genes in extrafollicular lymphocytes but no apparent reinduction in germinal center reactions in human tonsils.

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• Frequent expression of the Epstein–Barr virus (EBV)‐induced gene, EBI3, an IL‐12 p40‐related cytokine, in Hodgkin and Reed–Sternberg cells

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• Epstein‐Barr virus infection and human malignancies

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• Epstein-Barr virus infection and human malignancies.

  2001cites this paper