Effects of Moderate, Central Fluid Percussion Traumatic Brain Injury on Nitric Oxide Synthase Activity in Rats

No abstract is available for this paper.

• Publication year

  1998

• Venue

  Journal of Neurotrauma

• Publication date

  1998-08-01

• Fields of study

  Medicine

• Identifiers
  DOI 10.1089/neu.1998.15.627PMID 9726261
• External record

  Open on Semantic Scholar

• Source metadata

  Semantic Scholar, PubMed

• No claims are published for this paper.

• No concepts are published for this paper.

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  1992cited by this paper
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  1991cited by this paper
• Alterations in Regional Cerebral Blood Flow following Brain Injury in the Rat

  1991cited by this paper
• The double-edged role of nitric oxide in brain function and superoxide-mediated injury.

  1991influential reference
• Effect of chemical sympathectomy on cerebral blood flow in rats.

  1991cited by this paper
• Cerebral circulation and metabolism after severe traumatic brain injury: the elusive role of ischemia.

  1991cited by this paper
• Relationship between body and brain temperature in traumatically brain-injured rodents.

  1991cited by this paper
• Nitric oxide mediates glutamate-linked enhancement of cGMP levels in the cerebellum.

  1989cited by this paper
• Effects of Traumatic Brain Injury on Regional Cerebral Blood Flow in Rats as Measured with Radiolabeled Microspheres

  1989cited by this paper
• The Effects of Traumatic Brain Injury on Regional Cerebral Blood Flow in Rats

  1988influential reference
• Experimental Traumatic Brain Injury Elevates Brain Prostaglandin E2 and Thromboxane B2 Levels in Rats

  1988cited by this paper
• Isoflurane hyperpolarizes neurones in rat and human cerebral cortex.

  1987cited by this paper
• Endothelium-derived relaxing factor produced and released from artery and vein is nitric oxide.

  1987cited by this paper
• A fluid percussion model of experimental brain injury in the rat.

  1987cited by this paper
• Eicosonoid metabolism and beta-adrenergic mechanisms in coronary arterial smooth muscle: potential compartmentation of cAMP.

  1986cited by this paper
• Inhibition by Free Radical Scavengers and by Cyclooxygenase Inhibitors of Pial Arteriolar Abnormalities from Concussive Brain Injury in Cats

  1981cited by this paper
• The obligatory role of endothelial cells in the relaxation of arterial smooth muscle by acetylcholine

  1980cited by this paper
• Ischaemic brain damage in fatal non-missile head injuries.

  1978cited by this paper

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  2013cites this paper
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  2011cites this paper
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  2008cites this paper
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  2005cites this paper
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  2004cites this paper
• The Effects of Traumatic Brain Injury on Cerebral Blood Flow and Brain Tissue Nitric Oxide Levels and Cytokine Expression

  2004cites this paper
• Concentrations of Inducible Nitric Oxide Synthase (iNOS) and Neuronal Nitric Oxide Synthase (nNOS) in Cerebrospinal Fluid of Patients With Severe Head Injuries

  2003cites this paper
• Traumatic Cerebral Vascular Injury: The Effects of Concussive Brain Injury on the Cerebral Vasculature

  2003cites this paper
• Reversal of attenuation of cerebrovascular reactivity to hypercapnia by a nitric oxide donor after controlled cortical impact in a rat model of traumatic brain injury.

  2002cites this paper
• Role of Endothelial Nitric Oxide and Smooth Muscle Potassium Channels in Cerebral Arteriolar Dilation in Response to Acidosis

  2002cites this paper
• Cerebral ischemia and trauma-different etiologies yet similar mechanisms: neuroprotective opportunities.

  2002cites this paper
• Cortical hypoperfusion precedes hyperperfusion following controlled cortical impact injury.

  2002cites this paper
• MOLECULAR AND CELLULAR MECHANISMS OF TRAUMATIC CEREBRAL VASCULAR INJURY

  2001cites this paper
• [Importance of morphostructural changes observed in cortical micro-vessels in patients who died after a severe head injury].

  2000cites this paper
• Changes of nitric oxide and its relationship with clinical features, intracranial pressure and outcome in acute head injury.

  2000cites this paper
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  1999cites this paper
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  1999cites this paper
• Traumatic Injury to Rat Brain Upregulates Neuronal Nitric Oxide Synthase Expression and l-[3H]Nitroarginine Binding

  1999cites this paper
• Ameliorating cerebral hypoperfusion after traumatic brain injury.

  1999cites this paper