Inhibitors of nitric oxide synthesis and ischemia/reperfusion attenuate coronary vasodilator response to pinacidil in isolated rat heart.

No abstract is available for this paper.

• Publication year

  1997

• Venue

  Journal of Physiology and Pharmacology

• Publication date

  1997-12-01

• Fields of study

  Biology, Medicine

• Identifiers
  PMID 9444621
• External record

  Open on Semantic Scholar

• Source metadata

  Semantic Scholar, PubMed

• No claims are published for this paper.

• No concepts are published for this paper.

• Contribution of nitric oxide to metabolic coronary vasodilation in the human heart.

  1997cited by this paper
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  1996cited by this paper
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  1995cited by this paper
• Glibenclamide, a selective inhibitor of ATP-sensitive K+ channels, attenuates metabolic coronary vasodilatation induced by pacing tachycardia in dogs.

  1995cited by this paper
• Inhibition of nitric oxide synthesis reduces infarct size by an adenosine-dependent mechanism.

  1995cited by this paper
• Contribution of nitric oxide to metabolic coronary vasodilation in the human heart.

  1995cited by this paper
• A protective role of nitric oxide in isolated ischaemic/reperfused rat heart.

  1995cited by this paper
• Inhibition of nitric oxide production aggravates myocardial hypoperfusion during exercise in the presence of a coronary artery stenosis.

  1994cited by this paper
• Effect of an ATP sensitive potassium channel opener, levcromakalim, on coronary arterial microvessels in the beating canine heart.

  1994cited by this paper
• Atrial natriuretic factor and isosorbide dinitrate modulate the gating of ATP-sensitive K+ channels in cultured vascular smooth muscle cells.

  1994cited by this paper
• Reduced nitric oxide formation causes coronary vasoconstriction and impaired dilator responses to endogenous agonists and hypoxia in dogs

  1994cited by this paper
• Ischemic preconditioning protects against coronary endothelial dysfunction induced by ischemia and reperfusion.

  1994cited by this paper
• Glibenclamide reduces the coronary vasoactivity of adenosine receptor agonists.

  1994cited by this paper
• Impairment of myocardial vascular responsiveness after transient myocardial ischemia and reperfusion.

  1994cited by this paper
• Nonendothelial‐derived nitric oxide activates the ATP‐sensitive K+ channel of vascular smooth muscle cells

  1994cited by this paper
• Adenosine contributes to hypoxia-induced vasodilation through ATP-sensitive K+ channel activation.

  1993cited by this paper
• Endothelial dysfunction in coronary artery disease.

  1993cited by this paper
• Preconditioning protects coronary arteriolar endothelium from ischemia-reperfusion injury.

  1993cited by this paper
• Role of K+ATP channels in coronary vasodilation during exercise.

  1993cited by this paper
• Role of endothelium-derived nitric oxide in myocardial reactive hyperemia.

  1992cited by this paper
• ATP-sensitive potassium channel is essential to maintain basal coronary vascular tone in vivo.

  1992cited by this paper
• Microvascular sites and mechanisms responsible for reactive hyperemia in the coronary circulation of the beating canine heart.

  1992cited by this paper
• Role of nitric oxide in reactive hyperemia of the guinea pig heart.

  1992cited by this paper
• Different endothelial mechanisms involved in coronary responses to known vasodilators.

  1992cited by this paper
• Hypoxic vasodilatation in isolated, perfused guinea‐pig heart: an analysis of the underlying mechanisms.

  1991cited by this paper
• Nitric oxide: physiology, pathophysiology, and pharmacology.

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• Time course and mechanism of endothelial dysfunction in isolated ischemic- and hypoxic-perfused rat hearts.

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• Metabolic control of coronary blood flow.

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• Pharmacological estimation of drug-receptor dissociation constants. Statistical evaluation. I. Agonists.

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• Coronary response to diadenosine triphosphate after ischemia–reperfusion in the isolated rat heart

  2012cites this paper
• Nitric Oxide–mediated Relaxation to Lactate of Coronary Circulation in the Isolated Perfused Rat Heart

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• Plasma brain natriuretic peptide correlates with infarct size but not with subsequent remodeling in the rat heart.

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• Functional cell biology and molecular pharmacology of ATP sensitive potassium channels.

  2003influential citation
• Effects of nicorandil on myocardial function and metabolism in the post‐ischaemic reperfused heart with or without inhalation anaesthetics

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• The role of myocardial KATP-channel blockade in the protective effects of glibenclamide against ischaemia in the rat heart.

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• The role of adenosine and ATP-sensitive potassium channels in the protection afforded by ischemic preconditioning against the post-ischemic endothelial dysfunction in guinea-pig hearts.

  1998cites this paper
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