Effect of exogenous MSH6 and POLD1 expression on the mutation rate of the HPRT locus in a human colon cancer cell line with mutator phenotype, DLD-1.

No abstract is available for this paper.

• Publication year

  2004

• Venue

  International Journal of Oncology

• Publication date

  2004-03-01

• Fields of study

  Biology, Medicine

• Identifiers
  DOI 10.3892/IJO.24.3.697PMID 14767555
• External record

  Open on Semantic Scholar

• Source metadata

  Semantic Scholar, PubMed

• No claims are published for this paper.

• No concepts are published for this paper.

• E‐cadherin gene variants in gastric cancer families whose probands are diagnosed with diffuse gastric cancer

  2002cited by this paper
• Defective DNA polymerase-delta proofreading causes cancer susceptibility in mice.

  2001cited by this paper
• Mismatch repair defects in cancer.

  2000cited by this paper
• Detection of mutations in the DNA polymerase δ gene of human sporadic colorectal cancers and colon cancer cell lines

  1999cited by this paper
• The alphaE-catenin gene (CTNNA1) acts as an invasion-suppressor gene in human colon cancer cells.

  1999cited by this paper
• Eukaryotic DNA mismatch repair.

  1999cited by this paper
• Effect of hMSH6 cDNA expression on the phenotype of mismatch repair-deficient colon cancer cell line HCT15.

  1999cited by this paper
• Mutation rate at the hprt locus in human cancer cell lines with specific mismatch repair-gene defects.

  1997cited by this paper
• Correction of hypermutability, N-methyl-N'-nitro-N-nitrosoguanidine resistance, and defective DNA mismatch repair by introducing chromosome 2 into human tumor cells with mutations in MSH2 and MSH6.

  1997cited by this paper
• Conditional mutator phenotypes in hMSH2-deficient tumor cell lines.

  1997cited by this paper
• Differences in the spectrum of spontaneous mutations in the hprt gene between tumor cells of the microsatellite mutator phenotype.

  1996cited by this paper
• Loss of heterozygosity and base substitution at the APRT locus in mismatch-repair-proficient and -deficient colorectal carcinoma cell lines

  1996cited by this paper
• Polymerase delta variants in RER colorectal tumours.

  1995cited by this paper
• DNA mismatch binding defects, DNA damage tolerance, and mutator phenotypes in human colorectal carcinoma cell lines.

  1995cited by this paper
• Mutations of GTBP in genetically unstable cells.

  1995cited by this paper
• Mutator phenotypes in human colorectal carcinoma cell lines.

  1994cited by this paper
• The 3′ to 5′ exonuclease activity located in the DNA polymerase delta subunit of Saccharomyces cerevisiae is required for accurate replication.

  1991cited by this paper
• N,N-dimethylformamide-induced alteration of cell culture characteristics and loss of tumorigenicity in cultured human colon carcinoma cells.

  1979cited by this paper

• HPRT promotes proliferation and metastasis in head and neck squamous cell carcinoma through direct interaction with STAT3.

  2020cites this paper
• The Mechanism of Precise Genome Engineering in Human Cells

  2015cites this paper
• The Mechanism of Gene Targeting in Human Somatic Cells

  2014cites this paper
• What do we mean when we write “senescence,”“apoptosis,”“necrosis,” or “clearance of dying cells”?

  2010cites this paper
• Methods Used to Study Apoptotic Cell Clearance

  2009cites this paper
• A cancer-associated DNA polymerase δ variant modeled in yeast causes a catastrophic increase in genomic instability

  2009cites this paper
• Transcriptional Mutagenesis Induced by 8-Oxoguanine in Mammalian Cells

  2009cites this paper
• hMSH6 protein phosphorylation : DNA mismatch repair or DNA damage signaling?

  2009cites this paper