Evidence for immunological priming and increased frequency of CD4+ CD25+ cord blood T cells in children born to mothers with type 1 diabetes

No abstract is available for this paper.

• Publication year

  2006

• Venue

  Clinical and Experimental Immunology

• Publication date

  2006-12-01

• Fields of study

  Medicine

• Identifiers
  DOI 10.1111/j.1365-2249.2006.03243.xPMID 17100770PMCID PMC1810404
• External record

  Open on Semantic Scholar

• Source metadata

  Semantic Scholar, PubMed

• No claims are published for this paper.

• No concepts are published for this paper.

• Autoimmunity: Regulatory T cells in autoimmunity

  2007cited by this paper
• Genetics of type 1 diabetes mellitus.

  2006cited by this paper
• Analysis of FOXP3 protein expression in human CD4+CD25+ regulatory T cells at the single‐cell level

  2005cited by this paper
• CD4+CD25+FOXP3+ regulatory T cells from human thymus and cord blood suppress antigen‐specific T cell responses

  2005cited by this paper
• Lymphocyte subsets and cytokines in women with gestational diabetes mellitus and their newborn.

  2005cited by this paper
• Functional analysis of highly defined, FACS-isolated populations of human regulatory CD4+CD25+ T cells.

  2005cited by this paper
• Cord blood CD4(+)CD25(+)-derived T regulatory cell lines express FoxP3 protein and manifest potent suppressor function.

  2004cited by this paper
• CD25+CD4+ T cells in human cord blood: an immunoregulatory subset with naive phenotype and specific expression of forkhead box p3 (Foxp3) gene.

  2004cited by this paper
• Human CD4+CD25+ regulatory T cells.

  2004cited by this paper
• Is presence of islet autoantibodies at birth associated with development of persistent islet autoimmunity? The Diabetes Autoimmunity Study in the Young (DAISY).

  2004cited by this paper
• Transmission of maternal islet antibodies and risk of autoimmune diabetes in offspring of mothers with type 1 diabetes.

  2004cited by this paper
• Skin‐versus gut‐skewed homing receptor expression and intrinsic CCR4 expression on human peripheral blood CD4+CD25+ suppressor T cells

  2003cited by this paper
• In vivo expression of interleukin‐8, and regulated on activation, normal, T‐cell expressed, and secreted, by human germinal centre B lymphocytes

  2003cited by this paper
• Diabetes Antibody Standardization Program: first assay proficiency evaluation.

  2003cited by this paper
• CD4+CD25+ regulatory cells from human peripheral blood express very high levels of CD25 ex vivo.

  2003cited by this paper
• Characterization of human CD25+ CD4+ T cells in thymus,
cord and adult blood

  2002cited by this paper
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  2002cited by this paper
• Tumor necrosis factor-α regulation of CD4+C25+ T cell levels in NOD mice

  2002cited by this paper
• Genetics of type 1 diabetes mellitus

  2002cited by this paper
• Unique Chemotactic Response Profile and Specific Expression of Chemokine Receptors Ccr4 and Ccr8 by Cd4+Cd25+ Regulatory T Cells

  2001cited by this paper
• Cutting Edge: CCR4 Mediates Antigen-Primed T Cell Binding to Activated Dendritic Cells

  2001cited by this paper
• Prevalence, characteristics and diabetes risk associated with transient maternally acquired islet antibodies and persistent islet antibodies in offspring of parents with type 1 diabetes.

  2001cited by this paper
• Human CD4(+)CD25(+) cells: a naturally occurring population of regulatory T cells.

  2001cited by this paper
• Stability of disease-associated antibody titers in pregnant women with type 1 diabetes with or without residual beta-cell function.

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• The World Health Organization International Collaborative Study for Islet Cell Antibodies

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• Naturally anergic and suppressive CD25(+)CD4(+) T cells as a functionally and phenotypically distinct immunoregulatory T cell subpopulation.

  2000cited by this paper
• Postnatal Elimination of Transplacentally Acquired Disease-Associated Antibodies in Infants Born to Families with Type 1 Diabetes1

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• Regulatory T cells in autoimmmunity*.

  2000cited by this paper
• Postnatal elimination of transplacentally acquired disease-associated antibodies in infants born to families with type 1 diabetes. The Finnish TRIGR Study Group. Trial to Reduce IDDM in the Genetically at Risk.

  2000cited by this paper
• A study on lymphocyte subpopulation in diabetic mothers at delivery and in their newborn.

  1999cited by this paper
• Islet autoantibodies in cord blood from children who developed Type I (insulin-dependent) diabetes mellitus before 15 years of age

  1999cited by this paper
• Immunologic self-tolerance maintained by CD25+CD4+ naturally anergic and suppressive T cells: induction of autoimmune disease by breaking their anergic/suppressive state.

  1998cited by this paper
• CD4+CD25+ Immunoregulatory T Cells Suppress Polyclonal T Cell Activation In Vitro by Inhibiting Interleukin 2 Production

  1998cited by this paper
• CD4+CD25+ T cells inhibit both the induction and effector function of autoreactive T cells and represent a unique lineage of immunoregulatory cells.

  1998cited by this paper
• Macrophage-derived Chemokine Is a Functional Ligand for the CC Chemokine Receptor 4*

  1998cited by this paper
• A simple and rapid microSepharose assay for GAD65 and ICA512 autoantibodies in diabetes

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• A simple and rapid microSepharose assay for GAD65 and ICA512 autoantibodies in diabetes. Diabetes Incidence Study in Sweden (DISS).

  1998cited by this paper
• The T Cell-directed CC Chemokine TARC Is a Highly Specific Biological Ligand for CC Chemokine Receptor 4*

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• Rapid HLA-DQB1 Genotyping for Four Alleles in the Assessment of Risk for IDDM in the Finnish Population

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• Triple-label hybridization assay for type-1 diabetes-related HLA alleles.

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  1995cited by this paper
• A Prospective Study of the Role of Coxsackie B and Other Enterovirus Infections in the Pathogenesis of IDDM

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• A prospective study of the role of coxsackie B and other enterovirus infections in the pathogenesis of IDDM. Childhood Diabetes in Finland (DiMe) Study Group.

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  1994cited by this paper
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• Alterations of lymphocyte subsets in children of diabetic mothers.

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• Risk of IDDM in Children of Diabetic Mothers Decreases With Increasing Maternal Age at Pregnancy

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• Immunology in diabetic pregnancy: activated T cells in diabetic mothers and neonates

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• Differences in risk of insulin-dependent diabetes in offspring of diabetic mothers and diabetic fathers.

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  2024cites this paper
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  2023cites this paper
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  2021cites this paper
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  2016cites this paper
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  2015cites this paper
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  2012cites this paper
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  2010cites this paper
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  2009cites this paper
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  2009cites this paper
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  2008cites this paper
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  2008cites this paper
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  2008cites this paper
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  2008cites this paper
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  2008cites this paper
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• Author Index

  2006cites this paper