Adenovirus-mediated gene transfer of A20 renders endothelial cells resistant to activation: a means of evaluating the role of endothelial cell activation in xenograft rejection.

No abstract is available for this paper.

• Publication year

  1997

• Venue

  Transplantation Proceedings

• Publication date

  1997-02-01

• Fields of study

  Biology, Medicine

• Identifiers
  DOI 10.1016/S0041-1345(96)00184-4PMID 9123564
• External record

  Open on Semantic Scholar

• Source metadata

  Semantic Scholar, PubMed

• No claims are published for this paper.

• No concepts are published for this paper.

• Delayed Xenograft Rejection

  1997cited by this paper
• A20 Blocks Endothelial Cell Activation through a NF-κB-dependent Mechanism*

  1996cited by this paper
• Activation of the CPP32 Apoptotic Protease by Distinct Signaling Pathways with Differential Sensitivity to Bcl-xL*

  1996cited by this paper
• Delayed xenograft rejection.

  1996cited by this paper
• Inhibition of NF-κB by Pyrrolidine Dithiocarbamate Blocks Endothelial Cell Activation

  1995cited by this paper
• Inhibition of NF-kappa B by pyrrolidine dithiocarbamate blocks endothelial cell activation.

  1995cited by this paper
• Adenovirus-mediated gene transfer.

  1993cited by this paper
• The A20 zinc finger protein protects cells from tumor necrosis factor cytotoxicity.

  1992cited by this paper
• The A20 cDNA induced by tumor necrosis factor alpha encodes a novel type of zinc finger protein.

  1990cited by this paper
• I kappa B: a specific inhibitor of the NF-kappa B transcription factor.

  1988cited by this paper

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