Mechanisms modulating estrogen-induced uterine vasodilation.

No abstract is available for this paper.

• Publication year

  2002

• Venue

  Vascular pharmacology

• Publication date

  2002-02-01

• Fields of study

  Biology, Medicine

• Identifiers
  DOI 10.1016/S0306-3623(02)00135-0PMID 12379958
• External record

  Open on Semantic Scholar

• Source metadata

  Semantic Scholar, PubMed

• No claims are published for this paper.

• No concepts are published for this paper.

• The protective effects of estrogen on the cardiovascular system.

  2002cited by this paper
• Erratum: Ca2+ activated K+ channels modulate basal and E2β-induced rises in uterine blood flow in ovine pregnancy (American Journal of Physiology - Heart and Circulatory Physiology (July 2001) 50 (H422-H431))

  2001cited by this paper
• Ca(2+)-activated K(+) channels modulate basal and E(2)beta-induced rises in uterine blood flow in ovine pregnancy.

  2001cited by this paper
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  2001cited by this paper
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  2000cited by this paper
• Regulation of types I and III NOS in ovine uterine arteries by daily and acute estrogen exposure.

  2000cited by this paper
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  2000cited by this paper
• Calcium-activated potassium channels and nitric oxide coregulate estrogen-induced vasodilation.

  2000cited by this paper
• The effects of nitric oxide synthase inhibition on the uterine vasculature of the late pregnant ewe

  1999cited by this paper
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  1999cited by this paper
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  1999cited by this paper
• Effect of nitric oxide synthase inhibition on the uterine vasculature of the late-pregnant ewe.

  1999cited by this paper
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  1998cited by this paper
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  1998cited by this paper
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  1997cited by this paper
• Roles of NO and Ca2+‐activated K+ channels in coronary vasodilation induced by 17β‐estradiol in ischemic heart failure

  1997cited by this paper
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  1997cited by this paper
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  1997cited by this paper
• POTASSIUM CHANNELS IN VASCULAR SMOOTH MUSCLE

  1996cited by this paper
• Nitric oxide contributes to estrogen-induced vasodilation of the ovine uterine circulation.

  1996cited by this paper
• Gender differences in coronary artery diameter involve estrogen, nitric oxide, and Ca(2+)-dependent K+ channels.

  1996cited by this paper
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  1996cited by this paper
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  1995cited by this paper
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  1995cited by this paper
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  1995cited by this paper
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  1995cited by this paper
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  1995cited by this paper
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  1993cited by this paper
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  1992cited by this paper
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  1992cited by this paper
• Estrogen-induced uterine vasodilatation is antagonized by L-nitroarginine methyl ester, an inhibitor of nitric oxide synthesis.

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  1990cited by this paper
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  1985cited by this paper
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  1979cited by this paper
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  1977cited by this paper
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  1975cited by this paper
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  1974cited by this paper
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  1973cited by this paper
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  1973cited by this paper
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  1972cited by this paper
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