Polymorphisms of IL-17 and ICAM-1 and their expression in Guillain–Barré syndrome

No abstract is available for this paper.

• Publication year

  2017

• Venue

  International Journal of Neuroscience

• Publication date

  2017-08-01

• Fields of study

  Medicine, Psychology

• Identifiers
  DOI 10.1080/00207454.2016.1231186PMID 27595159
• External record

  Open on Semantic Scholar

• Source metadata

  Semantic Scholar, PubMed

• No claims are published for this paper.

• No concepts are published for this paper.

• Mitigated Tregs and Augmented Th17 Cells and Cytokines are Associated with Severity of Experimental Autoimmune Neuritis

  2014cited by this paper
• Circulating Th17, Th22, and Th1 Cells Are Elevated in the Guillain-Barré Syndrome and Downregulated by IVIg Treatments

  2014cited by this paper
• Elevated Levels of Cerebrospinal Fluid and Plasma Interleukin-37 in Patients with Guillain-Barré Syndrome

  2013cited by this paper
• Tumour necrosis factor α enhances CCL2 and ICAM-1 expression in peripheral nerve microvascular endoneurial endothelial cells

  2013cited by this paper
• Increased Circulating Th17 Cell Populations and Elevated CSF Osteopontin and IL-17 Concentrations in Patients with Guillain-Barré Syndrome

  2013cited by this paper
• IL-17 and IL-22 in Cerebrospinal Fluid and Plasma Are Elevated in Guillain-Barré Syndrome

  2012cited by this paper
• IL-17 receptor signaling and T helper 17-mediated autoimmune demyelinating disease.

  2011cited by this paper
• Serum and synovial fluid levels of interleukin-17 in correlation with disease activity in patients with RA

  2011cited by this paper
• Association between IL‐17F Gene Polymorphisms and Susceptibility to and Severity of Rheumatoid Arthritis (RA)

  2010cited by this paper
• Membrane attack complex of complement is not essential for immune mediated demyelination in experimental autoimmune neuritis.

  2010influential reference
• The expression pattern of inflammatory mediators in cerebrospinal fluid differentiates Guillain-Barré syndrome from chronic inflammatory demyelinating polyneuropathy.

  2010cited by this paper
• Tumor necrosis factor-alpha polymorphisms and expression in Guillain-Barré syndrome.

  2010cited by this paper
• Distribution of Th17 cells and Th1 cells in peripheral blood and cerebrospinal fluid in chronic inflammatory demyelinating polyradiculoneuropathy

  2010influential reference
• Expression of ICAM1 and VCAM1 Serum Levels in Rheumatoid Arthritis Clinical Activity. Association with Genetic Polymorphisms

  2009cited by this paper
• IL-17 and Th17 Cells.

  2009cited by this paper
• Compound A, a Plant Origin Ligand of Glucocorticoid Receptors, Increases Regulatory T Cells and M2 Macrophages to Attenuate Experimental Autoimmune Neuritis with Reduced Side Effects

  2009cited by this paper
• Structure-function relationships in the IL-17 receptor: implications for signal transduction and therapy.

  2008cited by this paper
• Interleukin-17F gene polymorphisms in Korean patients with Behçet’s disease

  2008cited by this paper
• Role of the novel Th17 cytokine IL‐17F in inflammatory bowel disease (IBD): Upregulated colonic IL‐17F expression in active Crohn's disease and analysis of the IL17F p.His161Arg polymorphism in IBD

  2008cited by this paper
• The function of interleukin 17 in the pathogenesis of rheumatoid arthritis

  2007cited by this paper
• The Influence of Polymorphisms of Interleukin-17A and Interleukin-17F Genes on the Susceptibility to Ulcerative Colitis

  2007cited by this paper
• Human TH17 lymphocytes promote blood-brain barrier disruption and central nervous system inflammation

  2007cited by this paper
• Increased serum levels of ICAM-1, ELAM-1 and TNF-α in inflammatory disorders of the peripheral nervous system

  2006cited by this paper
• IL-17F sequence variant (His161Arg) is associated with protection against asthma and antagonizes wild-type IL-17F activity.

  2006cited by this paper
• IL-22 is increased in active Crohn's disease and promotes proinflammatory gene expression and intestinal epithelial cell migration.

  2006cited by this paper
• [Guillain-Barré syndrome].

  2005cited by this paper
• DMA polymorphisms in adhesion molecule genes — a new risk factor for early atherosclerosis

  2004cited by this paper
• Intercellular adhesion molecule-1 polymorphisms in Korean patients with rheumatoid arthritis.

  2004cited by this paper
• Association between Gly241Arg ICAM-1 gene polymorphism and serum sICAM-1 concentration in the Stanislas cohort

  2003cited by this paper
• Increased expression of interleukin 17 in inflammatory bowel disease

  2003cited by this paper
• Intercellular adhesion molecule-1 K469E polymorphism: study of association with multiple sclerosis.

  2003cited by this paper
• Costimulatory Molecules and Autoimmune Thyroid Diseases

  2002cited by this paper
• Combined effects of calcineurin inhibitors or sirolimus with anti-CD40L mAb on alloengraftment under nonmyeloablative conditions.

  2002cited by this paper
• Chronic effects of Campylobacter infection.

  2002influential reference
• Variants in the VCAM1 gene and risk for symptomatic stroke in sickle cell disease.

  2002cited by this paper
• Immunoglobulin binding to brain in autoimmune mice.

  2001cited by this paper
• Preceding infections, immune factors, and outcome in Guillain–Barré syndrome

  2001cited by this paper
• Guillain Barré syndrome.

  2001cited by this paper
• ICAM-1 expression in autoimmune encephalitis visualized using magnetic resonance imaging.

  2000cited by this paper
• Intercellular adhesion molecule 1 (ICAM-1) gene polymorphisms in Italian patients with rheumatoid arthritis.

  2000cited by this paper
• Requirement of Endogenous Stem Cell Factor and Granulocyte-Colony-Stimulating Factor for IL-17-Mediated Granulopoiesis1

  2000cited by this paper
• Differential adhesion molecule requirements for immune surveillance and inflammatory recruitment.

  2000cited by this paper
• Intracellular adhesion molecule‐1 polymorphisms and genetic susceptibility to multiple sclerosis: Additional data and meta‐analysis

  2000cited by this paper
• Immunohistochemical localization of cytokines, C5b-9 and ICAM-1 in peripheral nerve of Guillain-Barré syndrome.

  2000cited by this paper
• Adhesion molecule expression and regulation on cells of the central nervous system.

  1999cited by this paper
• Interleukin-17 mRNA expression in blood and CSF mononuclear cells is augmented in multiple sclerosis

  1999cited by this paper
• Induction of adhesion molecules on human schwann cells by proinflammatory cytokines, an immunofluorescence study.

  1999cited by this paper
• Immunological profile of patients with primary progressive multiple sclerosis. Expression of adhesion molecules.

  1999cited by this paper
• IL-17 is produced by nickel-specific T lymphocytes and regulates ICAM-1 expression and chemokine production in human keratinocytes: synergistic or antagonist effects with IFN-gamma and TNF-alpha.

  1999cited by this paper
• IL-17 Is Produced by Nickel-Specific T Lymphocytes and Regulates ICAM-1 Expression and Chemokine Production in Human Keratinocytes: Synergistic or Antagonist Effects with IFN-γ and TNF-α

  1999cited by this paper
• Regulation of granulocyte colony-stimulating factor gene expression by interleukin-17.

  1998cited by this paper
• Electrophysiological classification of guillain‐barré syndrome: Clinical associations and outcome

  1998cited by this paper
• Multiple sclerosis: The increased frequency of the ICAM‐1 exon 6 gene point mutation genetic type K469

  1998cited by this paper
• IL-17 stimulates the production and expression of proinflammatory cytokines, IL-beta and TNF-alpha, by human macrophages.

  1998cited by this paper
• Cytokine production and the pathogenesis of experimental autoimmune neuritis and Guillain-Barré syndrome.

  1998influential reference
• Association between Campylobacter infection and Guillain-Barré syndrome.

  1997cited by this paper
• Upregulation of intercellular adhesion molecule-1 (ICAM-1) on rat Schwann cells in vitro: comparison of interferon-gamma, tumor necrosis factor-alpha and interleukin-1.

  1997cited by this paper
• Adhesion molecules in autoimmune disease.

  1996cited by this paper
• T cell interleukin-17 induces stromal cells to produce proinflammatory and hematopoietic cytokines

  1996cited by this paper
• Vascular changes and demyelination induced by the intraneural injection of tumour necrosis factor.

  1995influential reference
• Increased serum levels of ICAM-1, ELAM-1 and TNF-alpha in inflammatory disorders of the peripheral nervous system.

  1994cited by this paper
• Tumor necrosis factor-alpha in immune-mediated demyelination and Wallerian degeneration of the rat peripheral nervous system.

  1993influential reference
• Tumor necrosis factor-α in immune-mediated demyelination and Wallerian degeneration of the rat peripheral nervous system

  1993cited by this paper
• Suppression of experimental allergic neuritis by an antibody to the intracellular adhesion molecule ICAM-1.

  1993influential reference
• Binding of the integrin Mac-1 (CD11b/CD18) to the third immunoglobulin-like domain of ICAM-1 (CD54) and its regulation by glycosylation.

  1991cited by this paper
• Assessment of current diagnostic criteria for Guillain‐Barré syndrome

  1990cited by this paper
• A simple salting out procedure for extracting DNA from human nucleated cells.

  1988cited by this paper

• From glial cells to pain pathways: ICAM-1 as a central player in neuroinflammation and neuropathy

  2025cites this paper
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  2024cites this paper
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  2023cites this paper
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  2022cites this paper
• Acute Effects of the Consumption of Passiflora setacea Juice on Metabolic Risk Factors and Gene Expression Profile in Humans

  2020cites this paper
• A review of the role of genetic factors in Guillain–Barré syndrome

  2020cites this paper
• Genetic polymorphisms in Guillain-Barré Syndrome: A field synopsis and systematic meta-analysis.

  2020cites this paper
• Role of Ginkgolides in the Inflammatory Immune Response of Neurological Diseases: A Review of Current Literatures

  2020cites this paper
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  2019cites this paper
• Guillain–Barré Syndrome developing in a patient with Graves’ Disease

  2019cites this paper
• Peripheral Blood and Cerebrospinal Fluid Cytokine Levels in Guillain Barré Syndrome: A Systematic Review and Meta-Analysis

  2019cites this paper
• Genetic Factors in Neuroimmune Diseases

  2019cites this paper
• Guillain–Barré syndrome, transverse myelitis and infectious diseases

  2018cites this paper
• Chemokine and Adhesion Molecule Profiles in Aqueous Humor of Clinically Quiescent Uveitic Cataracts

  2018cites this paper
• C-Reactive Protein in Guillainbarré Syndrome: Recent Advances and Future Prospective

  2018cites this paper
• Th17 pathway signatures in a large Indian cohort of Guillain Barré syndrome.

  2018cites this paper
• Roles of macrophage migration inhibitory factor in Guillain-Barré syndrome and experimental autoimmune neuritis: beneficial or harmful?

  2018cites this paper
• IL‐23/IL‐17 immune axis in Guillain Barré Syndrome: Exploring newer vistas for understanding pathobiology and therapeutic implications

  2018cites this paper
• IL-23 and IL-27 Levels in Serum are Associated with the Process and the Recovery of Guillain-Barré Syndrome

  2018cites this paper
• Novel pathomechanisms in inflammatory neuropathies

  2017cites this paper