Suppression of AP‐1 constitutive activity interferes with polyomavirus MT antigen transformation ability

No abstract is available for this paper.

• Publication year

  2003

• Venue

  Journal of Cellular Biochemistry

• Publication date

  2003-10-01

• Fields of study

  Biology, Medicine

• Identifiers
  DOI 10.1002/jcb.10628PMID 14505342
• External record

  Open on Semantic Scholar

• Source metadata

  Semantic Scholar, PubMed

• No claims are published for this paper.

• No concepts are published for this paper.

• Inhibition of AP-1 transcription factor causes blockade of multiple signal transduction pathways and inhibits breast cancer growth

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• Expression of a human polyomavirus oncoprotein and tumour suppressor proteins in medulloblastomas

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• Transactivation of EGF receptor induced by angiotensin II regulates fibronectin and TGF-beta gene expression via transcriptional and post-transcriptional mechanisms.

  2000cited by this paper
• Polyoma virus middle T and small t antigens cooperate to antagonize p53-induced cell cycle arrest and apoptosis.

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  2000cited by this paper
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  2000cited by this paper
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  2000cited by this paper
• Retraction: Transactivation of EGF receptor induced by angiotensin II regulates fibronectin and TGF-β gene expression via transcriptional and post-transcriptional mechanisms

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  1999cited by this paper
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  1999cited by this paper
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  1999cited by this paper
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  1999cited by this paper
• Activation of the cyclin D1 Gene by the E1A-associated Protein p300 through AP-1 Inhibits Cellular Apoptosis*

  1999cited by this paper
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• Serine 257 Phosphorylation Regulates Association of Polyomavirus Middle T Antigen with 14-3-3 Proteins

  1998cited by this paper
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  1998cited by this paper
• Signaling from Polyomavirus Middle T and Small T Defines Different Roles for Protein Phosphatase 2A

  1998cited by this paper
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  1997cited by this paper
• The Role of the Shc Phosphotyrosine Interaction/Phosphotyrosine Binding Domain and Tyrosine Phosphorylation Sites in Polyoma Middle T Antigen-mediated Cell Transformation*

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  1984cited by this paper
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