Changes in corticocortical and corticohippocampal network during absence seizures in WAG/Rij rats revealed with time varying Granger causality.

No abstract is available for this paper.

• Publication year

  2016

• Venue

  Epilepsy & Behavior

• Publication date

  2016-11-01

• Fields of study

  Biology, Medicine, Psychology

• Identifiers
  DOI 10.1016/j.yebeh.2016.08.009PMID 27728902
• External record

  Open on Semantic Scholar

• Source metadata

  Semantic Scholar, PubMed

• No claims are published for this paper.

• No concepts are published for this paper.

• The genetic absence epilepsy rat from Strasbourg as a model to decipher the neuronal and network mechanisms of generalized idiopathic epilepsies.

  2016cited by this paper
• [Thalamo-Cortical Mechanisms of Initiation, Maintenance and Termination of Spike-wave Discharges at WAG/Rij rats].

  2016influential reference
• Choosing the optimal model parameters for Granger causality in application to time series with main timescale

  2016cited by this paper
• The hippocampus participates in a pharmacological rat model of absence seizures.

  2016cited by this paper
• Dynamics of directional coupling underlying spike-wave discharges.

  2016influential reference
• Detecting changes in coupling with Granger causality method from time series with fast transient processes

  2015cited by this paper
• Termination of ongoing spike-wave discharges investigated by cortico-thalamic network analyses.

  2014cited by this paper
• Functional and structural brain networks in epilepsy: What have we learned?

  2013cited by this paper
• The involvement of limbic structures in typical and atypical absence epilepsy.

  2013cited by this paper
• Peri-ictal network dynamics of spike-wave discharges: phase and spectral characteristics.

  2013cited by this paper
• Focal corticothalamic sources during generalized absence seizures: a MEG study.

  2013cited by this paper
• The dynamics of cortico-thalamo-cortical interactions at the transition from pre-ictal to ictal LFPs in absence epilepsy.

  2012cited by this paper
• Spurious causalities due to low temporal resolution: Towards detection of bidirectional coupling from time series

  2012cited by this paper
• A brief history on the oscillating roles of thalamus and cortex in absence seizures

  2012cited by this paper
• WAG/Rij rats show a reduced expression of CB1 receptors in thalamic nuclei and respond to the CB1 receptor agonist, R(+)WIN55,212‐2, with a reduced incidence of spike‐wave discharges

  2010cited by this paper
• Revised terminology and concepts for organization of seizures and epilepsies: Report of the ILAE Commission on Classification and Terminology, 2005–2009

  2010cited by this paper
• Onset and propagation of spike and slow wave discharges in human absence epilepsy: A MEG study

  2009cited by this paper
• Cortical local and long-range synchronization interplay in human absence seizure initiation

  2009cited by this paper
• Granger causality and transfer entropy are equivalent for Gaussian variables.

  2009cited by this paper
• Does spatiotemporal synchronization of EEG change prior to absence seizures?

  2008cited by this paper
• Glutamatergic alterations in the cortex of genetic absence epilepsy rats

  2007cited by this paper
• Absence seizures are reduced by the enhancement of GABA-ergic inhibition in the hippocampus in WAG/Rij rats.

  2007cited by this paper
• Rhythms of the brain

  2006cited by this paper
• Source analysis of generalized spike-wave complexes

  2005cited by this paper
• CHAPTER 18 – Genetic Models of Absence Epilepsy in the Rat

  2005cited by this paper
• Analyzing multiple nonlinear time series with extended Granger causality

  2004cited by this paper
• Estimating mutual information.

  2003cited by this paper
• Genetic Animal Models for Absence Epilepsy: A Review of the WAG/Rij Strain of Rats

  2003cited by this paper
• Cortical Focus Drives Widespread Corticothalamic Networks during Spontaneous Absence Seizures in Rats

  2002influential reference
• Treatment of Typical Absence Seizures and Related Epileptic Syndromes

  2001influential reference
• Hippocampal extracellular amino acids and EEG spectral analysis in a genetic rat model of absence epilepsy.

  2000cited by this paper
• Intrinsic properties of reticular thalamic neurons relevant to genetically determined spike-wave generation.

  1999cited by this paper
• Nonlinear dynamics: Death by delay

  1998cited by this paper
• Genetic absence epilepsy in rats from Strasbourg--a review.

  1992cited by this paper
• Autoradiographic measurement of local cerebral beta-hydroxybutyrate uptake in the rat during postnatal development.

  1991cited by this paper
• Amplitude response of coupled oscillators

  1990cited by this paper
• gamma-Hydroxybutyrate model of generalized absence seizures: further characterization and comparison with other absence models.

  1988cited by this paper
• γ‐Hydroxybutyrate Model of Generalized Absence Seizures: Further Characterization and Comparison with Other Absence Models

  1988cited by this paper
• Two types of electrocortical paroxysms in an inbred strain of rats.

  1986cited by this paper
• The Rat Brain in Stereotaxic Coordinates

  1983cited by this paper
• The Rat Brain in Stereotaxic Coordinates

  1983cited by this paper
• Prediction of Spike‐Wave Bursts in Absence Epilepsy by EEG Power‐Spectrum Signals

  1982cited by this paper
• Nearest Neighbour Searches and the Curse of Dimensionality

  1979cited by this paper
• "Generalized" epileptic seizures elicited by electrical stimulation of the frontal lobe in man.

  1974cited by this paper

• The Role of Acetylcholinesterase Enzyme Inhibitor Rivastigmine on Spike-Wave Discharges, Learning-Memory, Anxiety, and TRPV1 Channel Expression in Genetic Absence Epileptic WAG/Rij Rats

  2025cites this paper
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  2025cites this paper
• Dynamics of Cerebral Function in Patients with Acute Cerebellar Infarction

  2023cites this paper
• Neural oscillations after acute large artery atherosclerotic cerebral infarction during resting state and sleep spindles

  2023cites this paper
• Transient dynamics in a class of mathematical models of epileptic seizures

  2022cites this paper
• Testing approaches to statistical evaluation of connectivity estimates in epileptic brain based on simple oscillatory models

  2022cites this paper
• Research on Top Archer’s EEG Microstates and Source Analysis in Different States

  2022cites this paper
• Blood-brain barrier targeted delivery of lacosamide-conjugated gold nanoparticles: Improving outcomes in absence seizures.

  2022cites this paper
• The combined effects of the thalamic feed-forward inhibition and feed-back inhibition in controlling absence seizures

  2022cites this paper
• Retrosplenial Cortex Contributes to Network Changes during Seizures in the GAERS Absence Epilepsy Rat Model

  2021cites this paper
• Altered peri-seizure EEG microstate dynamics in patients with absence epilepsy.

  2021cites this paper
• Clinical and experimental insight into pathophysiology, comorbidity and therapy of absence seizures

  2020cites this paper
• Connectivity detection in application to spike-wave discharge study

  2020cites this paper
• Modeling spike and wave discharge initiation, maintenance and termination with hierarchical networks of neurooscillators

  2020cites this paper
• Dynamical mesoscale model of absence seizures in genetic models

  2020cites this paper
• Modeling spike-wave discharges by a complex network of neuronal oscillators

  2018cites this paper
• Reconstruction of ensembles of generalized Van der Pol oscillators from vector time series

  2018cites this paper
• Directional functional coupling during limbic seizures in rats revealed by nonlinear Granger causality

  2018cites this paper
• [Comparative analysis of methods for estimation of undirected coupling from time series of intracranial EEGs of cortex of rats-genetic models of absence epilepsy]

  2017cites this paper
• Contribution of M1 and M2 muscarinic receptor subtypes to convulsions in fasted mice treated with scopolamine and given food

  2017cites this paper