Electron microscopic and confocal laser microscopy analysis of amyloid plaques in chronic wasting disease transmitted to transgenic mice

No abstract is available for this paper.

• Publication year

  2017

• Venue

  Prion

• Publication date

  2017-11-02

• Fields of study

  Biology, Medicine, Chemistry

• Identifiers
  DOI 10.1080/19336896.2017.1384109PMID 29105545PMCID PMC5786356
• External record

  Open on Semantic Scholar

• Source metadata

  Semantic Scholar, PubMed

• No claims are published for this paper.

• No concepts are published for this paper.

• Amyloid-β pathology and cerebral amyloid angiopathy are frequent in iatrogenic Creutzfeldt-Jakob disease after dural grafting.

  2016cited by this paper
• Dura mater is a potential source of Aβ seeds

  2016cited by this paper
• First case of chronic wasting disease in Europe in a Norwegian free-ranging reindeer

  2016cited by this paper
• A neuroprotective role for microglia in prion diseases

  2016cited by this paper
• Evidence for human transmission of amyloid-β pathology and cerebral amyloid angiopathy

  2015cited by this paper
• Fibrillar Amyloid Plaque Formation Precedes Microglial Activation

  2015cited by this paper
• Serial propagation of distinct strains of Aβ prions from Alzheimer’s disease patients

  2014cited by this paper
• Microglial Cx3cr1 knockout reduces prion disease incubation time in mice

  2014cited by this paper
• Biology and genetics of prions causing neurodegeneration.

  2013cited by this paper
• Kuru: Genes, Cannibals and Neuropathology

  2012cited by this paper
• Evaluation of Prion Deposits and Microglial Activation in Scrapie-Infected Mice Using Molecular Imaging Probes

  2010cited by this paper
• Ultrastructural Characteristics (or Evaluation) of Creutzfeldt-Jakob Disease and Other Human Transmissible Spongiform Encephalopathies or Prion Diseases

  2010cited by this paper
• Ultrastructural study of florid plaques in variant Creutzfeldt–Jakob disease: a comparison with amyloid plaques in kuru, sporadic Creutzfeldt–Jakob disease and Gerstmann–Sträussler–Scheinker disease

  2009cited by this paper
• Rapid appearance and local toxicity of amyloid-β plaques in a mouse model of Alzheimer’s disease

  2008cited by this paper
• Strain Fidelity of Chronic Wasting Disease upon Murine Adaptation

  2006cited by this paper
• Sporadic Creutzfeldt-Jakob Disease: The Extent of Microglia Activation Is Dependent on the Biochemical Type of PrPSc

  2005cited by this paper
• Ultrastructural neuropathology of chronic wasting disease in captive mule deer

  2004cited by this paper
• Microglia is a component of the prion protein amyloid plaque in the Gerstmann-Sträussler-Scheinker syndrome

  2004cited by this paper
• Topographic distribution of scrapie amyloid-immunoreactive plaques in chronic wasting disease in captive mule deer (Odocoileus hemionus hemionus)

  2004cited by this paper
• PDAPP; YFP double transgenic mice: A tool to study amyloid‐β associated changes in axonal, dendritic, and synaptic structures

  2003cited by this paper
• Unique inflammatory RNA profiles of microglia in Creutzfeldt–Jakob disease

  2003cited by this paper
• Microglia from Creutzfeldt-Jakob Disease-Infected Brains Are Infectious and Show Specific mRNA Activation Profiles

  2002cited by this paper
• Deposition patterns of disease-associated prion protein in captive mule deer brains with chronic wasting disease

  2001cited by this paper
• Microglia derive from progenitors, originating from the yolk sac, and which proliferate in the brain.

  1999cited by this paper
• Microglia and the Development of Spongiform Change in Creutzfeldt-Jakob Disease

  1998cited by this paper
• Creutzfeldt-Jakob disease

  1998cited by this paper
• Ultrastructural pathology of Gerstmann-Sträussler-Scheinker disease.

  1995cited by this paper
• Electron microscopic findings in brain of Rocky Mountain elk with chronic wasting disease.

  1994cited by this paper
• Fibrils in brain of Rocky Mountain elk with chronic wasting disease contain scrapie amyloid.

  1993cited by this paper
• Immunolocalization of scrapie amyloid (PrP27-30) in chronic wasting disease of Rocky Mountain elk and hybrids of captive mule deer and white-tailed deer.

  1991cited by this paper
• A comparative immunohistochemical study of Kuru and senile plaques with a special reference to glial reactions at various stages of amyloid plaque formation.

  1991cited by this paper
• Amyloid plaques in spongiform encephalopathy of mule deer.

  1985cited by this paper
• CHRONIC WASTING DISEASE OF CAPTIVE MULE DEER: A SPONGIFORM ENCEPHALOPATHY1

  1980cited by this paper

• No citing papers are available for this paper.