Chronic rejection in cyclosporin A treated renal allograft recipients with allopeptide reactive T cells.

No abstract is available for this paper.

• Publication year

  1999

• Venue

  Transplantation Proceedings

• Publication date

  1999-02-01

• Fields of study

  Medicine

• Identifiers
  DOI 10.1016/S0041-1345(98)01949-6PMID 10083522
• External record

  Open on Semantic Scholar

• Source metadata

  Semantic Scholar, PubMed

• No claims are published for this paper.

• No concepts are published for this paper.

• The role of T-cell costimulatory activation pathways in transplant rejection.

  1998cited by this paper
• Persistent allopeptide reactivity and epitope spreading in chronic rejection of organ allografts.

  1998cited by this paper
• Indirect allorecognition of major histocompatibility complex allopeptides in human renal transplant recipients with chronic graft dysfunction.

  1997cited by this paper
• Indirect recognition of donor HLA-DR peptides in organ allograft rejection.

  1996cited by this paper
• Limited T cell response to donor MHC peptides during allograft rejection. Implications for selective immune therapy in transplantation.

  1994cited by this paper
• Acute rejection episodes – Best predictor of long‐term primary cadaveric renal transplant survival

  1994cited by this paper
• Risk factors for chronic rejection in renal allograft recipients.

  1993cited by this paper
• Correlation of in vitro CD4+ T helper cell function with clinical graft status in immunosuppressed kidney transplant recipients.

  1991cited by this paper
• Effects of cyclosporin, FK506, and rapamycin on graft-vessel disease.

  1991cited by this paper
• Acquired donor-specific hyporesponsiveness in long-term kidney allograft recipients.

  1991cited by this paper
• Differential sensitivity of human T helper cell pathways by in vitro exposure to cyclosporin A.

  1990cited by this paper

• No citing papers are available for this paper.