Differential Responses of Normal Human Melanocytes to Intra- and Extracellular dsRNA

No abstract is available for this paper.

• Publication year

  2015

• Venue

  DNA and Cell Biology

• Publication date

  2015-06-01

• Fields of study

  Biology, Medicine, Chemistry

• Identifiers
  DOI 10.1089/dna.2014.2711PMID 25803620PMCID PMC4485883
• External record

  Open on Semantic Scholar

• Source metadata

  Semantic Scholar, PubMed

• No claims are published for this paper.

• No concepts are published for this paper.

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  2014influential reference
• Molecular basis of NF-κB signaling.

  2013cited by this paper
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  2011cited by this paper
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  2011cited by this paper
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  2011influential reference
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  2011influential reference
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  2011cited by this paper
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  2011cited by this paper
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  2011cited by this paper
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  2011cited by this paper
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  2009cited by this paper
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  2009cited by this paper
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  2009cited by this paper
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  2008cited by this paper
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  2008influential reference
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  2008cited by this paper
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  2007influential reference
• Melanocyte biology and skin pigmentation

  2007cited by this paper
• IKK-i signals through IRF3 and NFkappaB to mediate the production of inflammatory cytokines.

  2006cited by this paper
• TAK1 Is a Master Regulator of Epidermal Homeostasis Involving Skin Inflammation and Apoptosis*

  2006cited by this paper
• Vitiligo with inflammatory raised borders with hepatitis C virus infection

  2006cited by this paper
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  2006cited by this paper
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  2005cited by this paper
• Toll-like receptor 3-mediated activation of NF-kappaB and IRF3 diverges at Toll-IL-1 receptor domain-containing adapter inducing IFN-beta.

  2004cited by this paper
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  2004cited by this paper
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  2003cited by this paper
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  2003cited by this paper
• IRF-3-dependent, NFκB- and JNK-independent activation of the 561 and IFN-β genes in response to double-stranded RNA

  2002cited by this paper
• IRF-3-dependent, NFkappa B- and JNK-independent activation of the 561 and IFN-beta genes in response to double-stranded RNA.

  2002cited by this paper
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  2000cited by this paper
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  2000cited by this paper
• Participation of Bax-alpha in IFN-alpha-mediated apoptosis in Daudi B lymphoma cells.

  1998cited by this paper
• Participation of Bax-α in IFN-α-Mediated Apoptosis in Daudi B Lymphoma Cells

  1998cited by this paper
• Cytomegalovirus DNA identified in skin biopsy specimens of patients with vitiligo.

  1996cited by this paper
• Egress of varicella-zoster virus from the melanoma cell: a tropism for the melanocyte

  1995cited by this paper
• Human immunodeficiency virus—associated vitiligo: Expression of autoimmunity with immunodeficiency?

  1987cited by this paper

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  2020cites this paper
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  2020cites this paper
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  2019cites this paper
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  2018cites this paper
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  2018cites this paper
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  2017cites this paper
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  2017cites this paper