Identification of processing elements and interactors implicate SMN, coilin and the pseudogene-encoded coilp1 in telomerase and box C/D scaRNP biogenesis

No abstract is available for this paper.

• Publication year

  2016

• Venue

  RNA Biology

• Publication date

  2016-08-17

• Fields of study

  Biology, Medicine

• Identifiers
  DOI 10.1080/15476286.2016.1211224PMID 27419845PMCID PMC5056767
• External record

  Open on Semantic Scholar

• Source metadata

  Semantic Scholar, PubMed

• No claims are published for this paper.

• No concepts are published for this paper.

• A role for the survival of motor neuron protein in mRNP assembly and transport.

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  2015influential reference
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  2014influential reference
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• The coilin interactome identifies hundreds of small noncoding RNAs that traffic through Cajal bodies.

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• Spinal muscular atrophy: a motor neuron disorder or a multi‐organ disease?

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• Developmental arrest of Drosophila survival motor neuron (Smn) mutants accounts for differences in expression of minor intron-containing genes

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• Spinal muscular atrophy: going beyond the motor neuron.

  2013cited by this paper
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  2013cited by this paper
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  2013cited by this paper
• Biogenesis of telomerase ribonucleoproteins.

  2012cited by this paper
• Regulated specific proteolysis of the Cajal body marker protein coilin

  2012influential reference
• In Vitro RNase and Nucleic Acid Binding Activities Implicate Coilin in U snRNA Processing

  2012cited by this paper
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  2012cited by this paper
• A Drosophila model of spinal muscular atrophy uncouples snRNP biogenesis functions of survival motor neuron from locomotion and viability defects.

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  2011cited by this paper
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  2005cited by this paper
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  2004influential reference
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  2001cited by this paper
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  2001cited by this paper
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  2001cited by this paper
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  2001cited by this paper
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  1999cited by this paper
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  1999cited by this paper
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  1999cited by this paper
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  1998cited by this paper
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  1995cited by this paper
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  1994cited by this paper
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  1993cited by this paper
• Classification of spinal muscular atrophies.

  1980cited by this paper

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