Preconditioning in rat hearts is independent of mitochondrial F1F0ATPase inhibition.

No abstract is available for this paper.

• Publication year

  1998

• Venue

  American Journal of Physiology. Heart and Circulatory Physiology

• Publication date

  Unknown publication date

• Fields of study

  Biology, Medicine

• Identifiers
  DOI 10.1152/ajpheart.1998.274.1.H90PMID 29591532
• External record

  Open on Semantic Scholar

• Source metadata

  Semantic Scholar

• No claims are published for this paper.

• No concepts are published for this paper.

• Paradoxical effect of ischemic preconditioning on ischemic contracture? NMR studies of energy metabolism and intracellular pH in the rat heart.

  1996cited by this paper
• Effect of reversible ischemia on the activity of the mitochondrial ATPase: relationship to ischemic preconditioning.

  1996cited by this paper
• ATPase activity, IF1 content, and proton conductivity of ESMP from control and ischemic slow and fast heart-rate hearts

  1995cited by this paper
• The KATP blocker sodium 5-hydroxydecanoate does not abolish preconditioning in isolated rat hearts.

  1995cited by this paper
• Preconditioning by brief ischaemic episodes in the isolated rat heart assessed by 31P NMR spectroscopy: dissociation between metabolic and functional recovery?

  1995cited by this paper
• A redox-based mechanism for cardioprotection induced by ischemic preconditioning in perfused rat heart.

  1995cited by this paper
• Mechanisms of ischemic preconditioning in rat myocardium. Roles of adenosine, cellular energy state, and mitochondrial F1F0-ATPase.

  1995cited by this paper
• Pyruvate increases threshold for preconditioning in globally ischemic rat hearts.

  1994cited by this paper
• Importance of metabolic inhibition and cellular pH in mediating preconditioning contractile and metabolic effects in rat hearts.

  1994cited by this paper
• Ischemic preconditioning stimulates anaerobic glycolysis in the isolated rabbit heart.

  1994cited by this paper
• Preconditioning against myocardial dysfunction after ischemia and reperfusion by an alpha 1-adrenergic mechanism.

  1993cited by this paper
• Loss of Myocardial Protection After Preconditioning Correlates With the Time Course of Glycogen Recovery Within the Preconditioned Segment

  1993cited by this paper
• Laboratory Investigation The Cardioprotective Effects of Ischemic 'Preconditioning' Are Not Mediated by Adenosine Receptors in Rat Hearts

  1993cited by this paper
• Use of Percoll gradients for isolation of human placenta mitochondria suitable for investigating outer membrane proteins.

  1993cited by this paper
• Effects of duration of ischaemia during preconditioning on mechanical function, enzyme release and energy production in the isolated working rat heart.

  1992cited by this paper
• Blockade of ATP-sensitive potassium channels prevents myocardial preconditioning in dogs.

  1992cited by this paper
• Ischemic preconditioning attenuates acidosis and postischemic dysfunction in isolated rat heart.

  1992cited by this paper
• Protection Against Infarction Afforded by Preconditioning is Mediated by A1 Adenosine Receptors in Rabbit Heart

  1991cited by this paper
• Effect of inhibition of the mitochondrial ATPase on net myocardial ATP in total ischemia.

  1991cited by this paper
• Cardioprotective effects of the potassium channel opener cromakalim: stereoselectivity and effects on myocardial adenine nucleotides.

  1991cited by this paper
• Control of mitochondrial ATP synthesis in the heart.

  1991cited by this paper
• Ischemic preconditioning slows energy metabolism and delays ultrastructural damage during a sustained ischemic episode.

  1990cited by this paper
• Effects of oligomycin and acidosis on rates of ATP depletion in ischemic heart muscle.

  1986cited by this paper
• Studies on the mechanism of oxidative phosphorylation. Catalytic site cooperativity in ATP synthesis.

  1985cited by this paper
• Improvement of Mitochondrial Energy Production in Ischemic Myocardium by in Vivo Infusion of Ruthenium Reb

  1980cited by this paper
• The mode of inhibition of oxidative phosphorylation by efrapeptin (A23871). Evidence for an alternating site mechanism for ATP synthesis.

  1978cited by this paper
• Studies on the mechanism of oxidative phosphorylation: VII. Preparation of a submitochondrial particle (ETPH) which is capable of fully coupled oxidative phosphorylation

  1964cited by this paper
• A NATURALLY OCCURRING INHIBITOR OF MITOCHONDRIAL ADENOSINE TRIPHOSPHATASE.

  1963cited by this paper
• Lactic Dehydrogenase Activity in Blood.∗

  1955cited by this paper

• Heart Transplant: Donation after Circulatory Death

  2019cites this paper
• Is there an effect of ischemic conditioning on myocardial contractile function following acute myocardial ischemia/reperfusion injury?

  2019cites this paper
• The potential role of mitochondrial ATP synthase inhibitory factor 1 (IF1) in coronary heart disease: a literature review

  2017cites this paper
• The role of hexokinase in cardioprotection – mechanism and potential for translation

  2014cites this paper
• Resistance exercise training modulates acute gene expression during human skeletal muscle hypertrophy.

  2014cites this paper
• Cellular and Molecular Aspects of Cardioprotection

  2014cites this paper
• Did a Classic Preconditioning Study Provide a Clue to the Identity of the Mitochondrial Permeability Transition Pore?

  2013cites this paper
• Altered expression of mitochondrial electron transport chain proteins and improved myocardial energetic state during late ischemic preconditioning.

  2012cites this paper
• Mitochondrial adenine nucleotide transport and cardioprotection.

  2012cites this paper
• Cardioprotection and Signaling Pathways

  2011cites this paper
• What makes the mitochondria a killer? Can we condition them to be less destructive?

  2011cites this paper
• Mitochondrial and cell-surface F0F1ATPsynthase in innate and acquired cardioprotection

  2009cites this paper
• The role of O-GlcNAc signaling in acute myocardial ischemia.

  2009cites this paper
• Pharmacological profile of the selective mitochondrial F1F0 ATP hydrolase inhibitor BMS-199264 in myocardial ischemia.

  2008cites this paper
• Mechanisms underlying acute protection from cardiac ischemia-reperfusion injury.

  2008cites this paper
• Preconditioning Results in S-Nitrosylation of Proteins Involved in Regulation of Mitochondrial Energetics and Calcium Transport

  2007cites this paper
• Local delivery of PKCε-activating peptide mimics ischemic preconditioning in aged hearts through GSK-3β but not F1-ATPase inactivation

  2007cites this paper
• CHAPTER 10 – Cellular Pathways and Molecular Events in Cardioprotection

  2007cites this paper
• Mitochondrial respiration and membrane potential after low-flow ischemia are not affected by ischemic preconditioning.

  2007cites this paper
• Regulation of Mitochondrial Respiration in Heart Muscle

  2007cites this paper
• Preconditioning: the mitochondrial connection.

  2007cites this paper
• Myocardial protection from ischemic preconditioning is not blocked by sub-chronic inhibition of carnitine palmitoyltransferase I.

  2005cites this paper
• Biochemical dysfunction in heart mitochondria exposed to ischaemia and reperfusion.

  2005cites this paper
• Cellular and molecular events in ischemic preconditioning: potential therapeutic applications in cardioprotection.

  2005cites this paper
• EPICARDIAL AUTO-FLUORESCENCE NAD(P)H KINETICS IN THE ISCHEMICALLY PRECONDITIONED LANGERDORFF RAT HEART. EFFECTS OF CAPSAICIN. PART 2.

  2005cites this paper
• Leukemia inhibitory factor reduces contractile function and induces alterations in energy metabolism in isolated cardiomyocytes.

  2004cites this paper
• F0F1 ATP synthase activity is differently modulated by coronary reactive hyperemia before and after ischemic preconditioning in the goat.

  2004cites this paper
• Excessive ATP hydrolysis in ischemic myocardium by mitochondrial F1F0-ATPase: effect of selective pharmacological inhibition of mitochondrial ATPase hydrolase activity.

  2004cites this paper
• Review Mitochondria Play a Critical Role in Cardioprotection

  2004cites this paper
• Mitochondria play a critical role in cardioprotection.

  2004cites this paper
• Linoleic acid epoxide promotes the maintenance of mitochondrial function and active Na+ transport following hypoxia.

  2004cites this paper
• In vitro and in vivo studies of F0F1ATP synthase regulation by inhibitor protein IF1 in goat heart

  2004cites this paper
• Cellular energy state and calcium in myocardial substrate oxidation, ischemia and preconditioning

  2003cites this paper
• Protection afforded by ischemic preconditioning is not mediated by effects on cell-to-cell electrical coupling during myocardial ischemia-reperfusion.

  2003cites this paper
• Ischaemic preconditioning and a mitochondrial KATP channel opener both produce cardioprotection accompanied by F1F0-ATPase inhibition in early ischaemia

  2003cites this paper
• Myocardial energy metabolism in ischemic preconditioning, role of adenosine catabolism

  2002cites this paper
• Understanding myocardial ischemic preconditioning, and the implications for a role of adenosine catabolism

  2002cites this paper
• Metabolic plasticity and the promotion of cardiac protection in ischemia and ischemic preconditioning.

  2002cites this paper
• The effect of K(atp)channel activation on myocardial cationic and energetic status during ischemia and reperfusion: role in cardioprotection.

  2001cites this paper
• Myocardial ischemic preconditioning and mitochondrial F1F0-ATPase activity

  2000cites this paper
• Role of energy metabolism in the preconditioned heart--a possible contribution of mitochondria.

  1999cites this paper
• The Mitochondrial Permeability Transition: Role in Ischemia/Reperfusion Injury

  1999cites this paper