TGF alpha differentially expressed in liver foci induced by diethylnitrosamine initiation and peroxisome proliferator promotion.

No abstract is available for this paper.

• Publication year

  1995

• Venue

  Carcinogenesis

• Publication date

  Unknown publication date

• Fields of study

  Biology, Medicine, Environmental Science

• Identifiers
  DOI 10.1093/CARCIN/16.1.77PMID 7530605
• External record

  Open on Semantic Scholar

• Source metadata

  Semantic Scholar, PubMed

• No claims are published for this paper.

• No concepts are published for this paper.

• Hepatic peroxisome proliferation in rodents and its significance for humans.

  1993cited by this paper
• Transforming growth factor-alpha immunoreactivity in a variety of epithelial tissues.

  1993cited by this paper
• Influence of Orotic Acid on Multistage Hepatocarcinogenesis in the Rat: Resistance of Hepatocytes from Nodules to the Mitoinhibitory Effects of Orotic Acid

  1993cited by this paper
• Collaboration between growth factors and diverse chemical carcinogens in hepatocarcinogenesis of transforming growth factor alpha transgenic mice.

  1993cited by this paper
• Intrahepatic distribution of transforming growth factor‐alpha (TGFα) during liver regeneration following carbon tetrachlorideinduced necrosis

  1993cited by this paper
• Association between expression of transforming growth factor-alpha and progression of hepatocellular foci to neoplasms.

  1992cited by this paper
• Transforming growth factor‐alpha in human hepatocellular carcinoma and coexpression with hepatitis B surface antigen in adjacent liver

  1992cited by this paper
• Changes in expression of cellular oncogenes and endogenous retrovirus-like sequences during hepatocarcinogenesis induced by a peroxisome proliferator.

  1991cited by this paper
• Age-related susceptibility to the carcinogenic effect of the peroxisome proliferator WY-14,643 in rat liver.

  1991cited by this paper
• Role of oxidative stress in age dependent hepatocarcinogenesis by the peroxisome proliferator nafenopin in the rat.

  1991cited by this paper
• Altered expression of transforming growth factor-alpha in hereditary rat renal cell carcinoma.

  1991cited by this paper
• TGF alpha overexpression in transgenic mice induces liver neoplasia and abnormal development of the mammary gland and pancreas.

  1990cited by this paper
• Basic fibroblast growth factor and transforming growth factor‐α are hepatotrophic mitogens in vitro

  1990cited by this paper
• ImmunohistochemicaI and UItrastructuraI LocaIization of Epidermal Growth Factor Receptor in Human Liver and Hepatocellular Carcinoma Tissues

  1990cited by this paper
• Expression of transforming growth factor alpha and epidermal growth factor receptor messenger RNA in neoplastic and nonneoplastic human kidney tissue.

  1989cited by this paper
• Transforming growth factor alpha may be a physiological regulator of liver regeneration by means of an autocrine mechanism.

  1989cited by this paper
• Phenobarbital decreases hepatocyte EGF receptor expression independent of protein kinase C activation.

  1989cited by this paper
• Regulation of liver growth: protooncogenes and transforming growth factors.

  1989cited by this paper
• Synthesis of messenger RNAs for transforming growth factors alpha and beta and the epidermal growth factor receptor by human tumors.

  1987cited by this paper

• Chemicals with Carcinogenic Activity Primarily in Rodent Liver

  2018cites this paper
• Vitamin D3 supplementation attenuates the early stage of mouse hepatocarcinogenesis promoted by hexachlorobenzene fungicide.

  2017cites this paper
• Chemicals with Carcinogenic Activity in Rodent Liver

  2010cites this paper
• Disruption of Smad-dependent signaling for growth of GST-P-positive lesions from the early stage in a rat two-stage hepatocarcinogenesis model.

  2010cites this paper
• Crosstalk between PTEN/Akt2 and TGFβ signaling involving EGF receptor down‐regulation during the tumor promotion process from the early stage in a rat two‐stage hepatocarcinogenesis model

  2009cites this paper
• On the incorporation of biokinetic and mechanistic data in modeling for risk assessment

  2007cites this paper
• Key Issues in the Modes of Action and Effects of Trichloroethylene Metabolites for Liver and Kidney Tumorigenesis

  2006cites this paper
• HMG-CoA reductase and PPARα are involved in clofibrate-induced apoptosis in human keratinocytes

  2006cites this paper
• Response to Comments by Caldwell et al. on Article “Applying Mode-of-Action and Pharmacokinetic Considerations in Contemporary Cancer Risk Assessments: An Example with Trichloroethylene”

  2006cites this paper
• Differences in cell proliferation in rodent and human hepatic derived cell lines exposed to ciprofibrate.

  2005cites this paper
• Inherent growth advantage of (pre)malignant hepatocytes associated with nuclear translocation of pro-transforming growth factor α

  2004cites this paper
• Applying Mode-of-Action and Pharmacokinetic Considerations in Contemporary Cancer Risk Assessments: An Example with Trichloroethylene

  2004cites this paper
• Expression of peroxisome proliferator-activated receptor alpha, and PPARalpha regulated genes in spontaneously developed hepatocellular carcinomas in fatty acyl-CoA oxidase null mice.

  2002cites this paper
• Role of Thyroid Hormones in Hepatic Effects of Peroxisome Proliferators

  2001cites this paper
• Under What Conditions Is Trichloroethylene Likely To Be a Carcinogen in Humans?

  2001cites this paper
• Role of transforming growth factor alpha and prostaglandins in preferential growth of preneoplastic rat hepatocytes.

  2001cites this paper
• The functions of cytokines and their uses in toxicology

  2001cites this paper
• Are the Phenotypes of Preneoplastic Lesions of Significance for Cancer Prevention? 1. Liver.

  2001cites this paper
• Hepatitis C virus core protein activates the MAPK/ERK cascade synergistically with tumor promoter TPA, but not with epidermal growth factor or transforming growth factor α

  2000cites this paper
• Apoptosis, mitosis and cyclophilin-40 expression in regressing peroxisome proliferator-induced adenomas.

  2000cites this paper
• Hepatitis C virus core protein activates the MAPK/ERK cascade synergistically with tumor promoter TPA, but not with epidermal growth factor or transforming growth factor alpha.

  2000cites this paper
• Central role of PPARalpha in the mechanism of action of hepatocarcinogenic peroxisome proliferators.

  2000cites this paper
• EGF receptors of hepatocytes from rats treated with phenobarbital are sensitized to down-regulation by phenobarbital in culture.

  2000cites this paper
• Alteration of protein kinase C isoform-specific expression during rat hepatocarcinogenesis after exposure to the peroxisome proliferator WY-14,643.

  1999cites this paper
• Hepatitis C Virus and Hepatocarcinogenesis

  1999cites this paper
• Hepatic expression of acute‐phase protein genes during carcinogenesis induced by peroxisome proliferators

  1999cites this paper
• Role of peroxisome proliferator-activated receptor alpha in altered cell cycle regulation in mouse liver.

  1998cites this paper
• Interspecies comparison of liver carcinogenesis: implications for cancer risk assessment.

  1997cites this paper
• Foci of Altered Hepatocytes, Rat

  1997cites this paper
• Quantitative Relationship Between Transforming Growth Factor-Alpha and Hepatic Focal Phenotype and Progression in Female Mouse Liver

  1997cites this paper
• Chemicals with carcinogenic activity in the rodent liver; mechanistic evaluation of human risk.

  1997cites this paper
• Transforming growth factor-alpha in carcinogen-induced F344 rat hepatic foci.

  1996cites this paper
• Quantitation of Multistage Carcinogenesis in Rat Liver

  1996cites this paper
• Focal altered compartmentation of repetitive B2 (Alu-like) sequences in rat liver following hepatocarcinogen exposure.

  1996cites this paper
• Expression of hepatocyte growth factor and c-met mRNAs during rat chemically induced hepatocarcinogenesis.

  1996cites this paper
• Effect on the expression of c-met, c-myc and PPAR-alpha in liver and liver tumors from rats chronically exposed to the hepatocarcinogenic peroxisome proliferator WY-14,643.

  1996cites this paper
• The quantitation of altered hepatic foci during multistage hepatocarcinogenesis in the rat: transforming growth factor alpha expression as a marker for the stage of progression.

  1995cites this paper