New PTH Signals Mediating Bone Anabolism

No abstract is available for this paper.

• Publication year

  2017

• Venue

  Current Molecular Biology Reports

• Publication date

  2017-04-22

• Fields of study

  Biology, Medicine, Chemistry

• Identifiers
  DOI 10.1007/s40610-017-0060-z
• External record

  Open on Semantic Scholar

• Source metadata

  Semantic Scholar

• No claims are published for this paper.

• No concepts are published for this paper.

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• Lipoprotein receptor–related protein 6 is required for parathyroid hormone–induced Sost suppression

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• β-Catenin-Independent Roles of Wnt/LRP6 Signaling.

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• PTH-Induced Osteoblast Proliferation Requires Upregulation of the Ubiquitin-Specific Peptidase 2 (Usp2) Expression

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• HDAC5 Controls MEF2C‐Driven Sclerostin Expression in Osteocytes

  2015cited by this paper
• PTH receptor-1 signalling—mechanistic insights and therapeutic prospects

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• N‐cadherin Restrains PTH Activation of Lrp6/β‐catenin Signaling and Osteoanabolic Action

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• PTH Receptor Signaling in Osteoblasts Regulates Endochondral Vascularization in Maintenance of Postnatal Growth Plate

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• Sirtuin 1 Is a Negative Regulator of Parathyroid Hormone Stimulation of Matrix Metalloproteinase 13 Expression in Osteoblastic Cells

  2015cited by this paper
• Loss of Gsα in the Postnatal Skeleton Leads to Low Bone Mass and a Blunted Response to Anabolic Parathyroid Hormone Therapy*

  2015cited by this paper
• T Cell–Expressed CD40L Potentiates the Bone Anabolic Activity of Intermittent PTH Treatment

  2015cited by this paper
• Low-dose PTH increases osteoblast activity via decreased Mef2c/Sost in senescent osteopenic mice.

  2014cited by this paper
• The PTH-Gαs-Protein Kinase A Cascade Controls αNAC Localization To Regulate Bone Mass

  2014cited by this paper
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  2014cited by this paper
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  2014cited by this paper
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  2013cited by this paper
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  2013cited by this paper
• Disruption of LRP6 in osteoblasts blunts the bone anabolic activity of PTH

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• Parathyroid Hormone (PTH)/PTH-related Peptide Type 1 Receptor (PPR) Signaling in Osteocytes Regulates Anabolic and Catabolic Skeletal Responses to PTH*

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  2012cited by this paper
• Targeted deletion of Sost distal enhancer increases bone formation and bone mass

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  2010cited by this paper
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  2010cited by this paper
• PTH Receptor Signaling in Osteocytes Governs Periosteal Bone Formation and Intracortical Remodeling

  2010cited by this paper
• Regulatory signal transduction pathways for class IIa histone deacetylases.

  2010cited by this paper
• Treatment of Osteoporosis with Parathyroid Hormone and Teriparatide

  2009cited by this paper
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• Nuclear αNAC Influences Bone Matrix Mineralization and Osteoblast Maturation In Vivo

  2009cited by this paper
• A β-Arrestin–Biased Agonist of the Parathyroid Hormone Receptor (PTH1R) Promotes Bone Formation Independent of G Protein Activation

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• Critical Role of Activating Transcription Factor 4 in the Anabolic Actions of Parathyroid Hormone in Bone

  2009cited by this paper
• The transcription factor ATF4 regulates glucose metabolism in mice through its expression in osteoblasts.

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  2007cited by this paper
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• Sequence-Specific DNA Binding by the αNAC Coactivator Is Required for Potentiation of c-Jun-Dependent Transcription of the Osteocalcin Gene

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• Effects of parathyroid hormone on Wnt signaling pathway in bone

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• Bone response to intermittent parathyroid hormone is altered in mice null for {beta}-Arrestin2.

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• Gα12/Gα13 Subunits of Heterotrimeric G Proteins Mediate Parathyroid Hormone Activation of Phospholipase D in UMR-106 Osteoblastic Cells

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  2004cited by this paper
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  2003cited by this paper
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