TNFα is required for the production of T-type Ca(2+) channel-dependent long-term potentiation in visual cortex.

No abstract is available for this paper.

• Publication year

  2015

• Venue

  Neurosciences research

• Publication date

  2015-07-01

• Fields of study

  Biology, Medicine, Chemistry

• Identifiers
  DOI 10.1016/j.neures.2015.02.005PMID 25701075
• External record

  Open on Semantic Scholar

• Source metadata

  Semantic Scholar, PubMed

• No claims are published for this paper.

• No concepts are published for this paper.

• Ni(2+)-sensitive T-type Ca(2+) channel currents are regulated in parallel with synaptic and visual response plasticity in visual cortex.

  2014cited by this paper
• Silent synapses persist into adulthood in layer 2/3 pyramidal neurons of visual cortex in dark-reared mice.

  2013cited by this paper
• Synaptic and Intrinsic Homeostatic Mechanisms Cooperate to Increase L2/3 Pyramidal Neuron Excitability during a Late Phase of Critical Period Plasticity

  2013cited by this paper
• TNF-α Downregulates Inhibitory Neurotransmission through Protein Phosphatase 1-Dependent Trafficking of GABAA Receptors

  2013cited by this paper
• Growth factors in synaptic function

  2013cited by this paper
• Homeostatic plasticity mechanisms are required for juvenile, but not adult, ocular dominance plasticity

  2012cited by this paper
• TNFα in synaptic function: switching gears.

  2012cited by this paper
• The Discovery and Characterization of ML218: A Novel, Centrally Active T-Type Calcium Channel Inhibitor with Robust Effects in STN Neurons and in a Rodent Model of Parkinson's Disease.

  2011cited by this paper
• Regulation of the proteolytic disintegrin metalloproteinases, the 'Sheddases'.

  2009influential reference
• Essential role for a long-term depression mechanism in ocular dominance plasticity

  2009cited by this paper
• Cannabinoid receptor blockade reveals parallel plasticity mechanisms in different layers of mouse visual cortex.

  2008cited by this paper
• The self-tuning neuron: synaptic scaling of excitatory synapses.

  2008cited by this paper
• Tumor necrosis factor-alpha mediates one component of competitive, experience-dependent plasticity in developing visual cortex.

  2008influential reference
• Involvement of T‐type Ca2+ channels in the potentiation of synaptic and visual responses during the critical period in rat visual cortex

  2008influential reference
• Synaptic scaling mediated by glial TNF-α

  2006influential reference
• Synaptic scaling mediated by glial TNF-alpha.

  2006cited by this paper
• Small-molecule inhibition of TNF-alpha.

  2005cited by this paper
• Small-Molecule Inhibition of TNF-α

  2005cited by this paper
• Actions of TNF‐α on glutamatergic synaptic transmission in the central nervous system

  2005cited by this paper
• Actions of TNF-alpha on glutamatergic synaptic transmission in the central nervous system.

  2005cited by this paper
• ReportHow Monocular Deprivation Shifts Ocular Dominance in Visual Cortex of Young Mice

  2004cited by this paper
• Age and experience dependence of N-methyl-D-aspartate receptor-independent long-term potentiation in rat visual cortex.

  2003cited by this paper
• Molecular mechanism for loss of visual cortical responsiveness following brief monocular deprivation

  2003cited by this paper
• Control of Synaptic Strength by Glial TNFα

  2002cited by this paper
• Control of synaptic strength by glial TNFalpha.

  2002cited by this paper
• Electrical activity and development of neural circuits

  2001cited by this paper
• Activity-dependent scaling of quantal amplitude in neocortical neurons

  1998cited by this paper
• A metalloproteinase disintegrin that releases tumour-necrosis factor-α from cells

  1997cited by this paper
• Cloning of a disintegrin metalloproteinase that processes precursor tumour-necrosis factor-α

  1997cited by this paper
• A metalloproteinase disintegrin that releases tumour-necrosis factor-alpha from cells.

  1997cited by this paper
• Interleukin-1β (IL-1β) and tumour necrosis factor (TNF) inhibit long-term potentiation in the rat dentate gyrus in vitro

  1996cited by this paper
• Experience-Dependent Plasticity of Binocular Responses in the Primary Visual Cortex of the Mouse

  1996cited by this paper
• Interleukin-1 beta (IL-1 beta) and tumour necrosis factor (TNF) inhibit long-term potentiation in the rat dentate gyrus in vitro.

  1996cited by this paper
• Synaptic Activity and the Construction of Cortical Circuits

  1996cited by this paper
• Development and plasticity of cortical processing architectures.

  1995cited by this paper
• Development and Plasticity of Cortical Processing Architectures

  1995cited by this paper
• Functional postnatal development of the rat primary visual cortex and the role of visual experience: dark rearing and monocular deprivation.

  1994cited by this paper
• Age-dependent long-term potentiation of inhibitory synaptic transmission in rat visual cortex

  1994cited by this paper
• Tumor necrosis factor alters synaptic transmission in rat hippocampal slices.

  1992cited by this paper
• Low-threshold Ca2+ channels mediate induction of long-term potentiation in kitten visual cortex.

  1992cited by this paper
• Long-term potentiation of synaptic transmission in kitten visual cortex.

  1988cited by this paper
• A physiological basis for a theory of synapse modification.

  1987cited by this paper
• The postnatal development of the visual cortex and the influence of environment.

  1982cited by this paper
• Exploration of the primary visual cortex, 1955–78

  1982cited by this paper
• Postnatal development of the visual cortex and the influence of environment

  1982cited by this paper
• Comparison of the effects of dark rearing and binocular suture on development and plasticity of cat visual cortex.

  1981cited by this paper
• Prolonged sensitivity to monocular deprivation in dark-reared cats.

  1980influential reference
• A physiological mechanism for Hebb's postulate of learning.

  1973cited by this paper
• The period of susceptibility to the physiological effects of unilateral eye closure in kittens

  1970influential reference

• Alcohol, HMGB1, and Innate Immune Signaling in the Brain

  2024cites this paper
• Tumor necrosis factor alpha induces NOX2-dependent reactive oxygen species production in hypothalamic paraventricular nucleus neurons following angiotensin II infusion.

  2024cites this paper
• Diet Control and Swimming Exercise Ameliorate HFD-Induced Cognitive Impairment Related to the SIRT1-NF-κB/PGC-1α Pathways in ApoE-/- Mice

  2023cites this paper
• Effects of tumor necrosis factor-α polymorphism on the brain structural changes of the patients with major depressive disorder

  2018cites this paper
• Innate Immune Signaling and Alcohol Use Disorders.

  2018cites this paper
• Visual experience regulates the development of long-term synaptic modifications induced by low-frequency stimulation in mouse visual cortex.

  2017cites this paper
• Requirement of keratan sulfate proteoglycan phosphacan with a specific sulfation pattern for critical period plasticity in the visual cortex.

  2015influential citation