Near Death Experience Cells Escaping a Non-MHC Gene-Controlled Enhanced Pathogenicity of Diabetogenic

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  2004

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• MHC Class II Molecules Play a Role in the Selection of Autoreactive Class I-Restricted CD8 T Cells That Are Essential Contributors to Type 1 Diabetes Development in Nonobese Diabetic Mice1

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• Mouse T-cell receptor variable gene segment families

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• A Defect in Tryptophan Catabolism Impairs Tolerance in Nonobese Diabetic Mice

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• Surface T‐cell antigen receptor expression and availability for long‐term antigenic signaling

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• Efficient T cell receptor–mediated apoptosis in nonobese diabetic mouse thymocytes

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• Apoptosis during lymphoid development.

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• T-cell compartments of prediabetic NOD mice.

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• Cutting Edge: Identification of the Targets of Clonal Deletion in an Unmanipulated Thymus1

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• Susceptible MHC alleles, not background genes, select an autoimmune T cell reactivity.

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• Thymic generation and regeneration

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• Genetic Disassociation of Autoimmunity and Resistance to Costimulation Blockade-Induced Transplantation Tolerance in Nonobese Diabetic Mice1

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• Dissociation of peripheral T cell responses from thymocyte negative selection by weak agonists supports a spare receptor model of T cell activation

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• Nuclear hormone receptors in T lymphocytes.

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• Failure to Censor Forbidden Clones of CD4 T Cells in Autoimmune Diabetes

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• Maintaining the norm: T-cell homeostasis

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• Autoreactive CD8 T cells in organ-specific autoimmunity: emerging targets for therapeutic intervention.

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  2002cited by this paper
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• Phenotypic and Functional Analysis of Cd8+ T Cells Undergoing Peripheral Deletion in Response to Cross-Presentation of Self-Antigen

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• HLA associations in type 1 diabetes: merging genetics and immunology.

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• β-Cell death during progression to diabetes

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• Autoreactive diabetogenic T-cells in NOD mice can efficiently expand from a greatly reduced precursor pool.

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• Defective CD8+ T Cell Peripheral Tolerance in Nonobese Diabetic Mice1

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• Identification of a CD8 T Cell That Can Independently Mediate Autoimmune Diabetes Development in the Complete Absence of CD4 T Cell Helper Functions1

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• Selection of the T cell repertoire.

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• Major histocompatibility complex class I-restricted T cells are required for all but the end stages of diabetes development in nonobese diabetic mice and use a prevalent T cell receptor alpha chain gene rearrangement.

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• B lymphocytes are essential for the initiation of T cell-mediated autoimmune diabetes: analysis of a new "speed congenic" stock of NOD.Ig mu null mice

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• A role for the orphan steroid receptor Nur77 in apoptosis accompanying antigen-induced negative selection.

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• Are Diabetes and Insulitis Resistant

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• Defects in the differentiation and function of antigen presenting cells in NOD/Lt mice.

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• The nonobese diabetic scid mouse: model for spontaneous thymomagenesis associated with immunodeficiency.

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• Development of diabetogenic T cells from NOD/Lt marrow is blocked when an allo-H-2 haplotype is expressed on cells of hemopoietic origin, but not on thymic epithelium.

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• Tolerance induction in double specific T-cell receptor transgenic mice varies with antigen

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• Thymus cell migration: Quantitative aspects of cellular traffic from the thymus to the periphery in mice

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• No citing papers are available for this paper.