Neutrophil CD I I B expression and neutrophil activation in pre-eclam psia

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  2012

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• Neutrophil-mediated vascular injury.

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• Clinical and biochemical evidence of endothelial cell dysfunction in the pregnancy syndrome preeclampsia.

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• Women with preeclampsia have higher plasma endothelin levels than women with normal pregnancies.

  1990cited by this paper
• Effect of platelet activating factor on leukocyte-endothelial cell interactions.

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• Decreased prostacyclin biosynthesis preceding the clinical manifestation of pregnancy-induced hypertension.

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• Plasma fibronectin levels in preeclampsia: a possible biochemical marker for vascular endothelial damage.

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• Thromboxane A2 mediates augmented polymorphonuclear leukocyte adhesiveness.

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• Platelet life-span in normal pregnancy and pre-eclampsia as determined by a non-radioisotope technique.

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• Early platelet consumption in pre-eclampsia.

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• Factor-VIII consumption in pre-eclampsia.

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• No citing papers are available for this paper.