Involvement of nitric oxide in 3-nitropropionic acid-induced depression of spinal reflexes in neonatal rat spinal cord in vitro.

No abstract is available for this paper.

• Publication year

  2009

• Venue

  European Journal of Pharmacology

• Publication date

  2009-09-01

• Fields of study

  Biology, Medicine, Chemistry

• Identifiers
  DOI 10.1016/j.ejphar.2009.06.054PMID 19577555
• External record

  Open on Semantic Scholar

• Source metadata

  Semantic Scholar, PubMed

• No claims are published for this paper.

• No concepts are published for this paper.

• Concepts of neural nitric oxide-mediated transmission

  2008cited by this paper
• 3-Nitropropionic acid-induced depression of spinal reflexes involves mechanisms different from ischemia-induced depression.

  2008cited by this paper
• Role of GABA receptors in nitric oxide inhibition of dorsolateral periaqueductal gray neurons.

  2008cited by this paper
• Tiagabine, a GABA uptake inhibitor, attenuates 3-nitropropionic acid-induced alterations in various behavioral and biochemical parameters in rats.

  2008cited by this paper
• NO/cGMP-dependent modulation of synaptic transmission.

  2008cited by this paper
• 3-Nitropropionic acid depresses spinal reflexes involving GABAergic and glycinergic transmission in neonatal rat spinal cord in vitro.

  2008cited by this paper
• 3-Nitropropionic acid-induced depression of spinal reflexes does not involve 5-hydroxytryptaminergic system in contrast to ischemia-induced depression in neonatal rat spinal cord in vitro.

  2008cited by this paper
• Nitric oxide in the central nervous system: neuroprotection versus neurotoxicity

  2007cited by this paper
• Differential sensitivity of excitatory and inhibitory synaptic transmission to modulation by nitric oxide in rat nucleus tractus solitarii

  2007cited by this paper
• Involvement of nitric oxide in 3-nitropropionic acid-induced striatal toxicity in rats.

  2006cited by this paper
• Aglycemia and ischemia depress monosynaptic and polysynaptic reflexes in neonatal rat spinal cord in vitro by involving different types of 5-hydroxytryptamine receptors.

  2004cited by this paper
• Nitric Oxide and Memory

  2004cited by this paper
• Differential depression of spinal synaptic transmission in vitro by different hypoxic insults.

  2004cited by this paper
• Estrogen blocks 3-nitropropionic acid-induced[Ca[2+]]i increase and cell damage in cultured rat cerebral endothelial cells

  2003cited by this paper
• Effect of ischaemia & aglycaemia on the synaptic transmission in neonatal rat spinal cord in vitro.

  2003cited by this paper
• Age-associated changes in SAPK/JNK and p38 MAPK signaling in response to the generation of ROS by 3-nitropropionic acid.

  2003cited by this paper
• Deprenyl blocks the aglycemia-induced depression of the synaptic transmission but not the ischemia-induced depression in neonatal rat spinal cord in vitro.

  2003cited by this paper
• Aglycemia and ischemia depress spinal synaptic transmission via inhibitory systems involving NMDA receptors.

  2003cited by this paper
• Involvement of N-methyl-D-aspartate receptors for the Ptychodiscus brevis toxin-induced depression of monosynaptic and polysynaptic reflexes in neonatal rat spinal cord in vitro.

  2002cited by this paper
• Estrogen blocks 3-nitropropionic acid-induced Ca2+i increase and cell damage in cultured rat cerebral endothelial cells.

  2002cited by this paper
• Nitric oxide as modulator of neuronal function.

  2001cited by this paper
• Excessive release of [3H]noradrenaline and glutamate in response to simulation of ischemic conditions in rat spinal cord slice preparation: effect of NMDA and AMPA receptor antagonists.

  1999cited by this paper
• Nitric Oxide Acutely Inhibits Neuronal Energy Production

  1999cited by this paper
• Nitric oxide acutely inhibits neuronal energy production. The Committees on Neurobiology and Cell Physiology.

  1999cited by this paper
• Neuroprotection by both NMDA and non-NMDA receptor antagonists in in vitro ischemia.

  1997cited by this paper
• Roles of nitric oxide in compression injury of rat spinal cord.

  1996cited by this paper
• Methods in nitric oxide research

  1996cited by this paper
• Lipid peroxidation in rats intoxicated with 3-nitropropionic acid.

  1995cited by this paper
• The Nitric Oxide ‐ Cyclic GMP Pathway and Synaptic Depression in Rat Hippocampal Slices

  1994cited by this paper
• Borrelia burgdorferi and Escherichia coli lipopolysaccharides induce nitric oxide and interleukin-6 production in cultured rat brain cells.

  1994cited by this paper
• Effect of exogenous and endogenous nitric oxide on mitochondrial respiration of rat hepatocytes.

  1991cited by this paper
• Gender‐specific action of thyrotropin‐releasing hormone in the mammalian spinal cord 1

  1987cited by this paper
• Overview of enzyme systems involved in bio-reduction of drugs and in redox cycling.

  1986cited by this paper
• Metabolic fate of nitric oxide

  1980cited by this paper
• Inactivation of succinate dehydrogenase by 3-nitropropionate.

  1979cited by this paper
• 3-Nitropropionate, the toxic substance of Indigofera, is a suicide inactivator of succinate dehydrogenase.

  1977cited by this paper
• Electrophysiology of mammalian spinal cord in vitro

  1974cited by this paper

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