Dual effect of insulin-like growth factor on the apical 70-pS K channel in the thick ascending limb of rat kidney.

No abstract is available for this paper.

• Publication year

  2004

• Venue

  American Journal of Physiology - Cell Physiology

• Publication date

  2004-06-01

• Fields of study

  Biology, Medicine

• Identifiers
  DOI 10.1152/AJPCELL.00441.2003PMID 15151916
• External record

  Open on Semantic Scholar

• Source metadata

  Semantic Scholar, PubMed

• No claims are published for this paper.

• No concepts are published for this paper.

• The luminal K+ channel of the thick ascending limb of Henle's loop

  2004cited by this paper
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• Regulation of Insulin-like Growth Factor I Receptor Dephosphorylation by SHPS-1 and the Tyrosine Phosphatase SHP-2*

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• IGF-1 up-regulates K+ channels via PI3-kinase, PDK1 and SGK1

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• Identification of a Novel Inhibitor of Mitogen-activated Protein Kinase Kinase*

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• Nitric oxide increases the activity of the apical 70-pS K+ channel in TAL of rat kidney.

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• Localization of the ROMK protein on apical membranes of rat kidney nephron segments.

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• Effect of angiotensin II on the apical K+ channel in the thick ascending limb of the rat kidney

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• Insulin-like growth factors and their binding proteins: biological actions.

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• Potentiation by insulin and insulin-like growth factor-1 of glibenclamide-sensitive K+ currents in follicle-enclosed Xenopus oocytes.

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• A potassium channel in the apical membrane of rabbit thick ascending limb of Henle's loop.

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• Printed in U.S.A. Copyright © 2000 by The Endocrine Society High Insulin-Like Growth Factor 1 (IGF-1) and Insulin Concentrations Trigger Apoptosis in the Mouse Blastocyst via Down-Regulation of the IGF-1 Receptor*

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