Synergetic inhibition of the brain mitochondrial NADH: Ubiquinone oxidoreductase (Complex I) by fatty acids and Ca2+

No abstract is available for this paper.

• Publication year

  2011

• Venue

  Biochemistry (Moscow)

• Publication date

  2011-08-16

• Fields of study

  Biology, Medicine, Chemistry

• Identifiers
  DOI 10.1134/S000629791108013XPMID 22022971
• External record

  Open on Semantic Scholar

• Source metadata

  Semantic Scholar, PubMed

• No claims are published for this paper.

• No concepts are published for this paper.

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  2003cited by this paper
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  2003cited by this paper
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  2003cited by this paper
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  2002cited by this paper
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  2002cited by this paper
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  2001cited by this paper
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  2001cited by this paper
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  1999cited by this paper
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  1999cited by this paper
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  1998cited by this paper
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  1998cited by this paper
• Uncoupling: new approaches to an old problem of bioenergetics.

  1998cited by this paper
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  1995cited by this paper
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  1993cited by this paper
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  1993cited by this paper
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  1993cited by this paper
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  1992cited by this paper
• Conservation of sequences of subunits of mitochondrial complex I and their relationships with other proteins.

  1992cited by this paper
• A possible mechanism of mitochondrial dysfunction during cerebral ischemia: inhibition of mitochondrial respiration activity by arachidonic acid.

  1991cited by this paper
• Slow active/inactive transition of the mitochondrial NADH-ubiquinone reductase.

  1990cited by this paper
• Ion Pair Transport Across Membranes

  1989cited by this paper
• Effects of arachidonic acid on respiratory activities in isolated brain mitochondria

  1988cited by this paper
• The modes of action of long chain alkyl compounds on the respiratory chain-linked energy transducing system in submitochondrial particles.

  1986cited by this paper
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  1985cited by this paper
• Ion pair absorption of ionized drugs —fact or fiction ?

  1983cited by this paper
• Activity of phospholipase A2 in the inner membrane of rat-liver mitochondria.

  1981cited by this paper
• Selective inactivation of the NADH‐ubiquinone segment of the respiratory chain of submitochondrial particles by endogenous free fatty acids during hyperthermia

  1978cited by this paper
• Uncoupling activity of endogenous free fatty acids in rat liver mitochondria.

  1978cited by this paper
• [Slow irreversible inhibition of the respiratory chain of non-phosphorylating submitochondrial particles by free fatty acids and their methyl esters and monoglycerides].

  1977cited by this paper
• The organic chemistry of electrolyte solutions

  1975cited by this paper
• On a slow inhibitory effect of free fatty acids on the respiratory chain of non‐phosphorylating submitochondrial particles from beef heart

  1974cited by this paper
• [Relation between molecular structure and inhibiting effect of fatty acids and monoglycerides on the respiratory chain of mitochondrial particles].

  1971cited by this paper
• Comparative study of thermal degradation of electron transfer particle and reconstituted respiratory chain. Relation of electron transfer to reactivation of submitochondrial particles.

  1970cited by this paper

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